Vascular Ehlers-Danlos syndrome (vEDS): CT and histologic findings of pleural and lung parenchymal damage.


Journal

European radiology
ISSN: 1432-1084
Titre abrégé: Eur Radiol
Pays: Germany
ID NLM: 9114774

Informations de publication

Date de publication:
Aug 2021
Historique:
received: 19 07 2020
accepted: 21 01 2021
revised: 17 12 2020
pubmed: 3 3 2021
medline: 14 7 2021
entrez: 2 3 2021
Statut: ppublish

Résumé

To describe CT features of lung involvement in patients with vascular Ehlers-Danlos syndrome (vEDS), a rare genetic condition caused by pathogenic variants within the COL3A1 gene, characterized by recurrent arterial, digestive, and pulmonary events. All consecutive vEDS patients referred to the national tertiary referral center for vEDS, between 2004 and 2016, were included. Chest CT scans obtained during the initial vascular work-up were reviewed retrospectively by two chest radiologists for lung involvement. Five surgical samples underwent histologic examination. Among 136 enrolled patients (83 women, 53 men; mean age 37 years) with molecularly confirmed vEDS, 24 (17.6%) had a history of respiratory events: 17 with pneumothorax, 4 with hemothorax, and 3 with hemoptysis that required thoracic surgery in 11. CT scans detected lung parenchymal abnormalities in 78 (57.3%) patients: emphysema (mostly centrilobular and paraseptal) in 44 (32.3%), comparable for smokers and non-smokers; clusters of calcified small pulmonary nodules in 9 (6.6%); and cavitated nodules in 4 (2.9%). Histologic examination of surgical samples found arterial abnormalities, emphysema with alveolar ruptures in 3, accompanied by diffuse hemorrhage and increased hemosiderin resorption. In vEDS patients, identification of lung parenchymal abnormalities is common on CT. The most frequently observed CT finding was emphysema suggesting alveolar wall rupture which might facilitate the diagnostic screening of the disease in asymptomatic carriers of a genetic COL3A1 gene mutation. The prognostic value and evolution of these parenchymal abnormalities remain to be evaluated. • Patients with vEDS can have lung parenchymal changes on top of or next to thoracal vascular abnormalities and that these changes can be present in asymptomatic cases. • The presence of these parenchymal changes is associated with a slightly higher incidence of respiratory events (although not statistically significant). • Identification of the described CT pattern by radiologists and chest physicians may facilitate diagnostic screening.

Identifiants

pubmed: 33651202
doi: 10.1007/s00330-021-07710-6
pii: 10.1007/s00330-021-07710-6
doi:

Substances chimiques

Collagen Type III 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6275-6285

Informations de copyright

© 2021. European Society of Radiology.

Références

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Auteurs

Samia Boussouar (S)

Imagerie Cardiovasculaire et Thoracique (ICT), Hôpital Pitié-Salpêtrière, Assistance Publique-Hôpitaux de Paris, Paris, France, ICT Cardiovascular and Thoracic Imaging Unit, LIB Biomedical Imaging Laboratory INSERM, CNRS, ICAN Institute of Cardiometabolism and Nutrition, Sorbonne Université, Pitié-Salpêtrière Hospital (AP-HP), 47-83 Boulevard de l'hôpital, 75651, Paris CEDEX 13, France. samia.boussouar@aphp.fr.

Amira Benattia (A)

Service de Pneumologie, Hôpital Saint Louis, APHP, Paris, France.

Jean-Baptiste Escudié (JB)

Pôle Informatique Médicale et Santé Publique, Hôpital Avicenne, APHP, Paris, France.

Laure Gibault (L)

Université Paris Descartes - Service d'Anatomie Pathologique- Hôpital Européen Georges Pompidou - AP-HP, Paris, France.

Frédérique Capron (F)

Sorbonne université - Service d'anatomie pathologique- Hôpital Pitié-Salpêtrière-Ch. Foix- AP-HP, Paris, France.

Anne Legrand (A)

Université de Paris Centre de Référence des Maladies Vasculaires Rares et service de génétique, HEGP, AP-HP et Inserm U 970, Paris, France.

Pierre-Yves Brillet (PY)

Université Paris 13, Sorbonne Paris Cité, EA2363 'hypoxie et poumon', Paris- Service de Radiologie- Hôpital Avicenne- AP-HP-, Bobigny, France.

Xavier Jeunemaitre (X)

Université de Paris Centre de Référence des Maladies Vasculaires Rares et service de génétique, HEGP, AP-HP et Inserm U 970, Paris, France.
Université de Paris Centre de Référence des Maladies Vasculaires Rares et service de génétique, VASCERN MSA European Reference Centre, HEGP, AP-HP et Inserm U 970, Paris, France.

Philippe A Grenier (PA)

Département de Recherche Clinique et Innovation, Hôpital Foch, Suresnes, France.

Elie Mousseaux (E)

Université de Paris Centre de Référence des Maladies Vasculaires Rares et service de génétique, VASCERN MSA European Reference Centre, HEGP, AP-HP et Inserm U 970, Paris, France.
Université de Paris, Service d'Imagerie Médicale, Hôpital Européen Georges-Pompidou, APHP, Paris Centre de Recherche Cardiovasculaire (PARCC), Paris, France.

Michael Frank (M)

Université de Paris Centre de Référence des Maladies Vasculaires Rares et service de génétique, HEGP, AP-HP et Inserm U 970, Paris, France.
Université de Paris Centre de Référence des Maladies Vasculaires Rares et service de génétique, VASCERN MSA European Reference Centre, HEGP, AP-HP et Inserm U 970, Paris, France.

Olivier Sanchez (O)

Université de Paris - Service de Pneumologie et Soins Intensifs, Centre de Compétences Maladies Rares Pulmonaires - Hôpital Européen Georges Pompidou - AP-HP - INSERM UMRS 1140, Paris, France.

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