Prostate Cancer Peripheral Blood NK Cells Show Enhanced CD9, CD49a, CXCR4, CXCL8, MMP-9 Production and Secrete Monocyte-Recruiting and Polarizing Factors.


Journal

Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960

Informations de publication

Date de publication:
2020
Historique:
received: 22 07 2020
accepted: 08 12 2020
entrez: 11 2 2021
pubmed: 12 2 2021
medline: 16 6 2021
Statut: epublish

Résumé

Natural killer (NK) cells, effector lymphocytes of the innate immunity, have been shown to be altered in several cancers, both at tissue and peripheral levels. We have shown that in Non-Small Cell Lung Cancer (NSCLC) and colon cancer, tumour associated circulating NK (TA-NK) and tumour infiltrating NK (TI-NK) exhibit pro-angiogenic phenotype/functions. However, there is still a lack of knowledge concerning the phenotype of peripheral blood (PB) NK (pNK) cells in prostate cancer (PCa). Here, we phenotypically and functionally characterized pNK from PCa patients (PCa TA-NKs) and investigated their interactions with endothelial cells and monocytes/macrophages. NK cell subset distribution in PB of PCa patients was investigated, by multicolor flow cytometry, for surface antigens expression. Protein arrays were performed to characterize the secretome on FACS-sorted pNK cells. Conditioned media (CM) from FACS-sorted PCa pTA-NKs were used to determine their ability to induce pro-inflammatory/pro-angiogenic phenotype/functions in endothelial cells, monocytes, and macrophages. CM from three different PCa (PC-3, DU-145, LNCaP) cell lines, were used to assess their effects on human NK cell polarization

Identifiants

pubmed: 33569050
doi: 10.3389/fimmu.2020.586126
pmc: PMC7868409
doi:

Substances chimiques

Biomarkers, Tumor 0
Cytokines 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

586126

Informations de copyright

Copyright © 2021 Gallazzi, Baci, Mortara, Bosi, Buono, Naselli, Guarneri, Dehò, Capogrosso, Albini, Noonan and Bruno.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Matteo Gallazzi (M)

Laboratory of Immunology and General Pathology, Department of Biotechnology and Life Sciences, University of Insubria, Varese, Italy.

Denisa Baci (D)

Laboratory of Immunology and General Pathology, Department of Biotechnology and Life Sciences, University of Insubria, Varese, Italy.

Lorenzo Mortara (L)

Laboratory of Immunology and General Pathology, Department of Biotechnology and Life Sciences, University of Insubria, Varese, Italy.

Annalisa Bosi (A)

Laboratory of Pharmacology, Department of Medicine and Surgery, University of Insubria, Varese, Italy.

Giuseppe Buono (G)

Unit of Immunology, IRCCS MultiMedica, Milan, Italy.

Angelo Naselli (A)

Unit of Urology, San Giuseppe Hospital, IRCCS MultiMedica, Milan, Italy.

Andrea Guarneri (A)

Unit of Urology, San Giuseppe Hospital, IRCCS MultiMedica, Milan, Italy.

Federico Dehò (F)

S.C. of Urology, ASST Settelaghi, Ospedale di Circolo e Fondazione Macchi, Varese, Italy.

Paolo Capogrosso (P)

S.C. of Urology, ASST Settelaghi, Ospedale di Circolo e Fondazione Macchi, Varese, Italy.

Adriana Albini (A)

Laboratory of Vascular Biology and Angiogenesis, IRCCS MultiMedica, Milano, Italy.

Douglas M Noonan (DM)

Laboratory of Immunology and General Pathology, Department of Biotechnology and Life Sciences, University of Insubria, Varese, Italy.
Laboratory of Vascular Biology and Angiogenesis, IRCCS MultiMedica, Milano, Italy.

Antonino Bruno (A)

Unit of Immunology, IRCCS MultiMedica, Milan, Italy.

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