Definitions, outcomes, and management of hyperprogression in patients with non-small-cell lung cancer treated with immune checkpoint inhibitors.

Cytotoxic T-lymphocyte-associated protein 4 antigen Hyperprogressive disease Immunotherapy Non-small-cell lung cancer Programmed cell death 1 ligand 2 protein Programmed cell death 1 receptor

Journal

Lung cancer (Amsterdam, Netherlands)
ISSN: 1872-8332
Titre abrégé: Lung Cancer
Pays: Ireland
ID NLM: 8800805

Informations de publication

Date de publication:
02 2021
Historique:
received: 08 11 2020
revised: 29 11 2020
accepted: 21 12 2020
pubmed: 2 1 2021
medline: 22 6 2021
entrez: 1 1 2021
Statut: ppublish

Résumé

The advent of immune checkpoint inhibitors (ICI) has been a breakthrough in the care of patients with non-small-cell lung cancers (NSCLC). However, physicians are now facing a previously unidentified clinical situation called hyperprogression (HP), which presents as a fast and unexpected increase in tumor burden. HP's existence and specificity to ICIs remains controversial because a widely acknowledged definition is currently lacking. Meanwhile, management remains elusive. Medical records from all consecutive NSCLC patients who were treated with ICI from 2015 to 2018 were retrospectively analyzed. The HP incidence rate was calculated according to five definitions (tumor growth rate [TGR]ratio, ΔTGR, tumor growth kinetic [TGK], RECIST, and time to treatment failure [TTF]), and the agreement between such definitions was determined. The HP impact on overall survival (OS) was then assessed. The association between HP (defined using the TGRratio definition) and clinical and biological variables was also assessed. Clinical HP management and its impact on outcomes were described. We identified 169 consecutive ICI-treated patients, with potential HP accounting for 11.3 %, 5.7 %, 17.0 %, 9.6 %, and 31.7 % patients, according to TGRratio, ΔTGR, TGK, RECIST, and TTF definitions. Agreement between the different HP definitions was highly heterogeneous (range 29 %-77 %) and globally poor. HP was associated with shorter OS, compared to standard RECIST progressive disease, but this difference only reached statistical significance when using the TTF definition. TGRratio-based HP was significantly associated with hepatic metastases. In TGRratio-based HP patients, neither resuming chemotherapy nor corticosteroids use was associated with statistically significant impact on overall survival. We found fairly heterogeneous HP rates using different definitions. TTF was the only definition leading to significantly worsened OS. Further studies are needed to provide consensus recommendations for the assessment, definition, and management of HP, whose existence is likely real.

Sections du résumé

BACKGROUND
The advent of immune checkpoint inhibitors (ICI) has been a breakthrough in the care of patients with non-small-cell lung cancers (NSCLC). However, physicians are now facing a previously unidentified clinical situation called hyperprogression (HP), which presents as a fast and unexpected increase in tumor burden. HP's existence and specificity to ICIs remains controversial because a widely acknowledged definition is currently lacking. Meanwhile, management remains elusive.
METHODS
Medical records from all consecutive NSCLC patients who were treated with ICI from 2015 to 2018 were retrospectively analyzed. The HP incidence rate was calculated according to five definitions (tumor growth rate [TGR]ratio, ΔTGR, tumor growth kinetic [TGK], RECIST, and time to treatment failure [TTF]), and the agreement between such definitions was determined. The HP impact on overall survival (OS) was then assessed. The association between HP (defined using the TGRratio definition) and clinical and biological variables was also assessed. Clinical HP management and its impact on outcomes were described.
RESULTS
We identified 169 consecutive ICI-treated patients, with potential HP accounting for 11.3 %, 5.7 %, 17.0 %, 9.6 %, and 31.7 % patients, according to TGRratio, ΔTGR, TGK, RECIST, and TTF definitions. Agreement between the different HP definitions was highly heterogeneous (range 29 %-77 %) and globally poor. HP was associated with shorter OS, compared to standard RECIST progressive disease, but this difference only reached statistical significance when using the TTF definition. TGRratio-based HP was significantly associated with hepatic metastases. In TGRratio-based HP patients, neither resuming chemotherapy nor corticosteroids use was associated with statistically significant impact on overall survival.
CONCLUSION
We found fairly heterogeneous HP rates using different definitions. TTF was the only definition leading to significantly worsened OS. Further studies are needed to provide consensus recommendations for the assessment, definition, and management of HP, whose existence is likely real.

Identifiants

pubmed: 33385736
pii: S0169-5002(20)30754-6
doi: 10.1016/j.lungcan.2020.12.026
pii:
doi:

Substances chimiques

B7-H1 Antigen 0
Immune Checkpoint Inhibitors 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

109-118

Informations de copyright

Copyright © 2020. Published by Elsevier B.V.

Auteurs

B Abbar (B)

Department of Thoracic Oncology, Bichat-Claude Bernard Hospital, AP-HP, Sorbonne University, Paris, France.

V De Castelbajac (V)

Breast Disease Center, Saint Louis Hospital, AP-HP, Université de Paris, Paris, France.

P Gougis (P)

Department of Pharmacology, Department of Medical Oncology, Pitié-Salpêtrière Hospital, AP-HP, Sorbonne University, INSERM, CIC-1901 Paris-Est, CLIP(2)Galilée, Paris, France.

S Assoun (S)

Department of Thoracic Oncology, CIC 1425 INSERM, Bichat-Claude Bernard Hospital, AP-HP, Université de Paris, Paris, France.

J Pluvy (J)

Department of Thoracic Oncology, CIC 1425 INSERM, Bichat-Claude Bernard Hospital, AP-HP, Université de Paris, Paris, France.

C Tesmoingt (C)

Department of Clinical Pharmacy, Bichat-Claude Bernard Hospital, AP-HP, Paris, France.

N Théou-Anton (N)

Department of Genetics, Bichat-Claude Bernard Hospital, AP-HP, Paris, France.

A Cazes (A)

Department of Pathology, Bichat-Claude Bernard Hospital, Université de Paris, AP-HP, Paris, France.

C Namour (C)

Department of Thoracic Oncology, CIC 1425 INSERM, Bichat-Claude Bernard Hospital, AP-HP, Université de Paris, Paris, France.

A Khalil (A)

Department of Radiology, University Hospital Bichat-Claude Bernard, AP-HP, Paris, France.

V Gounant (V)

Department of Thoracic Oncology, CIC 1425 INSERM, Bichat-Claude Bernard Hospital, AP-HP, Université de Paris, Paris, France.

B Besse (B)

Department of Cancer Medicine, Institut Gustave Roussy, Saclay University Villejuif, France.

G Zalcman (G)

Department of Thoracic Oncology, CIC 1425 INSERM, Bichat-Claude Bernard Hospital, AP-HP, Université de Paris, Paris, France. Electronic address: gerard.zalcman@aphp.fr.

S Brosseau (S)

Department of Thoracic Oncology, CIC 1425 INSERM, Bichat-Claude Bernard Hospital, AP-HP, Université de Paris, Paris, France.

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Classifications MeSH