Oxocrebanine: A Novel Dual Topoisomerase inhibitor, Suppressed the Proliferation of Breast Cancer Cells MCF-7 by Inducing DNA Damage and Mitotic Arrest.


Journal

Phytomedicine : international journal of phytotherapy and phytopharmacology
ISSN: 1618-095X
Titre abrégé: Phytomedicine
Pays: Germany
ID NLM: 9438794

Informations de publication

Date de publication:
Apr 2021
Historique:
received: 26 10 2020
revised: 28 01 2021
accepted: 07 02 2021
pubmed: 22 2 2021
medline: 2 6 2021
entrez: 21 2 2021
Statut: ppublish

Résumé

DNA topoisomerase (Topo) inhibition plays key role in breast cancer treatment. Stephania hainanensis H. S. Lo et Y. Tsoong (S. hainanensis), a Li nationality plant that has abundant aporphine alkaloids, can inhibit Topo. To identify a dual Topo inhibitor, a deep and systematic study of active aporphine alkaloids in S. hainanensis and their mechanisms of inhibiting breast cancer proliferation and Topo activity are essential. This study aimed to assess the anti-breast cancer and Topo inhibitory activities of oxocrebanine and explore the underlying mechanisms. The growth inhibitory activities of 12 compounds in S. hainanensis were screened by MTT assay in MCF-7, SGC-7901, HepG-2 cells, and compared with the effects on human normal mammary epithelial MCF-10A cells as non cancer control cells. The Topo inhibitory activity was assessed by DNA relaxation and unwinding assays, kDNA decatenation assay and western blot. Cell cycle and autophagy analyses were carried out with flow cytometry and staining. Acridine orange staining and α-tubulin morphology were observed by fluorescence microscopy. Western blot was used to examine microtubule assembly dynamics and the expression levels of key proteins associated with DNA damage, autophagy and mitotic arrest. Oxocrebanine was the anti-breast cancer active alkaloid in S. hainanensis. It exhibited the best inhibitory effect on MCF-7 cells with an IC Oxocrebanine was the anti-breast cancer active aporphine alkaloid in S. hainanensis. Oxocrebanine was a Topo I/IIα dual inhibitor, catalytic inhibitor and DNA intercalator. Oxocrebanine caused DNA damage, autophagy, and mitotic arrest in MCF-7 cells. Oxocrebanine also disrupted tubulin polymerization. Accordingly, oxocrebanine held a great potential for development as a novel dual Topo inhibitor for effective breast cancer treatment.

Sections du résumé

BACKGROUND BACKGROUND
DNA topoisomerase (Topo) inhibition plays key role in breast cancer treatment. Stephania hainanensis H. S. Lo et Y. Tsoong (S. hainanensis), a Li nationality plant that has abundant aporphine alkaloids, can inhibit Topo.
PURPOSE OBJECTIVE
To identify a dual Topo inhibitor, a deep and systematic study of active aporphine alkaloids in S. hainanensis and their mechanisms of inhibiting breast cancer proliferation and Topo activity are essential.
STUDY DESIGN METHODS
This study aimed to assess the anti-breast cancer and Topo inhibitory activities of oxocrebanine and explore the underlying mechanisms.
METHODS METHODS
The growth inhibitory activities of 12 compounds in S. hainanensis were screened by MTT assay in MCF-7, SGC-7901, HepG-2 cells, and compared with the effects on human normal mammary epithelial MCF-10A cells as non cancer control cells. The Topo inhibitory activity was assessed by DNA relaxation and unwinding assays, kDNA decatenation assay and western blot. Cell cycle and autophagy analyses were carried out with flow cytometry and staining. Acridine orange staining and α-tubulin morphology were observed by fluorescence microscopy. Western blot was used to examine microtubule assembly dynamics and the expression levels of key proteins associated with DNA damage, autophagy and mitotic arrest.
RESULTS RESULTS
Oxocrebanine was the anti-breast cancer active alkaloid in S. hainanensis. It exhibited the best inhibitory effect on MCF-7 cells with an IC
CONCLUSION CONCLUSIONS
Oxocrebanine was the anti-breast cancer active aporphine alkaloid in S. hainanensis. Oxocrebanine was a Topo I/IIα dual inhibitor, catalytic inhibitor and DNA intercalator. Oxocrebanine caused DNA damage, autophagy, and mitotic arrest in MCF-7 cells. Oxocrebanine also disrupted tubulin polymerization. Accordingly, oxocrebanine held a great potential for development as a novel dual Topo inhibitor for effective breast cancer treatment.

Identifiants

pubmed: 33611211
pii: S0944-7113(21)00046-5
doi: 10.1016/j.phymed.2021.153504
pii:
doi:

Substances chimiques

Alkaloids 0
Antineoplastic Agents, Phytogenic 0
Aporphines 0
Topoisomerase Inhibitors 0
oxocrebanine 0
DNA Topoisomerases, Type I EC 5.99.1.2
TOP1 protein, human EC 5.99.1.2
DNA Topoisomerases, Type II EC 5.99.1.3

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

153504

Informations de copyright

Copyright © 2021 Elsevier GmbH. All rights reserved.

Auteurs

Lei Yu (L)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China.

Shuang Han (S)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China; Engineering Research Center of Natural Anticancer Drugs, Ministry of Education, Harbin, 150076, China.

Lang Lang (L)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China.

Hui Song (H)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China.

CaiYun Zhang (C)

Key Laboratory of Tropical Translational Medicine of Ministry of Education, Hainan Key Laboratory for Research and Development of Tropical Herbs, School of Pharmacy, Hainan Medical University, Haikou, 57199, China.

Lin Dong (L)

Key Laboratory of Tropical Translational Medicine of Ministry of Education, Hainan Key Laboratory for Research and Development of Tropical Herbs, School of Pharmacy, Hainan Medical University, Haikou, 57199, China.

ShaoHua Jia (S)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China.

Ying Zhang (Y)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China; Engineering Research Center of Natural Anticancer Drugs, Ministry of Education, Harbin, 150076, China.

Di Xiao (D)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China; Engineering Research Center of Natural Anticancer Drugs, Ministry of Education, Harbin, 150076, China.

Jun Liu (J)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China.

Ying Xu (Y)

College of Pharmacy, Engineering Research Center for Medicine, Harbin University of Commerce, Harbin, 150076, China. Electronic address: xuying030709@163.com.

XiaoPo Zhang (X)

Key Laboratory of Tropical Translational Medicine of Ministry of Education, Hainan Key Laboratory for Research and Development of Tropical Herbs, School of Pharmacy, Hainan Medical University, Haikou, 57199, China. Electronic address: z_xp1412@163.com.

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Classifications MeSH