The Age-Sensitive Efficacy of Calorie Restriction on Mitochondrial Biogenesis and mtDNA Damage in Rat Liver.


Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
07 Feb 2021
Historique:
received: 28 12 2020
revised: 03 02 2021
accepted: 04 02 2021
entrez: 10 2 2021
pubmed: 11 2 2021
medline: 13 4 2021
Statut: epublish

Résumé

Calorie restriction (CR) is the most efficacious treatment to delay the onset of age-related changes such as mitochondrial dysfunction. However, the sensitivity of mitochondrial markers to CR and the age-related boundaries of CR efficacy are not fully elucidated. We used liver samples from ad libitum-fed (AL) rats divided in: 18-month-old (AL-18), 28-month-old (AL-28), and 32-month-old (AL-32) groups, and from CR-treated (CR) 28-month-old (CR-28) and 32-month-old (CR-32) counterparts to assay the effect of CR on several mitochondrial markers. The age-related decreases in citrate synthase activity, in TFAM, MFN2, and DRP1 protein amounts and in the mtDNA content in the AL-28 group were prevented in CR-28 counterparts. Accordingly, CR reduced oxidative mtDNA damage assessed through the incidence of oxidized purines at specific mtDNA regions in CR-28 animals. These findings support the anti-aging effect of CR up to 28 months. Conversely, the protein amounts of LonP1, Cyt c, OGG1, and APE1 and the 4.8 Kb mtDNA deletion content were not affected in CR-28 rats. The absence of significant differences between the AL-32 values and the CR-32 counterparts suggests an age-related boundary of CR efficacy at this age. However, this only partially curtails the CR benefits in counteracting the generalized aging decline and the related mitochondrial involvement.

Identifiants

pubmed: 33562258
pii: ijms22041665
doi: 10.3390/ijms22041665
pmc: PMC7915472
pii:
doi:

Substances chimiques

DNA, Mitochondrial 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Ministero dell'Istruzione, dell'Università e della Ricerca
ID : FFABR2018

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Auteurs

Guglielmina Chimienti (G)

Department of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari Aldo Moro, Via Orabona 4, 70125 Bari, Italy.

Anna Picca (A)

Fondazione Policlinico Universitario "Agostino Gemelli" IRCCS, L.go F. Vito 8, 00168 Rome, Italy.
Aging Research Center, Department of Neurobiology, Care Sciences and Society, Karolinska Institutet and Stockholm University, 11330 Stockholm, Sweden.

Flavio Fracasso (F)

Department of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari Aldo Moro, Via Orabona 4, 70125 Bari, Italy.

Francesco Russo (F)

Laboratory of Nutritional Pathophysiology, National Institute of Gastroenterology "S. de Bellis", Research Hospital, 70013 Castellana Grotte, Italy.

Antonella Orlando (A)

Laboratory of Nutritional Pathophysiology, National Institute of Gastroenterology "S. de Bellis", Research Hospital, 70013 Castellana Grotte, Italy.

Giuseppe Riezzo (G)

Laboratory of Nutritional Pathophysiology, National Institute of Gastroenterology "S. de Bellis", Research Hospital, 70013 Castellana Grotte, Italy.

Christiaan Leeuwenburgh (C)

Department of Aging and Geriatric Research, Institute on Aging, Division of Biology of Aging, University of Florida, Gainesville, FL 32611, USA.

Vito Pesce (V)

Department of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari Aldo Moro, Via Orabona 4, 70125 Bari, Italy.

Angela Maria Serena Lezza (AMS)

Department of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari Aldo Moro, Via Orabona 4, 70125 Bari, Italy.

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Classifications MeSH