Epidermolytic epidermal nevus caused by a somatic mutation in KRT2.


Journal

Pediatric dermatology
ISSN: 1525-1470
Titre abrégé: Pediatr Dermatol
Pays: United States
ID NLM: 8406799

Informations de publication

Date de publication:
Mar 2021
Historique:
pubmed: 9 2 2021
medline: 15 5 2021
entrez: 8 2 2021
Statut: ppublish

Résumé

Superficial epidermolytic ichthyosis (formerly Ichthyosis bullosa of Siemens) is an uncommon condition caused by dominant mutations in KRT2 encoding keratin 2. Epidermolytic epidermal nevus due to somatic mutations in KRT2 is even rarer. Here, we report the third case of KRT2-associated epidermal nevus and review the literature.

Identifiants

pubmed: 33555633
doi: 10.1111/pde.14529
doi:

Substances chimiques

KRT2 protein, human 0
Keratin-2 0
Keratins 68238-35-7

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

538-540

Subventions

Organisme : Ram family foundation

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

Rothnagel JA, Traupe H, Wojcik S, et al. Mutations in the rod domain of keratin 2e in patients with ichthyosis bullosa of Siemens. Nat Genet. 1994;7(4):485-490.
Paller AS, Syder AJ, Chan YM, et al. Genetic and clinical mosaicism in a type of epidermal nevus. N Engl J Med. 1994;331(21):1408-1415.
Tsubota A, Akiyama M, Sakai K, et al. Keratin 1 gene mutation detected in epidermal nevus with epidermolytic hyperkeratosis. J Invest Dermatol. 2007;127(6):1371-1374.
Li Y, Cheng R, Liang J, Yao Z, Li M. The first case of a mosaic superficial epidermolytic ichthyosis diagnosed by Ultra-Deep Sequence. Mol Genet Genom Med. 2020;8(11):e1457.
Diociaiuti A, Castiglia D, Corbeddu M, et al. First case of KRT2 epidermolytic nevus and novel clinical and genetic findings in 26 Italian patients with keratinopathic ichthyoses. Int J Mol Sci. 2020;21(20):7707.
Hotz A, Oji V, Bourrat E, et al. Expanding the clinical and genetic spectrum of KRT1, KRT2 and KRT10 mutations in keratinopathic ichthyosis. Acta Derm Venereol. 2016;96(4):473-478.

Auteurs

Janan Mohamad (J)

Division of Dermatology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.
Department of Human Molecular Genetics & Biochemistry, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Liat Samuelov (L)

Division of Dermatology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.
Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Sari Assaf (S)

Division of Dermatology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.
Department of Human Molecular Genetics & Biochemistry, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Dan Vodo (D)

Division of Dermatology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Ofer Sarig (O)

Division of Dermatology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Eli Sprecher (E)

Division of Dermatology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.
Department of Human Molecular Genetics & Biochemistry, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

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Classifications MeSH