Peginterferon lambda for the treatment of outpatients with COVID-19: a phase 2, placebo-controlled randomised trial.


Journal

The Lancet. Respiratory medicine
ISSN: 2213-2619
Titre abrégé: Lancet Respir Med
Pays: England
ID NLM: 101605555

Informations de publication

Date de publication:
05 2021
Historique:
received: 27 10 2020
revised: 03 12 2020
accepted: 04 12 2020
pubmed: 9 2 2021
medline: 14 5 2021
entrez: 8 2 2021
Statut: ppublish

Résumé

To date, only monoclonal antibodies have been shown to be effective for outpatients with COVID-19. Interferon lambda-1 is a type III interferon involved in innate antiviral responses with activity against respiratory pathogens. We aimed to investigate the safety and efficacy of peginterferon lambda in the treatment of outpatients with mild-to-moderate COVID-19. In this double-blind, placebo-controlled trial, outpatients with laboratory-confirmed COVID-19 were randomly assigned to a single subcutaneous injection of peginterferon lambda 180 μg or placebo within 7 days of symptom onset or first positive swab if asymptomatic. Participants were randomly assigned (1:1) using a computer-generated randomisation list created with a randomisation schedule in blocks of four. At the time of administration, study nurses received a sealed opaque envelope with the treatment allocation number. The primary endpoint was the proportion of patients who were negative for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) RNA on day 7 after the injection, analysed by a χ Between May 18, and Sept 4, 2020, we recruited 30 patients per group. The decline in SARS-CoV-2 RNA was greater in those treated with peginterferon lambda than placebo from day 3 onwards, with a difference of 2·42 log copies per mL at day 7 (p=0·0041). By day 7, 24 (80%) participants in the peginterferon lambda group had an undetectable viral load, compared with 19 (63%) in the placebo group (p=0·15). After controlling for baseline viral load, patients in the peginterferon lambda group were more likely to have undetectable virus by day 7 than were those in the placebo group (odds ratio [OR] 4·12 [95% CI 1·15-16·73; p=0·029). Of those with baseline viral load above 10 Peginterferon lambda accelerated viral decline in outpatients with COVID-19, increasing the proportion of patients with viral clearance by day 7, particularly in those with high baseline viral load. Peginterferon lambda has potential to prevent clinical deterioration and shorten duration of viral shedding. The Toronto COVID-19 Action Initiative, University of Toronto, and the Ontario First COVID-19 Rapid Research Fund, Toronto General & Western Hospital Foundation.

Sections du résumé

BACKGROUND
To date, only monoclonal antibodies have been shown to be effective for outpatients with COVID-19. Interferon lambda-1 is a type III interferon involved in innate antiviral responses with activity against respiratory pathogens. We aimed to investigate the safety and efficacy of peginterferon lambda in the treatment of outpatients with mild-to-moderate COVID-19.
METHODS
In this double-blind, placebo-controlled trial, outpatients with laboratory-confirmed COVID-19 were randomly assigned to a single subcutaneous injection of peginterferon lambda 180 μg or placebo within 7 days of symptom onset or first positive swab if asymptomatic. Participants were randomly assigned (1:1) using a computer-generated randomisation list created with a randomisation schedule in blocks of four. At the time of administration, study nurses received a sealed opaque envelope with the treatment allocation number. The primary endpoint was the proportion of patients who were negative for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) RNA on day 7 after the injection, analysed by a χ
FINDINGS
Between May 18, and Sept 4, 2020, we recruited 30 patients per group. The decline in SARS-CoV-2 RNA was greater in those treated with peginterferon lambda than placebo from day 3 onwards, with a difference of 2·42 log copies per mL at day 7 (p=0·0041). By day 7, 24 (80%) participants in the peginterferon lambda group had an undetectable viral load, compared with 19 (63%) in the placebo group (p=0·15). After controlling for baseline viral load, patients in the peginterferon lambda group were more likely to have undetectable virus by day 7 than were those in the placebo group (odds ratio [OR] 4·12 [95% CI 1·15-16·73; p=0·029). Of those with baseline viral load above 10
INTERPRETATION
Peginterferon lambda accelerated viral decline in outpatients with COVID-19, increasing the proportion of patients with viral clearance by day 7, particularly in those with high baseline viral load. Peginterferon lambda has potential to prevent clinical deterioration and shorten duration of viral shedding.
FUNDING
The Toronto COVID-19 Action Initiative, University of Toronto, and the Ontario First COVID-19 Rapid Research Fund, Toronto General & Western Hospital Foundation.

Identifiants

pubmed: 33556319
pii: S2213-2600(20)30566-X
doi: 10.1016/S2213-2600(20)30566-X
pmc: PMC7906707
pii:
doi:

Substances chimiques

Antiviral Agents 0
Interleukins 0
RNA, Viral 0
peginterferon lambda-1a 0T0250N43U
Polyethylene Glycols 3WJQ0SDW1A

Banques de données

ClinicalTrials.gov
['NCT04354259']

Types de publication

Clinical Trial, Phase II Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

498-510

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

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Auteurs

Jordan J Feld (JJ)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada. Electronic address: jordan.feld@uhn.ca.

Christopher Kandel (C)

Faculty of Medicine, University of Toronto, Toronto, ON, Canada.

Mia J Biondi (MJ)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Robert A Kozak (RA)

Sunnybrook Health Sciences Centre, University of Toronto, Toronto, ON, Canada.

Muhammad Atif Zahoor (MA)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Camille Lemieux (C)

University Health Network, University of Toronto, Toronto, ON, Canada.

Sergio M Borgia (SM)

Division of Infectious Diseases, William Osler Health System and McMaster University, Hamilton, ON, Canada.

Andrea K Boggild (AK)

University Health Network, University of Toronto, Toronto, ON, Canada.

Jeff Powis (J)

Michael Garron Hospital, University of Toronto, Toronto, ON, Canada.

Janine McCready (J)

Michael Garron Hospital, University of Toronto, Toronto, ON, Canada.

Darrell H S Tan (DHS)

St Michael's Hospital, University of Toronto, Toronto, ON, Canada.

Tiffany Chan (T)

Trillium Health Partners, Toronto, ON, Canada.

Bryan Coburn (B)

University Health Network, University of Toronto, Toronto, ON, Canada.

Deepali Kumar (D)

Toronto General Hospital, Multiorgan Transplant Centre, University of Toronto, Toronto, ON, Canada.

Atul Humar (A)

Toronto General Hospital, Multiorgan Transplant Centre, University of Toronto, Toronto, ON, Canada.

Adrienne Chan (A)

Sunnybrook Health Sciences Centre, University of Toronto, Toronto, ON, Canada.

Braden O'Neil (B)

North York General Hospital, University of Toronto, Toronto, ON, Canada.

Seham Noureldin (S)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Joshua Booth (J)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Rachel Hong (R)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

David Smookler (D)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Wesam Aleyadeh (W)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Anjali Patel (A)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Bethany Barber (B)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Julia Casey (J)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Ryan Hiebert (R)

Sunnybrook Health Sciences Centre, University of Toronto, Toronto, ON, Canada.

Henna Mistry (H)

Sunnybrook Health Sciences Centre, University of Toronto, Toronto, ON, Canada.

Ingrid Choong (I)

Eiger BioPharmaceuticals, Palo Alto, CA, USA.

Colin Hislop (C)

Eiger BioPharmaceuticals, Palo Alto, CA, USA.

Deanna M Santer (DM)

The Li Ka Shing Institute of Virology, University of Alberta, Edmonton, AB, Canada.

D Lorne Tyrrell (D)

The Li Ka Shing Institute of Virology, University of Alberta, Edmonton, AB, Canada.

Jeffrey S Glenn (JS)

Departments of Medicine and Microbiology & Immunology, Stanford University School of Medicine, Palo Alto, CA, USA.

Adam J Gehring (AJ)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Harry L A Janssen (HLA)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada.

Bettina E Hansen (BE)

Toronto Centre for Liver Disease, University of Toronto, Toronto, ON, Canada; University Health Network, University of Toronto, Toronto, ON, Canada; Institute of Health Policy, Management and Evaluation, University of Toronto, Toronto, ON, Canada.

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Classifications MeSH