Potential APOBEC-mediated RNA editing of the genomes of SARS-CoV-2 and other coronaviruses and its impact on their longer term evolution.
APOBEC
Coronavirus
Innate immunity
SARS-CoV-2
Virus evolution
Journal
Virology
ISSN: 1096-0341
Titre abrégé: Virology
Pays: United States
ID NLM: 0110674
Informations de publication
Date de publication:
04 2021
04 2021
Historique:
received:
21
10
2020
revised:
21
12
2020
accepted:
21
12
2020
pubmed:
6
2
2021
medline:
13
3
2021
entrez:
5
2
2021
Statut:
ppublish
Résumé
Members of the APOBEC family of cytidine deaminases show antiviral activities in mammalian cells through lethal editing in the genomes of small DNA viruses, herpesviruses and retroviruses, and potentially those of RNA viruses such as coronaviruses. Consistent with the latter, APOBEC-like directional C→U transitions of genomic plus-strand RNA are greatly overrepresented in SARS-CoV-2 genome sequences of variants emerging during the COVID-19 pandemic. A C→U mutational process may leave evolutionary imprints on coronavirus genomes, including extensive homoplasy from editing and reversion at targeted sites and the occurrence of driven amino acid sequence changes in viral proteins. If sustained over longer periods, this process may account for the previously reported marked global depletion of C and excess of U bases in human seasonal coronavirus genomes. This review synthesizes the current knowledge on APOBEC evolution and function and the evidence of their role in APOBEC-mediated genome editing of SARS-CoV-2 and other coronaviruses.
Identifiants
pubmed: 33545556
pii: S0042-6822(20)30265-8
doi: 10.1016/j.virol.2020.12.018
pmc: PMC7831814
pii:
doi:
Substances chimiques
APOBEC Deaminases
EC 3.5.4.5
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
62-72Subventions
Organisme : Wellcome Trust
ID : WT103767MA
Pays : United Kingdom
Informations de copyright
Copyright © 2021 The Authors. Published by Elsevier Inc. All rights reserved.
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