Programmed cell death 1 (PDCD1) gene haplotypes and susceptibility of patients to basal cell carcinoma.


Journal

Molecular biology reports
ISSN: 1573-4978
Titre abrégé: Mol Biol Rep
Pays: Netherlands
ID NLM: 0403234

Informations de publication

Date de publication:
Mar 2021
Historique:
received: 27 09 2020
accepted: 20 12 2020
pubmed: 4 1 2021
medline: 20 5 2021
entrez: 3 1 2021
Statut: ppublish

Résumé

Programmed death-1 (PD-1), as an immunoinhibitory receptor encoded by programmed cell death-1 (PDCD1) gene, has a pivotal role in tolerance to self-antigens. Mutations of PDCD1 may participate in susceptibility to basal cell carcinoma (BCC) as the most common of skin cancer. We studied the impacts of two single nucleotide polymorphisms (SNPs) within PDCD1 and their haplotypes in BCC susceptibility in an Iranian population. The blood samples were collected from 210 BCC and 220 healthy individuals. After the extraction of genomic DNA, the genotypes and alleles of PD1.1 G/A (rs36084323) and PD1.6 G/A (rs10204525) SNPs were determined by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). Four haplotypes were estimated by these SNPs. Our data revealed that genotype and allele frequencies of PD1.1 and PD1.6 polymorphisms in BCC patients were similar to those in healthy individuals. The results of estimated haplotypes for PDCD1 indicated that GG and AA haplotypes of PDCD1 had protective effects on BCC susceptibility (OR = 0.7, 95% CI = 0.51-0.96, p = 0.03 and OR = 0.57, 95% CI = 0.35-0.91, p = 0.02, respectively), while GA and AG haplotypes served as the risk factors for developing BCC (OR = 1.76, 95% CI = 1.09-2.84, p = 0.02 and OR = 3.87, 95% CI = 1.95-7.69, p = <0.001, respectively). Based on these findings, frequency distributions of PDCD1 haplotypes have important roles in the determination of BCC development in the Iranian population. However, larger multicenter studies are required to confirm this conclusion.

Identifiants

pubmed: 33389528
doi: 10.1007/s11033-020-06115-w
pii: 10.1007/s11033-020-06115-w
doi:

Substances chimiques

PDCD1 protein, human 0
Programmed Cell Death 1 Receptor 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2047-2052

Subventions

Organisme : Kashan University of Medical Sciences
ID : 98019

Commentaires et corrections

Type : ErratumIn

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Auteurs

Farshid Fathi (F)

Department of Immunology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.

Batool Zamani (B)

Autoimmune Diseases Research Center, Kashan University of Medical Sciences, Kashan, Iran.

Ahmad Piroozmand (A)

Autoimmune Diseases Research Center, Kashan University of Medical Sciences, Kashan, Iran.
Department of Microbiology, Faculty of Medicine, Kashan University of Medical Sciences, Kashan, Iran.

Samaneh Mozafarpoor (S)

Department of Dermatology, Isfahan University of Medical Sciences, Isfahan, Iran.

Effat Seyedhashemi (E)

Department of Genetics, Faculty of Advanced Science and Technology, Tehran Medical Sciences, Islamic Azad University, Tehran, Iran.

Reza ArefNezhad (R)

Department of Anatomy, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.

Hossein Motedayyen (H)

Autoimmune Diseases Research Center, Shahid Beheshti Hospital, Kashan University of Medical Sciences, 5th kilometer of Ravand Road, Kashan, Iran. hmotedayyen@gmail.com.

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