The chemokine CXCL14 is negatively associated with obesity and concomitant type-2 diabetes in humans.


Journal

International journal of obesity (2005)
ISSN: 1476-5497
Titre abrégé: Int J Obes (Lond)
Pays: England
ID NLM: 101256108

Informations de publication

Date de publication:
03 2021
Historique:
received: 10 08 2020
accepted: 09 12 2020
revised: 23 10 2020
pubmed: 9 1 2021
medline: 27 1 2022
entrez: 8 1 2021
Statut: ppublish

Résumé

Chemokine (C-X-C motif) ligand-14 (CXCL14) levels are downregulated in experimental rodent models of obesity. Moreover, CXCL14 reportedly favors insulin sensitization in obese mice. Here we examined, for the first time, the role of CXCL14 in human obesity. We found that circulating levels of CXCL14 were decreased in patients with obesity and, especially, those with concomitant type-2 diabetes. CXCL14 levels were negatively associated with BMI and with indices of impaired glucose/insulin homeostasis. CXCL14 expression was decreased in subcutaneous adipose tissue from patients with obesity and type-2 diabetes. In adipose tissue, CXCL14 expression was negatively correlated with the expression of genes encoding pro-inflammatory molecules, and positively correlated with GLUT4 and adiponectin expression. In conclusion, obesity, and especially, concomitant type-2 diabetes are associated with abnormally decreased levels of CXCL14 in blood and impaired CXCL14 expression in adipose tissue. CXCL14 downregulation may be a novel biomarker of altered metabolism in obesity. CXCL14 also deserves further research as a therapeutic candidate.

Identifiants

pubmed: 33414488
doi: 10.1038/s41366-020-00732-y
pii: 10.1038/s41366-020-00732-y
doi:

Substances chimiques

CXCL14 protein, human 0
Chemokines, CXC 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

706-710

Références

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Auteurs

Rubén Cereijo (R)

Departament de Bioquímica i Biomedicina Molecular and Institut de Biomedicina (IBUB), Universitat de Barcelona, Barcelona, Catalonia, Spain.
Infectious Diseases Unit, Hospital de la Santa Creu i Sant Pau, Universitat Autònoma de Barcelona, Barcelona, Catalonia, Spain.
CIBER Fisiopatología de la Obesidad y Nutrición, Madrid, Spain.

Tania Quesada-López (T)

Departament de Bioquímica i Biomedicina Molecular and Institut de Biomedicina (IBUB), Universitat de Barcelona, Barcelona, Catalonia, Spain.
CIBER Fisiopatología de la Obesidad y Nutrición, Madrid, Spain.

Aleix Gavaldà-Navarro (A)

Departament de Bioquímica i Biomedicina Molecular and Institut de Biomedicina (IBUB), Universitat de Barcelona, Barcelona, Catalonia, Spain.
CIBER Fisiopatología de la Obesidad y Nutrición, Madrid, Spain.

Jordi Tarascó (J)

Germans Trias i Pujol Research Institute, Barcelona, Catalonia, Spain.

Silvia Pellitero (S)

Germans Trias i Pujol Research Institute, Barcelona, Catalonia, Spain.

Marjorie Reyes (M)

Germans Trias i Pujol Research Institute, Barcelona, Catalonia, Spain.

Manel Puig-Domingo (M)

Germans Trias i Pujol Research Institute, Barcelona, Catalonia, Spain.
CIBER Diabetes y Enfermedades Metabólicas Asociadas, Madrid, Spain.

Marta Giralt (M)

Departament de Bioquímica i Biomedicina Molecular and Institut de Biomedicina (IBUB), Universitat de Barcelona, Barcelona, Catalonia, Spain.
CIBER Fisiopatología de la Obesidad y Nutrición, Madrid, Spain.

David Sánchez-Infantes (D)

CIBER Fisiopatología de la Obesidad y Nutrición, Madrid, Spain. dsanchez@igtp.cat.
Germans Trias i Pujol Research Institute, Barcelona, Catalonia, Spain. dsanchez@igtp.cat.

Francesc Villarroya (F)

Departament de Bioquímica i Biomedicina Molecular and Institut de Biomedicina (IBUB), Universitat de Barcelona, Barcelona, Catalonia, Spain. fvillarroya@ub.edu.
CIBER Fisiopatología de la Obesidad y Nutrición, Madrid, Spain. fvillarroya@ub.edu.

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