Recovery of balance and gait after stroke is deteriorated by confluent white matter hyperintensities: Cohort study.


Journal

Annals of physical and rehabilitation medicine
ISSN: 1877-0665
Titre abrégé: Ann Phys Rehabil Med
Pays: Netherlands
ID NLM: 101502773

Informations de publication

Date de publication:
Jan 2022
Historique:
received: 04 09 2020
revised: 23 12 2020
accepted: 27 12 2020
pubmed: 16 1 2021
medline: 16 2 2022
entrez: 15 1 2021
Statut: ppublish

Résumé

White matter hyperintensities (WMHs) are well known to affect post-stroke disability, mainly by cognitive impairment. Their impact on post-stroke balance and gait disorders is unclear. We aimed to test the hypothesis that WMHs would independently deteriorate post-stroke balance and gait recovery. This study was performed in 210 individuals of the cohort Determinants of Balance Recovery After Stroke (DOBRAS), consecutively enrolled after a first-ever hemisphere stroke. Clinical data were systematically collected on day 30±3 (D30) post-stroke and at discharge from the rehabilitation ward. WMHs were searched on MRI, graded with the Fazekas scale, and dichotomized as no/mild (absence/sparse) or moderate/severe (confluent). The primary endpoint was the recovery of the single limb stance, assessed with the Postural Assessment Scale for Stroke (PASS). The secondary endpoint was the recovery of independent gait, assessed with the modified Fugl-Meyer Gait Assessment (mFMA). The adjusted hazard ratios (aHRs) of achievements of these endpoints by level of WMHs were estimated by using Cox models, accounting for other relevant clinical and imaging factors. Individuals with moderate/severe WMHs (n=86, 41%) had greater balance and gait disorders and were more often fallers than others (n=124, 59%). Overall, they had worse and slower recovery of single limb stance and independent gait (P<0.001). Moderate/severe WMHs was the most detrimental factor for recovery of balance (aHR 0.46, 95% confidence interval [CI] 0.32-0.68, P<0.001) and gait (0.51, 0.35-0.74, P<0.001), along with age, stroke severity, lesion volume and disrupted corticospinal tract. With cerebral infarct, endovascular treatments had an independent positive effect, both on the recovery of balance (aHR 1.65, 95% CI 1.13-2.4, P=0.009) and gait (1.78, 1.24-2.55, P=0.002). WMHs magnify balance and gait disorders after stroke and worsen their recovery. They should be better accounted for in post-stroke rehabilitation, especially to help establish a prognosis of mobility. CLINICALTRIALS. NCT03203109.

Sections du résumé

BACKGROUND BACKGROUND
White matter hyperintensities (WMHs) are well known to affect post-stroke disability, mainly by cognitive impairment. Their impact on post-stroke balance and gait disorders is unclear.
OBJECTIVES OBJECTIVE
We aimed to test the hypothesis that WMHs would independently deteriorate post-stroke balance and gait recovery.
METHODS METHODS
This study was performed in 210 individuals of the cohort Determinants of Balance Recovery After Stroke (DOBRAS), consecutively enrolled after a first-ever hemisphere stroke. Clinical data were systematically collected on day 30±3 (D30) post-stroke and at discharge from the rehabilitation ward. WMHs were searched on MRI, graded with the Fazekas scale, and dichotomized as no/mild (absence/sparse) or moderate/severe (confluent). The primary endpoint was the recovery of the single limb stance, assessed with the Postural Assessment Scale for Stroke (PASS). The secondary endpoint was the recovery of independent gait, assessed with the modified Fugl-Meyer Gait Assessment (mFMA). The adjusted hazard ratios (aHRs) of achievements of these endpoints by level of WMHs were estimated by using Cox models, accounting for other relevant clinical and imaging factors.
RESULTS RESULTS
Individuals with moderate/severe WMHs (n=86, 41%) had greater balance and gait disorders and were more often fallers than others (n=124, 59%). Overall, they had worse and slower recovery of single limb stance and independent gait (P<0.001). Moderate/severe WMHs was the most detrimental factor for recovery of balance (aHR 0.46, 95% confidence interval [CI] 0.32-0.68, P<0.001) and gait (0.51, 0.35-0.74, P<0.001), along with age, stroke severity, lesion volume and disrupted corticospinal tract. With cerebral infarct, endovascular treatments had an independent positive effect, both on the recovery of balance (aHR 1.65, 95% CI 1.13-2.4, P=0.009) and gait (1.78, 1.24-2.55, P=0.002).
CONCLUSIONS CONCLUSIONS
WMHs magnify balance and gait disorders after stroke and worsen their recovery. They should be better accounted for in post-stroke rehabilitation, especially to help establish a prognosis of mobility. CLINICALTRIALS.
GOV REGISTRATION UNASSIGNED
NCT03203109.

Identifiants

pubmed: 33450367
pii: S1877-0657(21)00006-3
doi: 10.1016/j.rehab.2021.101488
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT03203109']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

101488

Informations de copyright

Copyright © 2021. Published by Elsevier Masson SAS.

Auteurs

Shenhao Dai (S)

Neurorehabilitation Department, Institute of Rehabilitation, Grenoble-Alpes University Hospital, 38434 Échirolles, France; Laboratoire de psychologie et neurocognition, UMR CNRS 5105, University Grenoble-Alpes, Grenoble, France.

Céline Piscicelli (C)

Neurorehabilitation Department, Institute of Rehabilitation, Grenoble-Alpes University Hospital, 38434 Échirolles, France; Laboratoire de psychologie et neurocognition, UMR CNRS 5105, University Grenoble-Alpes, Grenoble, France.

Camille Lemaire (C)

Neurorehabilitation Department, Institute of Rehabilitation, Grenoble-Alpes University Hospital, 38434 Échirolles, France; Laboratoire de psychologie et neurocognition, UMR CNRS 5105, University Grenoble-Alpes, Grenoble, France.

Adélie Christiaens (A)

Neurorehabilitation Department, Institute of Rehabilitation, Grenoble-Alpes University Hospital, 38434 Échirolles, France; Laboratoire de psychologie et neurocognition, UMR CNRS 5105, University Grenoble-Alpes, Grenoble, France.

Michel Thiebaut de Schotten (MT)

Brain Connectivity and Behaviour Laboratory, Sorbonne Universities, 75013 Paris, France; Groupe d'imagerie neurofonctionnelle, institut des maladies neurodégénératives-UMR 5293, CNRS, CEA University of Bordeaux, 33000 Bordeaux, France.

Marc Hommel (M)

Stroke Unit, Neurology Department, Grenoble-Alpes University Hospital, 38043 Grenoble, France; AGEIS EA 7407, University Grenoble-Alpes, Grenoble, France.

Alexandre Krainik (A)

Department of Neuroradiology, Grenoble-Alpes University Hospital, 38043 Grenoble, France; Inserm, U 1216, Grenoble, France; Grenoble Institute of Neurosciences, University Grenoble-Alpes, 38042 Grenoble, France; Inserm, CNRS, IRMaGe, Grenoble-Alpes University Hospital, University Grenoble-Alpes, 38043 Grenoble, France.

Olivier Detante (O)

Stroke Unit, Neurology Department, Grenoble-Alpes University Hospital, 38043 Grenoble, France; Grenoble Institute of Neurosciences, University Grenoble-Alpes, 38042 Grenoble, France.

Dominic Pérennou (D)

Neurorehabilitation Department, Institute of Rehabilitation, Grenoble-Alpes University Hospital, 38434 Échirolles, France; Laboratoire de psychologie et neurocognition, UMR CNRS 5105, University Grenoble-Alpes, Grenoble, France. Electronic address: dperennou@chu-grenoble.fr.

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