Risk markers of incident atrial fibrillation in patients with coronary heart disease.


Journal

American heart journal
ISSN: 1097-6744
Titre abrégé: Am Heart J
Pays: United States
ID NLM: 0370465

Informations de publication

Date de publication:
03 2021
Historique:
received: 17 09 2020
accepted: 28 12 2020
pubmed: 6 1 2021
medline: 19 3 2021
entrez: 5 1 2021
Statut: ppublish

Résumé

In patients with coronary heart disease (CHD), atrial fibrillation (AF) is associated with increased morbidity and mortality. We investigated the associations between clinical risk factors and biomarkers with incident AF in patients with CHD. Around 13,153 patients with optimally treated CHD included in the STabilization of Atherosclerotic plaque By Initiation of darapLadIb TherapY (STABILITY) trial with plasma samples obtained at randomization. Mean follow-up time was 3.5 years. The association between clinical risk factors and biomarkers with incident AF was estimated with Cox-regression models. Validation was performed in 1,894 patients with non-ST-elevation acute coronary syndrome included in the FRISC-II trial. The median (min-max) age was 64 years (range 26-92) and 2,514 (19.1%) were women. A total of 541 patients, annual incidence rate of 1.2%, developed AF during follow-up. In multivariable models, older age, higher levels of NT-proBNP, higher body mass index (BMI), male sex, geographic regions, low physical activity, and heart failure were independently associated with increased risk of incident AF with hazard ratios ranging from 1.04 to 1.79 (P ≤ .05). NT-proBNP improved the C-index from 0.70 to 0.71. In the validation cohort, age, BMI, and NT-proBNP were associated with increased risk of incident AF with similar hazard ratios. In patients with optimally treated CHD, the incidence of new AF was 1.2% per year. Age, NT-proBNP as a marker of impaired cardiac function, and BMI were the strongest factors, independently and consistently associated with incident AF. Male sex and low physical activity may also contribute to the risk of AF in patients with CHD.

Sections du résumé

BACKGROUND
In patients with coronary heart disease (CHD), atrial fibrillation (AF) is associated with increased morbidity and mortality. We investigated the associations between clinical risk factors and biomarkers with incident AF in patients with CHD.
METHODS AND RESULTS
Around 13,153 patients with optimally treated CHD included in the STabilization of Atherosclerotic plaque By Initiation of darapLadIb TherapY (STABILITY) trial with plasma samples obtained at randomization. Mean follow-up time was 3.5 years. The association between clinical risk factors and biomarkers with incident AF was estimated with Cox-regression models. Validation was performed in 1,894 patients with non-ST-elevation acute coronary syndrome included in the FRISC-II trial. The median (min-max) age was 64 years (range 26-92) and 2,514 (19.1%) were women. A total of 541 patients, annual incidence rate of 1.2%, developed AF during follow-up. In multivariable models, older age, higher levels of NT-proBNP, higher body mass index (BMI), male sex, geographic regions, low physical activity, and heart failure were independently associated with increased risk of incident AF with hazard ratios ranging from 1.04 to 1.79 (P ≤ .05). NT-proBNP improved the C-index from 0.70 to 0.71. In the validation cohort, age, BMI, and NT-proBNP were associated with increased risk of incident AF with similar hazard ratios.
CONCLUSIONS
In patients with optimally treated CHD, the incidence of new AF was 1.2% per year. Age, NT-proBNP as a marker of impaired cardiac function, and BMI were the strongest factors, independently and consistently associated with incident AF. Male sex and low physical activity may also contribute to the risk of AF in patients with CHD.

Identifiants

pubmed: 33400910
pii: S0002-8703(20)30425-7
doi: 10.1016/j.ahj.2020.12.016
pii:
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

92-101

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2020 The Author(s). Published by Elsevier Inc. All rights reserved.

Auteurs

Maria Tomasdottir (M)

Department of Medical Sciences, Cardiology, Uppsala University, Sweden.

Claes Held (C)

Department of Medical Sciences, Cardiology, Uppsala University, Sweden; Uppsala Clinical Research Center, Uppsala University, Sweden.

Nermin Hadziosmanovic (N)

Uppsala Clinical Research Center, Uppsala University, Sweden.

Johan Westerbergh (J)

Uppsala Clinical Research Center, Uppsala University, Sweden.

Johan Lindbäck (J)

Uppsala Clinical Research Center, Uppsala University, Sweden.

Philip E Aylward (PE)

South Australian Health and Medical Research Institute, Flinders University and Medical Centre, Adelaide, SA, Australia.

Andrzej Budaj (A)

Centre of Postgraduate Medical Education, Grochowski Hospital, Warsaw, Poland.

Christopher P Cannon (CP)

Cardiovascular Innovation, Brigham and Women's Hospital, Boston, MA.

Johan Engdahl (J)

Department of Clinical Sciences, Karolinska Institutet, Danderyd University Hospital, Stockholm, Sweden.

Christopher B Granger (CB)

Duke Clinical Research Institute, Duke Medicine, Durham, NC.

Wolfgang Koenig (W)

Deutsches Herzzentrum München, Technische Universität München, Munich, Germany; DZHK (German Centre for Cardiovascular Research), Partner Site Munich Heart Alliance, Munich, Germany; Institute of Epidemiology and Medical Biometry, University of Ulm, Ulm, Germany.

Athanasios J Manolis (AJ)

Cardiology Department, Asklepeion Hospital, Athens, Greece.

Jonas Oldgren (J)

Department of Medical Sciences, Cardiology, Uppsala University, Sweden; Uppsala Clinical Research Center, Uppsala University, Sweden.

Ralph A H Stewart (RAH)

Green Lane Cardiovascular Service, Auckland City Hospital and University of Auckland, Auckland, New Zealand.

Emma Svennberg (E)

Department of Clinical Sciences, Karolinska Institutet, Danderyd University Hospital, Stockholm, Sweden; Department of Cardiology, Karolinska Hospital, Stockholm, Sweden.

Dragos Vinereanu (D)

University of Medicine and Pharmacy Carol Davila, University and Emergency Hospital, Bucharest, Romania.

Harvey D White (HD)

Green Lane Cardiovascular Service, Auckland City Hospital and University of Auckland, Auckland, New Zealand.

Agneta Siegbahn (A)

Uppsala Clinical Research Center, Uppsala University, Sweden; Department of Medical Sciences, Clinical Chemistry, Uppsala University, Uppsala, Sweden.

Lars Wallentin (L)

Department of Medical Sciences, Cardiology, Uppsala University, Sweden; Uppsala Clinical Research Center, Uppsala University, Sweden.

Ziad Hijazi (Z)

Department of Medical Sciences, Cardiology, Uppsala University, Sweden; Uppsala Clinical Research Center, Uppsala University, Sweden. Electronic address: Ziad.Hijazi@ucr.uu.se.

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