Long-term atmospheric exposure to PCB153 and breast cancer risk in a case-control study nested in the French E3N cohort from 1990 to 2011.


Journal

Environmental research
ISSN: 1096-0953
Titre abrégé: Environ Res
Pays: Netherlands
ID NLM: 0147621

Informations de publication

Date de publication:
04 2021
Historique:
received: 25 09 2020
revised: 18 12 2020
accepted: 08 01 2021
pubmed: 16 1 2021
medline: 22 4 2021
entrez: 15 1 2021
Statut: ppublish

Résumé

Although the genetic and hormonal risk factors of breast cancer are well identified, they cannot fully explain the occurrence of all cases. Epidemiological and experimental studies have suggested that exposure to environmental pollutants, especially those with potential estrogenic properties, as polychlorinated biphenyls (PCBs) may have a role in breast cancer development. Being the most abundantly detected in human tissues and in the environment, congener 153 (PCB153) is widely used in epidemiological studies as indicator for total PCBs exposure. We aimed to estimate the association between cumulative atmospheric exposure to PCB153 and breast cancer risk. We conducted a case-control study of 5222 cases and 5222 matched controls nested within the French E3N cohort from 1990 to 2011. Annual atmospheric PCB153 concentrations were simulated with the deterministic chemistry-transport model (CHIMERE) and were assigned to women using their geocoded residential history. Their cumulative PCB153 exposure was calculated for each woman from their cohort inclusion to their index date. Breast cancer odds ratios (ORs) associated with cumulative PCB153 exposure and their 95% confidence intervals (95% CIs) were estimated using multivariate conditional logistic regression models. Overall, our results showed a statistically significant linear increase in breast cancer risk related to cumulative atmospheric exposure to PCB153 as a continuous variable (adjusted OR = 1.19; 95% CI: 1.08-1.31, for an increment of one standard deviation among controls (55 pg/m This study is the first to have estimated the impact of atmospheric exposure to PCB153 on breast cancer risk. Our results showed a statistically significant increase in breast cancer risk, which may be limited to ER-positive breast cancer. These results warrant confirmation in further independent studies but raise the possibility that exposure to PCB153 increase breast cancer risk.

Sections du résumé

BACKGROUND
Although the genetic and hormonal risk factors of breast cancer are well identified, they cannot fully explain the occurrence of all cases. Epidemiological and experimental studies have suggested that exposure to environmental pollutants, especially those with potential estrogenic properties, as polychlorinated biphenyls (PCBs) may have a role in breast cancer development. Being the most abundantly detected in human tissues and in the environment, congener 153 (PCB153) is widely used in epidemiological studies as indicator for total PCBs exposure.
OBJECTIVES
We aimed to estimate the association between cumulative atmospheric exposure to PCB153 and breast cancer risk.
METHODS
We conducted a case-control study of 5222 cases and 5222 matched controls nested within the French E3N cohort from 1990 to 2011. Annual atmospheric PCB153 concentrations were simulated with the deterministic chemistry-transport model (CHIMERE) and were assigned to women using their geocoded residential history. Their cumulative PCB153 exposure was calculated for each woman from their cohort inclusion to their index date. Breast cancer odds ratios (ORs) associated with cumulative PCB153 exposure and their 95% confidence intervals (95% CIs) were estimated using multivariate conditional logistic regression models.
RESULTS
Overall, our results showed a statistically significant linear increase in breast cancer risk related to cumulative atmospheric exposure to PCB153 as a continuous variable (adjusted OR = 1.19; 95% CI: 1.08-1.31, for an increment of one standard deviation among controls (55 pg/m
DISCUSSION
This study is the first to have estimated the impact of atmospheric exposure to PCB153 on breast cancer risk. Our results showed a statistically significant increase in breast cancer risk, which may be limited to ER-positive breast cancer. These results warrant confirmation in further independent studies but raise the possibility that exposure to PCB153 increase breast cancer risk.

Identifiants

pubmed: 33450235
pii: S0013-9351(21)00037-2
doi: 10.1016/j.envres.2021.110743
pii:
doi:

Substances chimiques

Polychlorinated Biphenyls DFC2HB4I0K

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

110743

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Auteurs

Floriane Deygas (F)

Department of Prevention, Cancer and Environment, Centre Léon Bérard, Lyon, France; Inserm U1296, "Radiation: Defense, Health and Environment", Centre Léon Bérard, Lyon, France.

Amina Amadou (A)

Department of Prevention, Cancer and Environment, Centre Léon Bérard, Lyon, France; Inserm U1296, "Radiation: Defense, Health and Environment", Centre Léon Bérard, Lyon, France.

Thomas Coudon (T)

Department of Prevention, Cancer and Environment, Centre Léon Bérard, Lyon, France; Inserm U1296, "Radiation: Defense, Health and Environment", Centre Léon Bérard, Lyon, France; Ecole Centrale de Lyon, University Claude Bernard Lyon 1, Ecully, France.

Lény Grassot (L)

Department of Prevention, Cancer and Environment, Centre Léon Bérard, Lyon, France; Inserm U1296, "Radiation: Defense, Health and Environment", Centre Léon Bérard, Lyon, France.

Florian Couvidat (F)

National Institute for Industrial Environment and Risks (INERIS), Verneuil-en-Halatte, France.

Bertrand Bessagnet (B)

National Institute for Industrial Environment and Risks (INERIS), Verneuil-en-Halatte, France; Citepa, Technical Reference Center for Air Pollution and Climate Change, Paris, France.

Elodie Faure (E)

Inserm U1018, Centre for Research in Epidemiology and Population Health (CESP), Team "Exposome and Heredity", Gustave Roussy, Villejuif, France.

Pietro Salizzoni (P)

Ecole Centrale de Lyon, University Claude Bernard Lyon 1, Ecully, France.

John Gulliver (J)

Centre for Environmental Health and Sustainability, School of Geography, Geology and the Environment, University of Leicester, Leicester, United Kingdom.

Julien Caudeville (J)

National Institute for Industrial Environment and Risks (INERIS), Verneuil-en-Halatte, France.

Gianluca Severi (G)

Inserm U1018, Centre for Research in Epidemiology and Population Health (CESP), Team "Exposome and Heredity", Gustave Roussy, Villejuif, France; Department of Statistics, Computer Science and Applications "G. Parenti", University of Florence, Italy.

Francesca Romana Mancini (FR)

Inserm U1018, Centre for Research in Epidemiology and Population Health (CESP), Team "Exposome and Heredity", Gustave Roussy, Villejuif, France. Electronic address: francesca.mancini@gustaveroussy.fr.

Karen Leffondré (K)

University of Bordeaux, ISPED, Inserm U1219, Bordeaux Population Health Center, Bordeaux, France.

Béatrice Fervers (B)

Department of Prevention, Cancer and Environment, Centre Léon Bérard, Lyon, France; Inserm U1296, "Radiation: Defense, Health and Environment", Centre Léon Bérard, Lyon, France. Electronic address: beatrice.fervers@lyon.unicancer.fr.

Delphine Praud (D)

Department of Prevention, Cancer and Environment, Centre Léon Bérard, Lyon, France; Inserm U1296, "Radiation: Defense, Health and Environment", Centre Léon Bérard, Lyon, France.

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