The transcriptional regulator Zfat is essential for maintenance and differentiation of the adipocytes.


Journal

Journal of cellular biochemistry
ISSN: 1097-4644
Titre abrégé: J Cell Biochem
Pays: United States
ID NLM: 8205768

Informations de publication

Date de publication:
06 2021
Historique:
revised: 22 12 2020
received: 09 06 2020
accepted: 23 12 2020
pubmed: 2 2 2021
medline: 5 10 2021
entrez: 1 2 2021
Statut: ppublish

Résumé

Adipocytes play crucial roles in the control of whole-body energy homeostasis. Differentiation and functions of the adipocytes are regulated by various transcription factors. Zfat (zinc-finger protein with AT-hook) is a transcriptional regulator that controls messenger RNA expression of specific genes through binding to their transcription start sites. Here we report important roles of Zfat in the adipocytes. We establish inducible Zfat-knockout (Zfat iKO) mice where treatment with tamoxifen causes a marked reduction in Zfat expression in various tissues. Tamoxifen treatment of Zfat iKO mice reduces the white adipose tissues (WATs) mass, accompanied by the decreased triglyceride levels. Zfat is expressed in both the adipose-derived stem cells (ADSCs) and mature adipocytes in the WATs. In ex vivo assays of the mature adipocytes differentiated from the Zfat iKO ADSCs, loss of Zfat in the mature adipocytes reduces the triglyceride levels, suggesting cell autonomous roles of Zfat in the maintenance of the mature adipocytes. Furthermore, we identify the Atg13, Brf1, Psmc3, and Timm22 genes as Zfat-target genes in the mature adipocytes. In contrast, loss of Zfat in the ADSCs impairs adipocyte differentiation with the decreased expression of C/EBPα and adiponectin. Thus, we propose that Zfat plays crucial roles in maintenance and differentiation of the adipocytes.

Identifiants

pubmed: 33522619
doi: 10.1002/jcb.29890
pmc: PMC8248092
doi:

Substances chimiques

Adiponectin 0
CCAAT-Enhancer-Binding Proteins 0
Transcription Factors 0
ZFAT protein, mouse 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

626-638

Informations de copyright

© 2021 The Authors. Journal of Cellular Biochemistry published by Wiley Periodicals LLC.

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Auteurs

Shuhei Ishikura (S)

Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.
Center for Advanced Molecular Medicine, Fukuoka University, Fukuoka, Japan.

Masayoshi Nagai (M)

Center for Advanced Molecular Medicine, Fukuoka University, Fukuoka, Japan.

Toshiyuki Tsunoda (T)

Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.
Center for Advanced Molecular Medicine, Fukuoka University, Fukuoka, Japan.

Kensuke Nishi (K)

Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.

Yoko Tanaka (Y)

Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.

Midori Koyanagi (M)

Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.

Senji Shirasawa (S)

Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.
Center for Advanced Molecular Medicine, Fukuoka University, Fukuoka, Japan.

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Classifications MeSH