Obstructive sleep apnoea increases lipolysis and deteriorates glucose homeostasis in patients with type 2 diabetes mellitus.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
11 02 2021
Historique:
received: 07 06 2020
accepted: 25 01 2021
entrez: 12 2 2021
pubmed: 13 2 2021
medline: 16 11 2021
Statut: epublish

Résumé

Obstructive sleep apnoea (OSA) is associated with type 2 diabetes mellitus (T2DM). However, mechanisms mediating association between these two conditions remain unclear. This study investigated, whether the OSA-associated changes in adipose tissue lipolysis might contribute to impaired glucose homeostasis in patient with T2DM. Thirty-five matched subjects were recruited into three groups: T2DM + severe OSA (T2DM + OSA, n = 11), T2DM with mild/no OSA (T2DM, n = 10) and healthy controls (n = 14). Subcutaneous abdominal adipose tissue microdialysis assessed spontaneous, epinephrine- and isoprenaline-stimulated lipolysis. Glucose metabolism was assessed by intravenous glucose tolerance test. Spontaneous lipolysis was higher in the T2DM + OSA compared with the T2DM (60.34 ± 23.40 vs. 42.53 ± 10.16 μmol/L, p = 0.013), as well as epinephrine-stimulated lipolysis (236.84 ± 103.90 vs. 167.39 ± 52.17 µmol/L, p < 0.001). Isoprenaline-stimulated lipolysis was unaffected by the presence of OSA (p = 0.750). The α

Identifiants

pubmed: 33574418
doi: 10.1038/s41598-021-83018-1
pii: 10.1038/s41598-021-83018-1
pmc: PMC7878919
doi:

Substances chimiques

Insulin 0
Glucose IY9XDZ35W2
Isoproterenol L628TT009W
Epinephrine YKH834O4BH

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3567

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Auteurs

Minh Duc Trinh (MD)

Department of Pathophysiology, Third Faculty of Medicine, Charles University, Ruska 87, 100 00, Prague 10, Czech Republic.
Department of Cardiology, University Hospital Královské Vinohrady, Prague, Czech Republic.

Andrea Plihalova (A)

Department of Internal Medicine, University Hospital Královské Vinohrady, Prague, Czech Republic.
Centre for Research on Diabetes, Metabolism and Nutrition, Third Faculty of Medicine, Charles University, Prague, Czech Republic.

Jan Gojda (J)

Department of Internal Medicine, University Hospital Královské Vinohrady, Prague, Czech Republic.
Centre for Research on Diabetes, Metabolism and Nutrition, Third Faculty of Medicine, Charles University, Prague, Czech Republic.

Katerina Westlake (K)

Department of Pathophysiology, Third Faculty of Medicine, Charles University, Ruska 87, 100 00, Prague 10, Czech Republic.

Jan Spicka (J)

Department of Laboratory Diagnostics, University Hospital Královské Vinohrady, Prague, Czech Republic.

Zuzana Lattova (Z)

Department of Pathophysiology, Third Faculty of Medicine, Charles University, Ruska 87, 100 00, Prague 10, Czech Republic.

Martin Pretl (M)

Neurology and Sleep Laboratory, INSPAMED S.R.O., Prague, Czech Republic.

Jan Polak (J)

Department of Pathophysiology, Third Faculty of Medicine, Charles University, Ruska 87, 100 00, Prague 10, Czech Republic. jan.polak@lf3.cuni.cz.
Department of Internal Medicine, University Hospital Královské Vinohrady, Prague, Czech Republic. jan.polak@lf3.cuni.cz.

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