ChREBP-Mediated Regulation of Lipid Metabolism: Involvement of the Gut Microbiota, Liver, and Adipose Tissue.
Acetyl Coenzyme A
/ metabolism
Adipose Tissue
/ metabolism
Animals
Basic Helix-Loop-Helix Leucine Zipper Transcription Factors
/ metabolism
Fatty Acids
/ biosynthesis
Gastrointestinal Microbiome
Glucose
/ metabolism
Humans
Insulin Resistance
Lipogenesis
Lipoproteins, LDL
/ metabolism
Liver
/ metabolism
Mice
Oxidation-Reduction
carbohydrate response element-binding protein (Chrebp)
fatty acid synthesis
gut microbiota
ketogenesis
lipoprotein metabolism
β-oxidation
Journal
Frontiers in endocrinology
ISSN: 1664-2392
Titre abrégé: Front Endocrinol (Lausanne)
Pays: Switzerland
ID NLM: 101555782
Informations de publication
Date de publication:
2020
2020
Historique:
received:
25
07
2020
accepted:
09
11
2020
entrez:
21
12
2020
pubmed:
22
12
2020
medline:
28
5
2021
Statut:
epublish
Résumé
Carbohydrate response element-binding protein (ChREBP) plays an important role in the development of type 2 diabetes, dyslipidemia, and non-alcoholic fatty liver disease, as well as tumorigenesis. ChREBP is highly expressed in lipogenic organs, such as liver, intestine, and adipose tissue, in which it regulates the production of acetyl CoA from glucose by inducing
Identifiants
pubmed: 33343508
doi: 10.3389/fendo.2020.587189
pmc: PMC7744659
doi:
Substances chimiques
Basic Helix-Loop-Helix Leucine Zipper Transcription Factors
0
Fatty Acids
0
Lipoproteins, LDL
0
MLXIPL protein, human
0
Mlxipl protein, mouse
0
Acetyl Coenzyme A
72-89-9
Glucose
IY9XDZ35W2
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
587189Informations de copyright
Copyright © 2020 Iizuka, Takao and Yabe.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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