CD3

CCL17/TARC CD3(−)CD4(+) T cells Hypereosinophilic syndrome IL-5 Intracytoplasmic cytokines Lymphocytic variant T-cell phenotyping TCR gene rearrangement

Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
06 2021
Historique:
received: 07 10 2020
revised: 13 01 2021
accepted: 16 01 2021
pubmed: 6 2 2021
medline: 9 7 2021
entrez: 5 2 2021
Statut: ppublish

Résumé

Identification of patients with lymphocytic variant hypereosinophilic syndrome (L-HES) is challenging, and has important prognostic and therapeutic implications. This study was undertaken to assess diagnostic tools for L-HES and to develop evidence-based diagnostic recommendations. Biomarkers of T-cell-driven disease were compared between patients with L-HES versus idiopathic HES (I-HES) variants. Those performed routinely (serum immunoglobulin levels, T-cell phenotyping, T-cell receptor [TCR] gene rearrangement patterns) were collected from medical files, whereas others were prospectively assessed on stored blood samples (serum CCL17/thymus and activation regulated chemokine [TARC] levels, in vitro cytokine production). This study included 48 patients with I-HES and 20 with L-HES associated with a CD3 Adapting the standard of procedure for T-cell phenotyping in patients with unexplained hypereosinophilia is currently the most reliable means of identifying those with CD3

Sections du résumé

BACKGROUND
Identification of patients with lymphocytic variant hypereosinophilic syndrome (L-HES) is challenging, and has important prognostic and therapeutic implications.
OBJECTIVE
This study was undertaken to assess diagnostic tools for L-HES and to develop evidence-based diagnostic recommendations.
METHODS
Biomarkers of T-cell-driven disease were compared between patients with L-HES versus idiopathic HES (I-HES) variants. Those performed routinely (serum immunoglobulin levels, T-cell phenotyping, T-cell receptor [TCR] gene rearrangement patterns) were collected from medical files, whereas others were prospectively assessed on stored blood samples (serum CCL17/thymus and activation regulated chemokine [TARC] levels, in vitro cytokine production).
RESULTS
This study included 48 patients with I-HES and 20 with L-HES associated with a CD3
CONCLUSION
Adapting the standard of procedure for T-cell phenotyping in patients with unexplained hypereosinophilia is currently the most reliable means of identifying those with CD3

Identifiants

pubmed: 33545400
pii: S2213-2198(21)00158-6
doi: 10.1016/j.jaip.2021.01.030
pii:
doi:

Substances chimiques

CD3 Complex 0
Cytokines 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2426-2439.e7

Informations de copyright

Copyright © 2021 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

Auteurs

Caroline Carpentier (C)

Department of Internal Medicine, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.

Liliane Schandené (L)

Laboratory of Immunology, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.

Laurent Dewispelaere (L)

Department of Medical Genetics, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.

Pierre Heimann (P)

Department of Medical Genetics, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.

Elie Cogan (E)

Department of Internal Medicine, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.

Florence Roufosse (F)

Department of Internal Medicine, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium; Institute for Medical Immunology, Université Libre de Bruxelles, Gosselies, Belgium. Electronic address: froufoss@ulb.ac.be.

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Classifications MeSH