Identification of protein/mRNA network involving the PSORS1 locus gene CCHCR1 and the PSORS4 locus gene HAX1.


Journal

Experimental cell research
ISSN: 1090-2422
Titre abrégé: Exp Cell Res
Pays: United States
ID NLM: 0373226

Informations de publication

Date de publication:
15 02 2021
Historique:
received: 05 06 2020
revised: 22 12 2020
accepted: 30 12 2020
pubmed: 9 1 2021
medline: 12 6 2021
entrez: 8 1 2021
Statut: ppublish

Résumé

CCHCR1 (Coiled-Coil alpha-Helical Rod 1), maps to chromosomal region 6p21.3, within the major psoriasis susceptibility locus PSORS1. CCHCR1 itself is a plausible psoriasis candidate gene, however its role in psoriasis pathogenesis remains unclear. We previously demonstrated that CCHCR1 protein acts as a cytoplasmic docking site for RNA polymerase II core subunit 3 (RPB3) in cycling cells, suggesting a role for CCHCR1 in vesicular trafficking between cellular compartments. Here, we report a novel interaction between CCHCR1 and the RNA binding protein HAX1. HAX1 maps to chromosomal region 1q21.3 within the PSORS4 locus and is over-expressed in psoriasis. Both CCHCR1 and HAX1 share subcellular co-localization with mitochondria, nuclei and cytoplasmic vesicles as P-bodies. By a series of ribonucleoprotein immunoprecipitation (RIP) assays, we isolated a pool of mRNAs complexed with HAX1 and/or CCHCR1 proteins. Among the mRNAs complexed with both CCHCR1 and HAX1 proteins, there are Vimentin mRNA, previously described to be bound by HAX1, and CAMP/LL37 mRNA, whose gene product is over-expressed in psoriasis.

Identifiants

pubmed: 33417922
pii: S0014-4827(21)00002-1
doi: 10.1016/j.yexcr.2021.112471
pii:
doi:

Substances chimiques

Adaptor Proteins, Signal Transducing 0
CCHCR1 protein, human 0
HAX1 protein, human 0
Intracellular Signaling Peptides and Proteins 0
RNA, Messenger 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

112471

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Auteurs

Cinzia Pisani (C)

CNR-Institute of Molecular Biology and Pathology, Department of Molecular Medicine, Sapienza University of Rome, Italy. Electronic address: cinzia.pisani@cnr.it.

Annalisa Onori (A)

CNR-Institute of Molecular Biology and Pathology, Department of Molecular Medicine, Sapienza University of Rome, Italy. Electronic address: annalisa.onori@cnr.it.

Francesca Gabanella (F)

CNR-Institute of Molecular Biology and Pathology, Department of Molecular Medicine, Sapienza University of Rome, Italy; CNR-Institute of Biochemistry and Cell Biology, Department of Sense Organs, Sapienza University of Rome, Italy. Electronic address: francesca.gabanella@gmail.com.

Maria Grazia Di Certo (MG)

CNR-Institute of Biochemistry and Cell Biology, Department of Sense Organs, Sapienza University of Rome, Italy. Electronic address: mariagrazia.dicerto@cnr.it.

Claudio Passananti (C)

CNR-Institute of Molecular Biology and Pathology, Department of Molecular Medicine, Sapienza University of Rome, Italy. Electronic address: claudio.passananti@cnr.it.

Nicoletta Corbi (N)

CNR-Institute of Molecular Biology and Pathology, Department of Molecular Medicine, Sapienza University of Rome, Italy. Electronic address: nicoletta.corbi@cnr.it.

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Classifications MeSH