Baroreceptor Modulation of the Cardiovascular System, Pain, Consciousness, and Cognition.
Journal
Comprehensive Physiology
ISSN: 2040-4603
Titre abrégé: Compr Physiol
Pays: United States
ID NLM: 101574442
Informations de publication
Date de publication:
12 02 2021
12 02 2021
Historique:
entrez:
12
2
2021
pubmed:
13
2
2021
medline:
26
10
2021
Statut:
epublish
Résumé
Baroreceptors are mechanosensitive elements of the peripheral nervous system that maintain cardiovascular homeostasis by coordinating the responses to external and internal environmental stressors. While it is well known that carotid and cardiopulmonary baroreceptors modulate sympathetic vasomotor and parasympathetic cardiac neural autonomic drive, to avoid excessive fluctuations in vascular tone and maintain intravascular volume, there is increasing recognition that baroreceptors also modulate a wide range of non-cardiovascular physiological responses via projections from the nucleus of the solitary tract to regions of the central nervous system, including the spinal cord. These projections regulate pain perception, sleep, consciousness, and cognition. In this article, we summarize the physiology of baroreceptor pathways and responses to baroreceptor activation with an emphasis on the mechanisms influencing cardiovascular function, pain perception, consciousness, and cognition. Understanding baroreceptor-mediated effects on cardiac and extra-cardiac autonomic activities will further our understanding of the pathophysiology of multiple common clinical conditions, such as chronic pain, disorders of consciousness (e.g., abnormalities in sleep-wake), and cognitive impairment, which may result in the identification and implementation of novel treatment modalities. © 2021 American Physiological Society. Compr Physiol 11:1373-1423, 2021.
Identifiants
pubmed: 33577130
doi: 10.1002/cphy.c190038
pmc: PMC8480547
mid: NIHMS1732244
doi:
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1373-1423Subventions
Organisme : NINDS NIH HHS
ID : P01 NS045685
Pays : United States
Organisme : NINDS NIH HHS
ID : R21 NS112912
Pays : United States
Organisme : NIDCR NIH HHS
ID : U01 DE017018
Pays : United States
Informations de copyright
Copyright © 2021 American Physiological Society. All rights reserved.
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