The Effect of Hypoxia on the Expression of CXC Chemokines and CXC Chemokine Receptors-A Review of Literature.
Chemokines, CXC
/ metabolism
Chemotactic Factors
/ metabolism
Cytokines
/ metabolism
Endothelial Cells
/ metabolism
Gene Expression Regulation, Neoplastic
Humans
Hypoxia
Hypoxia-Inducible Factor 1, alpha Subunit
/ metabolism
Inflammation
Microcirculation
NF-kappa B p50 Subunit
/ metabolism
Neoplasms
/ drug therapy
Receptors, CXCR
/ metabolism
CXC chemokine
HIF-1α
IL-8
NF-κB
SDF-1
cancer
cycling hypoxia
hypoxia
hypoxia-inducible factor
tumor
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
15 Jan 2021
15 Jan 2021
Historique:
received:
09
12
2020
revised:
06
01
2021
accepted:
12
01
2021
entrez:
20
1
2021
pubmed:
21
1
2021
medline:
24
4
2021
Statut:
epublish
Résumé
Hypoxia is an integral component of the tumor microenvironment. Either as chronic or cycling hypoxia, it exerts a similar effect on cancer processes by activating hypoxia-inducible factor-1 (HIF-1) and nuclear factor (NF-κB), with cycling hypoxia showing a stronger proinflammatory influence. One of the systems affected by hypoxia is the CXC chemokine system. This paper reviews all available information on hypoxia-induced changes in the expression of all CXC chemokines (CXCL1, CXCL2, CXCL3, CXCL4, CXCL5, CXCL6, CXCL7, CXCL8 (IL-8), CXCL9, CXCL10, CXCL11, CXCL12 (SDF-1), CXCL13, CXCL14, CXCL15, CXCL16, CXCL17) as well as CXC chemokine receptors-CXCR1, CXCR2, CXCR3, CXCR4, CXCR5, CXCR6, CXCR7 and CXCR8. First, we present basic information on the effect of these chemoattractant cytokines on cancer processes. We then discuss the effect of hypoxia-induced changes on CXC chemokine expression on the angiogenesis, lymphangiogenesis and recruitment of various cells to the tumor niche, including myeloid-derived suppressor cells (MDSCs), tumor-associated macrophages (TAMs), tumor-associated neutrophils (TANs), regulatory T cells (T
Identifiants
pubmed: 33467722
pii: ijms22020843
doi: 10.3390/ijms22020843
pmc: PMC7830156
pii:
doi:
Substances chimiques
Chemokines, CXC
0
Chemotactic Factors
0
Cytokines
0
HIF1A protein, human
0
Hypoxia-Inducible Factor 1, alpha Subunit
0
NF-kappa B p50 Subunit
0
NFKB1 protein, human
0
Receptors, CXCR
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
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