Independent and cumulative coeliac disease-susceptibility loci are associated with distinct disease phenotypes.


Journal

Journal of human genetics
ISSN: 1435-232X
Titre abrégé: J Hum Genet
Pays: England
ID NLM: 9808008

Informations de publication

Date de publication:
Jun 2021
Historique:
received: 30 06 2020
accepted: 01 12 2020
revised: 01 12 2020
pubmed: 16 1 2021
medline: 3 9 2021
entrez: 15 1 2021
Statut: ppublish

Résumé

The phenotype of coeliac disease varies considerably for incompletely understood reasons. We investigated whether established coeliac disease susceptibility variants (SNPs) are individually or cumulatively associated with distinct phenotypes. We also tested whether a polygenic risk score (PRS) based on genome-wide associated (GWA) data could explain the phenotypic variation. The phenotypic association of 39 non-HLA coeliac disease SNPs was tested in 625 thoroughly phenotyped coeliac disease patients and 1817 controls. To assess their cumulative effects a weighted genetic risk score (wGRS39) was built, and stratified by tertiles. In our PRS model in cases, we took the summary statistics from the largest GWA study in coeliac disease and tested their association at eight P value thresholds (P

Identifiants

pubmed: 33446885
doi: 10.1038/s10038-020-00888-5
pii: 10.1038/s10038-020-00888-5
pmc: PMC8144013
doi:

Substances chimiques

ATXN2 protein, human 0
Adaptor Proteins, Signal Transducing 0
Ataxin-2 0
SH2B3 protein, human 0
TLR7 protein, human 0
TLR8 protein, human 0
Toll-Like Receptor 7 0
Toll-Like Receptor 8 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

613-623

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Auteurs

Juliana X M Cerqueira (JXM)

Coeliac Disease Research Center, Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland.

Päivi Saavalainen (P)

Research Programs Unit, Immunobiology, and the Haartman Institute, Department of Molecular Genetics, University of Helsinki, Helsinki, Finland.

Kalle Kurppa (K)

Center for Child Health Research, Tampere University and Tampere University Hospital, Tampere, and the University Consortium of Seinäjoki, Seinäjoki, Finland.

Pilvi Laurikka (P)

Coeliac Disease Research Center, Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland.

Heini Huhtala (H)

Faculty of Social Sciences, Tampere University, Tampere, Finland.

Matti Nykter (M)

Laboratory of Computational Biology, Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland.

Lotta L E Koskinen (L)

Research Programs Unit, Immunobiology, and the Haartman Institute, Department of Molecular Genetics, University of Helsinki, Helsinki, Finland.

Dawit A Yohannes (DA)

Research Programs Unit, Immunobiology, and the Haartman Institute, Department of Molecular Genetics, University of Helsinki, Helsinki, Finland.

Elina Kilpeläinen (E)

Institute for Molecular Medicine Finland (FIMM), University of Helsinki, Helsinki, Finland.

Anastasia Shcherban (A)

Institute for Molecular Medicine Finland (FIMM), University of Helsinki, Helsinki, Finland.

Aarno Palotie (A)

Institute for Molecular Medicine Finland (FIMM), University of Helsinki, Helsinki, Finland.
Broad Institute of the Massachusetts Institute of Technology and Harvard University, Cambridge, MA, USA.
Psychiatric & Neurodevelopmental Genetics Unit, Department of Psychiatry, Analytic and Translational Genetics Unit, Department of Medicine, and the Department of Neurology, Massachusetts General Hospital, Boston, MA, USA.

Katri Kaukinen (K)

Coeliac Disease Research Center, Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland.
Department of Internal Medicine, Tampere University Hospital, Tampere, Finland.

Katri Lindfors (K)

Coeliac Disease Research Center, Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland. katri.lindfors@tuni.fi.

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Classifications MeSH