A Novel Gene Delivery Vector of Agonistic Anti-Radioprotective 105 Expressed on Cell Membranes Shows Adjuvant Effect for DNA Immunization Against Influenza.


Journal

Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960

Informations de publication

Date de publication:
2020
Historique:
received: 15 09 2020
accepted: 18 11 2020
entrez: 8 1 2021
pubmed: 9 1 2021
medline: 22 6 2021
Statut: epublish

Résumé

Radioprotective 105 (RP105) (also termed CD180) is an orphan and unconventional Toll-like receptor (TLR) that lacks an intracellular signaling domain. The agonistic anti-RP105 monoclonal antibody (mAb) can cross-link RP105 on B cells, resulting in the proliferation and activation of B cells. Anti-RP105 mAb also has a potent adjuvant effect, providing higher levels of antigen-specific antibodies compared to alum. However, adjuvanticity is required for the covalent link between anti-RP105 mAb and the antigen. This is a possible obstacle to immunization due to the link between anti-RP105 mAb and some antigens, especially multi-transmembrane proteins. We have previously succeeded in inducing rapid and potent recombinant mAbs in mice using antibody gene-based delivery. To simplify the covalent link between anti-RP105 mAb and antigens, we generated genetic constructs of recombinant anti-RP105 mAb (αRP105) bound to the transmembrane domain of the IgG-B cell receptor (TM) (αRP105-TM), which could enable the anti-RP105 mAb to link the antigen

Identifiants

pubmed: 33414788
doi: 10.3389/fimmu.2020.606518
pmc: PMC7783388
doi:

Substances chimiques

Adjuvants, Immunologic 0
Antibodies, Monoclonal 0
Antigens, CD 0
Antigens, Surface 0
H1N1 virus hemagglutinin 0
Hemagglutinin Glycoproteins, Influenza Virus 0
Influenza Vaccines 0
Ly78 protein, mouse 0
Ly86 protein, mouse 0
Membrane Glycoproteins 0
Receptors, IgG 0
Vaccines, DNA 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

606518

Informations de copyright

Copyright © 2020 Yamazaki, Biswas, Kosugi, Nagashima, Inui, Tomono, Takagi, Ichimonji, Nagaoka, Ainai, Hasegawa, Chiba and Akashi-Takamura.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Tatsuya Yamazaki (T)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Mrityunjoy Biswas (M)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Kouyu Kosugi (K)

Department of Biological Science and Technology, Tokyo University of Science, Tokyo, Japan.

Maria Nagashima (M)

Department of Biological Science and Technology, Tokyo University of Science, Tokyo, Japan.

Masanori Inui (M)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Susumu Tomono (S)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Hidekazu Takagi (H)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Isao Ichimonji (I)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Fumiaki Nagaoka (F)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

Akira Ainai (A)

Department of Pathology, National Institute of Infectious Diseases, Tokyo, Japan.

Hideki Hasegawa (H)

Department of Pathology, National Institute of Infectious Diseases, Tokyo, Japan.

Joe Chiba (J)

Department of Biological Science and Technology, Tokyo University of Science, Tokyo, Japan.

Sachiko Akashi-Takamura (S)

Department of Microbiology and Immunology, School of Medicine, Aichi Medical University, Aichi, Japan.

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Classifications MeSH