Protective effects of vitamin D on learning and memory deficit induced by scopolamine in male rats: the roles of brain-derived neurotrophic factor and oxidative stress.


Journal

Naunyn-Schmiedeberg's archives of pharmacology
ISSN: 1432-1912
Titre abrégé: Naunyn Schmiedebergs Arch Pharmacol
Pays: Germany
ID NLM: 0326264

Informations de publication

Date de publication:
07 2021
Historique:
received: 04 11 2020
accepted: 04 02 2021
pubmed: 3 3 2021
medline: 15 1 2022
entrez: 2 3 2021
Statut: ppublish

Résumé

The beneficial effects of vitamin D (vit D) on central nervous system disorders have been suggested. In the current research, the protective effects of vit D on learning and memory deficit induced by scopolamine, oxidative stress criteria, brain-derived neurotrophic factor (BDNF), and nitric oxide (NO) in the brain were investigated. Rats were divided into five groups, including (1) Control, (2) Scopolamine (2 mg/kg), (3-5) Scopolamine + Vit D (100, 1000, and 10,000 IU/kg) groups. Vit D administrated for 2 weeks and in the third week scopolamine co-administrated with vit D and behavioral tests, including Morris water maze (MWM) and passive avoidance (PA) tests, were carried out. The cortical and hippocampal tissues were analyzed for BDNF, catalase (CAT), and superoxide dismutase (SOD) activities, thiol content, NO metabolites, and malondialdehyde (MDA) concentration. Scopolamine injection significantly impaired rats' performance on the MWM and PA test. It further enhanced the MDA and nitrite level while decreased thiol content and BDNF levels and SOD and CAT activities in the brain. Administration of both 1000 and 10,000 IU/kg vit D improved cognitive outcome in MWM and PA tests. In addition, vit D elevated thiol content, SOD and CAT activities, and BDNF levels, while reduced nitrite and MDA concentration. Vit D also increased the levels of vit D and calcium in the serum. The results demonstrated that vit D has protective effects on scopolamine-associated learning and memory impairment by improving BDNF levels and attenuating NO and brain tissue oxidative damage.

Identifiants

pubmed: 33649977
doi: 10.1007/s00210-021-02062-w
pii: 10.1007/s00210-021-02062-w
doi:

Substances chimiques

Adjuvants, Anesthesia 0
Antioxidants 0
Bdnf protein, rat 0
Brain-Derived Neurotrophic Factor 0
Vitamin D 1406-16-2
Nitric Oxide 31C4KY9ESH
Scopolamine DL48G20X8X

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1451-1466

Références

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Auteurs

Fatemeh Mansouri (F)

Department of Physiology, Faculty of Medicine, Hormozgan University of Medical Sciences, Bandar Abbas, Iran.

Hamideh Ghanbari (H)

Department of Physiology, Faculty of Medicine, Hormozgan University of Medical Sciences, Bandar Abbas, Iran. h.ghanbarih@gmail.com.

Narges Marefati (N)

Division of Neurocognitive Sciences, Psychiatry and Behavioral Sciences Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Applied Biomedical Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Zohre Arab (Z)

Applied Biomedical Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Hossein Salmani (H)

Division of Neurocognitive Sciences, Psychiatry and Behavioral Sciences Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Farimah Beheshti (F)

Neuroscience Research Center, Torbat Heydariyeh University of Medical Sciences, Torbat Heydariyeh, Iran.
Department of Physiology, School of Paramedical Sciences, Torbat Heydariyeh University of Medical Sciences, Torbat Heydariyeh, Iran.

Mahmoud Hosseini (M)

Division of Neurocognitive Sciences, Psychiatry and Behavioral Sciences Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. Hosseinim@mums.ac.ir.
Neuroscience Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. Hosseinim@mums.ac.ir.

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