Detection of nuclear receptors in gingival samples of diabetic and nondiabetic periodontitis patients.


Journal

Nigerian journal of clinical practice
ISSN: 1119-3077
Titre abrégé: Niger J Clin Pract
Pays: India
ID NLM: 101150032

Informations de publication

Date de publication:
Feb 2021
Historique:
entrez: 19 2 2021
pubmed: 20 2 2021
medline: 24 2 2021
Statut: ppublish

Résumé

Diabetes and periodontitis are two chronic inflammatory diseases sharing specific etiopathogenetic mechanisms, and both cause severe inflammation and destruction. The present study aimed to determine the receptor expressions of peroxisome proliferative-activated receptor (PPAR)-γ, retinoid X receptor (RXR)-α, vitamin D receptor (VDR), and nuclear factor kappa B (NF-κB) expressions in healthy gingiva and diseased gingival samples with or without diabetes. Forty-five participants as (1) healthy controls (C), (2) periodontitis group (P), and (3) diabetes and periodontitis group (DP) were enrolled. Plaque index (PI), gingival index (GI), probing depth (PD), clinical attachment levels (CAL), and bleeding on probing (BOP) were recorded in all participants. Two gingival biopsies from each participant were obtained, and one underwent histological tissue processing while the other underwent qRT-PCR analysis of nuclear receptors. Inflammatory and fibroblast cell counts, PPAR-γ, RXR-α, VDR, and NF-κB were evaluated. Fibroblast cells were lowest in the DP group and highest in the healthy group. PPAR-γ, VDR, RXR, and NF-κB expressions were higher in the healthy controls in the qRT-PCR analysis and similar in the other groups. Immunohistochemistry analysis also showed similar results. qRT-PCR results concluded that healthy gingival samples had higher PPAR-γ, RXR, VDR, and NF-κB expressions, and immunohistochemistry findings supported the results. In addition, healthy gingiva contained higher fibroblast cells and lower inflammatory cells.

Sections du résumé

BACKGROUND BACKGROUND
Diabetes and periodontitis are two chronic inflammatory diseases sharing specific etiopathogenetic mechanisms, and both cause severe inflammation and destruction.
AIMS OBJECTIVE
The present study aimed to determine the receptor expressions of peroxisome proliferative-activated receptor (PPAR)-γ, retinoid X receptor (RXR)-α, vitamin D receptor (VDR), and nuclear factor kappa B (NF-κB) expressions in healthy gingiva and diseased gingival samples with or without diabetes.
METHODS METHODS
Forty-five participants as (1) healthy controls (C), (2) periodontitis group (P), and (3) diabetes and periodontitis group (DP) were enrolled. Plaque index (PI), gingival index (GI), probing depth (PD), clinical attachment levels (CAL), and bleeding on probing (BOP) were recorded in all participants. Two gingival biopsies from each participant were obtained, and one underwent histological tissue processing while the other underwent qRT-PCR analysis of nuclear receptors. Inflammatory and fibroblast cell counts, PPAR-γ, RXR-α, VDR, and NF-κB were evaluated.
RESULTS RESULTS
Fibroblast cells were lowest in the DP group and highest in the healthy group. PPAR-γ, VDR, RXR, and NF-κB expressions were higher in the healthy controls in the qRT-PCR analysis and similar in the other groups. Immunohistochemistry analysis also showed similar results.
CONCLUSION CONCLUSIONS
qRT-PCR results concluded that healthy gingival samples had higher PPAR-γ, RXR, VDR, and NF-κB expressions, and immunohistochemistry findings supported the results. In addition, healthy gingiva contained higher fibroblast cells and lower inflammatory cells.

Identifiants

pubmed: 33605919
pii: NigerJClinPract_2021_24_2_269_309811
doi: 10.4103/njcp.njcp_216_20
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

269-276

Déclaration de conflit d'intérêts

None

Auteurs

O Karatas (O)

Department of Periodontology, Faculty of Dentistry, Tokat Gaziosmanpasa University, Tokat, Turkey.

H Balci Yuce (HB)

Department of Periodontology, Faculty of Dentistry, Tokat Gaziosmanpasa University, Tokat, Turkey.

M M Taskan (MM)

Department of Periodontology, Faculty of Dentistry, Tokat Gaziosmanpasa University, Tokat, Turkey.

F Gevrek (F)

Department of Histology and Embryology, Faculty of Medicine, Tokat Gaziosmanpasa University, Tokat, Turkey.

F Ucan Yarkac (FU)

Department of Periodontology, Faculty of Dentistry, Necmettin Erbakan University, Konya, Turkey.

E Cacan (E)

Department of Molecular Biology and Genetics, Faculty of Science and Art, Tokat Gaziosmanpasa University, Tokat, Turkey.

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Classifications MeSH