Striped occipital cortex and intragyral hemorrhage: Novel magnetic resonance imaging markers for cerebral amyloid angiopathy.


Journal

International journal of stroke : official journal of the International Stroke Society
ISSN: 1747-4949
Titre abrégé: Int J Stroke
Pays: United States
ID NLM: 101274068

Informations de publication

Date de publication:
12 2021
Historique:
pubmed: 5 2 2021
medline: 7 1 2022
entrez: 4 2 2021
Statut: ppublish

Résumé

To investigate whether a striped occipital cortex and intragyral hemorrhage, two markers recently detected on ultra-high-field 7-tesla-magnetic resonance imaging in hereditary cerebral amyloid angiopathy (CAA), also occur in sporadic CAA (sCAA) or non-sCAA intracerebral hemorrhage (ICH). We performed 7-tesla-magnetic resonance imaging in patients with probable sCAA and patients with non-sCAA-ICH. Striped occipital cortex (linear hypointense stripes perpendicular to the cortex) and intragyral hemorrhage (hemorrhage restricted to the juxtacortical white matter of one gyrus) were scored on T We included 33 patients with sCAA (median age 70 years) and 29 patients with non-sCAA-ICH (median age 58 years). Striped occipital cortex was detected in one (3%) patient with severe sCAA. Five intragyral hemorrhages were found in four (12%) sCAA patients. The markers were absent in the non-sCAA-ICH group. Patients with intragyral hemorrhages had more lobar ICHs (median count 6.5 vs. 1.0), lobar microbleeds (median count >50 vs. 15), and lower median cognitive scores (Mini Mental State Exam: 20 vs. 28, Montreal Cognitive Assessment: 18 vs. 24) compared with patients with sCAA without intragyral hemorrhage. In 12 (36%) patients, sCAA diagnosis was changed to mixed-type small vessel disease due to deep bleeds previously unobserved on lower field-magnetic resonance imaging. Whereas a striped occipital cortex is rare in sCAA, 12% of patients with sCAA have intragyral hemorrhages. Intragyral hemorrhages seem to be related to advanced disease and their absence in patients with non-sCAA-ICH could suggest specificity for CAA.

Sections du résumé

BACKGROUND AND AIM
To investigate whether a striped occipital cortex and intragyral hemorrhage, two markers recently detected on ultra-high-field 7-tesla-magnetic resonance imaging in hereditary cerebral amyloid angiopathy (CAA), also occur in sporadic CAA (sCAA) or non-sCAA intracerebral hemorrhage (ICH).
METHODS
We performed 7-tesla-magnetic resonance imaging in patients with probable sCAA and patients with non-sCAA-ICH. Striped occipital cortex (linear hypointense stripes perpendicular to the cortex) and intragyral hemorrhage (hemorrhage restricted to the juxtacortical white matter of one gyrus) were scored on T
RESULTS
We included 33 patients with sCAA (median age 70 years) and 29 patients with non-sCAA-ICH (median age 58 years). Striped occipital cortex was detected in one (3%) patient with severe sCAA. Five intragyral hemorrhages were found in four (12%) sCAA patients. The markers were absent in the non-sCAA-ICH group. Patients with intragyral hemorrhages had more lobar ICHs (median count 6.5 vs. 1.0), lobar microbleeds (median count >50 vs. 15), and lower median cognitive scores (Mini Mental State Exam: 20 vs. 28, Montreal Cognitive Assessment: 18 vs. 24) compared with patients with sCAA without intragyral hemorrhage. In 12 (36%) patients, sCAA diagnosis was changed to mixed-type small vessel disease due to deep bleeds previously unobserved on lower field-magnetic resonance imaging.
CONCLUSION
Whereas a striped occipital cortex is rare in sCAA, 12% of patients with sCAA have intragyral hemorrhages. Intragyral hemorrhages seem to be related to advanced disease and their absence in patients with non-sCAA-ICH could suggest specificity for CAA.

Identifiants

pubmed: 33535905
doi: 10.1177/1747493021991961
pmc: PMC8669214
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1031-1038

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Auteurs

E A Koemans (EA)

Department of Neurology, 4501Leiden University Medical Center, Leiden, the Netherlands.

S Voigt (S)

Department of Neurology, 4501Leiden University Medical Center, Leiden, the Netherlands.

I Rasing (I)

Department of Neurology, 4501Leiden University Medical Center, Leiden, the Netherlands.

Wmt Jolink (W)

Department of Neurology and Neurosurgery, University Medical Center Utrecht Brain Center, Utrecht University, Utrecht, the Netherlands.

T W van Harten (TW)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

J van der Grond (J)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

S van Rooden (S)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

Fhbm Schreuder (F)

Department of Neurology, Donders Institute for Brain, Cognition and Behaviour, Radboud University Medical Center, Nijmegen, the Netherlands.

W M Freeze (WM)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

M A van Buchem (MA)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

E W van Zwet (EW)

Department of Biomedical Data Sciences, 4501Leiden University Medical Center, Leiden, the Netherlands.

S J van Veluw (SJ)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

G M Terwindt (GM)

Department of Neurology, 4501Leiden University Medical Center, Leiden, the Netherlands.

Mjp van Osch (M)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

Cjm Klijn (C)

Department of Neurology, Donders Institute for Brain, Cognition and Behaviour, Radboud University Medical Center, Nijmegen, the Netherlands.

Maa van Walderveen (M)

Department of Radiology, 4501Leiden University Medical Center, Leiden, the Netherlands.

Mjh Wermer (M)

Department of Neurology, 4501Leiden University Medical Center, Leiden, the Netherlands.

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