TARM1 contributes to development of arthritis by activating dendritic cells through recognition of collagens.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
04 01 2021
Historique:
received: 11 07 2019
accepted: 20 11 2020
entrez: 5 1 2021
pubmed: 6 1 2021
medline: 15 1 2021
Statut: epublish

Résumé

TARM1 is a member of the leukocyte immunoglobulin-like receptor family and stimulates macrophages and neutrophils in vitro by associating with FcRγ. However, the function of this molecule in the regulation of the immune system is unclear. Here, we show that Tarm1 expression is elevated in the joints of rheumatoid arthritis mouse models, and the development of collagen-induced arthritis (CIA) is suppressed in Tarm1

Identifiants

pubmed: 33397982
doi: 10.1038/s41467-020-20307-9
pii: 10.1038/s41467-020-20307-9
pmc: PMC7782728
doi:

Substances chimiques

Ligands 0
Receptors, Immunologic 0
TARM1 protein, mouse 0
enhanced green fluorescent protein 0
Green Fluorescent Proteins 147336-22-9
Granulocyte-Macrophage Colony-Stimulating Factor 83869-56-1
Collagen 9007-34-5

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

94

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Auteurs

Rikio Yabe (R)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.
Medical Mycobiology Research Center, Chiba University, Chiba, Chiba, 260-8673, Japan.

Soo-Hyun Chung (SH)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Masanori A Murayama (MA)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Sachiko Kubo (S)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Kenji Shimizu (K)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Yukiko Akahori (Y)

Medical Mycobiology Research Center, Chiba University, Chiba, Chiba, 260-8673, Japan.

Takumi Maruhashi (T)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Akimasa Seno (A)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Tomonori Kaifu (T)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan.

Shinobu Saijo (S)

Medical Mycobiology Research Center, Chiba University, Chiba, Chiba, 260-8673, Japan. saijo@faculty.chiba-u.jp.

Yoichiro Iwakura (Y)

Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba, 278-0022, Japan. iwakura@rs.tus.ac.jp.
Medical Mycobiology Research Center, Chiba University, Chiba, Chiba, 260-8673, Japan. iwakura@rs.tus.ac.jp.

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