Gender difference in lipoprotein(a) concentration as a predictor of coronary revascularization in patients with known coronary artery disease.


Journal

Biochimica et biophysica acta. Molecular and cell biology of lipids
ISSN: 1879-2618
Titre abrégé: Biochim Biophys Acta Mol Cell Biol Lipids
Pays: Netherlands
ID NLM: 101731727

Informations de publication

Date de publication:
03 2021
Historique:
received: 08 09 2020
revised: 04 12 2020
accepted: 12 12 2020
pubmed: 18 12 2020
medline: 30 6 2021
entrez: 17 12 2020
Statut: ppublish

Résumé

Whether there is a gender difference in the impact of elevated plasma Lp(a) levels on recurrent coronary events remains unclear. We, therefore, evaluated the association between Lp(a) levels and the occurrence of major adverse coronary events in a large series of coronary patients (32% women). This single-center prospective cohort study investigated 3034 consecutive patients admitted to the Coronary Care Unit with a diagnosis of coronary ischemia. According to the inclusion criteria, 2374 patients completed the follow-up (mean of 2 years). The end-points were non-fatal myocardial infarction (MI), revascularization and coronary deaths. Elevated Lp(a) levels were significantly associated with rate of revascularization, but not with non-fatal MI and cardiac death. According to Lp(a) stratification (≤30 mg/dl, >30-50 mg/dl and ≥50 mg/dl), there was a significant rise of revascularization events in the whole sample of participants, with a trend in hazard ratio (HR) of 1.23 (95% CI 1.04-1.46) and a 6% rise for every 10 mg/dl increment in Lp(a) levels. This effect was mainly driven by women (HR 2.04, 95%CI 1.33-3.12) who showed a 14% incremental risk for every 10 mg/dl rise in Lp(a) levels. In patients with coronary artery disease, elevated plasma Lp(a) levels were found to be a potentially useful predictor of the need for coronary revascularizations, especially in women.

Sections du résumé

BACKGROUND AND AIMS
Whether there is a gender difference in the impact of elevated plasma Lp(a) levels on recurrent coronary events remains unclear. We, therefore, evaluated the association between Lp(a) levels and the occurrence of major adverse coronary events in a large series of coronary patients (32% women).
METHODS
This single-center prospective cohort study investigated 3034 consecutive patients admitted to the Coronary Care Unit with a diagnosis of coronary ischemia. According to the inclusion criteria, 2374 patients completed the follow-up (mean of 2 years). The end-points were non-fatal myocardial infarction (MI), revascularization and coronary deaths.
RESULTS
Elevated Lp(a) levels were significantly associated with rate of revascularization, but not with non-fatal MI and cardiac death. According to Lp(a) stratification (≤30 mg/dl, >30-50 mg/dl and ≥50 mg/dl), there was a significant rise of revascularization events in the whole sample of participants, with a trend in hazard ratio (HR) of 1.23 (95% CI 1.04-1.46) and a 6% rise for every 10 mg/dl increment in Lp(a) levels. This effect was mainly driven by women (HR 2.04, 95%CI 1.33-3.12) who showed a 14% incremental risk for every 10 mg/dl rise in Lp(a) levels.
CONCLUSIONS
In patients with coronary artery disease, elevated plasma Lp(a) levels were found to be a potentially useful predictor of the need for coronary revascularizations, especially in women.

Identifiants

pubmed: 33333178
pii: S1388-1981(20)30261-4
doi: 10.1016/j.bbalip.2020.158869
pii:
doi:

Substances chimiques

Lipoprotein(a) 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

158869

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Federico Bigazzi (F)

U.O. Lipoapheresis and Center for Inherited Dyslipidaemias, Fondazione Toscana Gabriele Monasterio, Pisa, Italy.

Fabrizio Minichilli (F)

Unit of Environmental Epidemiology and Disease Registries, Institute of Clinical Physiology, National Research Council, Pisa, Italy.

Francesco Sbrana (F)

U.O. Lipoapheresis and Center for Inherited Dyslipidaemias, Fondazione Toscana Gabriele Monasterio, Pisa, Italy.

Beatrice Dal Pino (BD)

U.O. Lipoapheresis and Center for Inherited Dyslipidaemias, Fondazione Toscana Gabriele Monasterio, Pisa, Italy.

Alberto Corsini (A)

Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, Italy; Multimedica IRCCS, Milano, Italy.

Gerald F Watts (GF)

School of Medicine, Faculty of Health and Medical Sciences, University of Western Australia, Perth, Australia; Lipid Disorders Clinic, Cardiometabolic Services, Department of Cardiology, Royal Perth Hospital, Australia.

Cesare R Sirtori (CR)

Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, Italy.

Massimiliano Ruscica (M)

Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, Italy. Electronic address: massimiliano.ruscica@unimi.it.

Tiziana Sampietro (T)

U.O. Lipoapheresis and Center for Inherited Dyslipidaemias, Fondazione Toscana Gabriele Monasterio, Pisa, Italy. Electronic address: tiziana.sampietro@ftgm.com.

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