Single Nucleotide Polymorphisms Associated With Gut Homeostasis Influence Risk and Age-at-Onset of Parkinson's Disease.
Parkinson's disease
gene-environment interactions
gut inflammation
gut-brain axis
peptidoglycan recognition protein
single nucleotide polymorphisms
toll-like receptors
Journal
Frontiers in aging neuroscience
ISSN: 1663-4365
Titre abrégé: Front Aging Neurosci
Pays: Switzerland
ID NLM: 101525824
Informations de publication
Date de publication:
2020
2020
Historique:
received:
08
09
2020
accepted:
20
10
2020
entrez:
17
12
2020
pubmed:
18
12
2020
medline:
18
12
2020
Statut:
epublish
Résumé
Research is increasingly focusing on gut inflammation as a contributor to Parkinson's disease (PD). Such gut inflammation is proposed to arise from a complex interaction between various genetic, environmental, and lifestyle factors, however these factors are under-characterized. This study investigated the association between PD and single-nucleotide polymorphisms (SNPs) in genes responsible for binding of bacterial metabolites and intestinal homeostasis, which have been implicated in intestinal infections or inflammatory bowel disease. A case-control analysis was performed utilizing the following cohorts: (i) patients from the Australian Parkinson's Disease Registry (APDR) (
Identifiants
pubmed: 33328979
doi: 10.3389/fnagi.2020.603849
pmc: PMC7718032
doi:
Types de publication
Journal Article
Langues
eng
Pagination
603849Informations de copyright
Copyright © 2020 Gorecki, Bakeberg, Theunissen, Kenna, Hoes, Pfaff, Akkari, Dunlop, Kõks, Mastaglia and Anderton.
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