Prolonged cetuximab treatment promotes p27


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
04 03 2021
Historique:
received: 13 08 2020
accepted: 22 02 2021
entrez: 5 3 2021
pubmed: 6 3 2021
medline: 15 12 2021
Statut: epublish

Résumé

Cetuximab, an anti-epidermal growth factor receptor (EGFR) monoclonal antibody, is an efficient anti-tumor therapeutic agent that inhibits the activation of EGFR; however, data related to the cellular effects of prolonged cetuximab treatment are limited. In this study, the long-term cellular outcome of prolonged cetuximab treatment and the related molecular mechanism were explored in a head and neck squamous cell carcinoma cell line constitutively expressing a fluorescent ubiquitination-based cell cycle indicator. Fluorescent time-lapse imaging was used to assess clonal growth, cell motility, and cell-cycle progression. Western blot analysis was performed to measure the level of phosphorylation and protein-expression following cetuximab treatment. Over 5 days cetuximab treatment decreased cell motility and enhanced G1 phase cell arrest in the central region of the colonies. Significantly decreased phosphorylation of retinoblastoma, Skp2, and Akt-mTOR proteins, accumulation of p27

Identifiants

pubmed: 33664437
doi: 10.1038/s41598-021-84877-4
pii: 10.1038/s41598-021-84877-4
pmc: PMC7933308
doi:

Substances chimiques

CDKN1B protein, human 0
MAP1LC3B protein, human 0
Microtubule-Associated Proteins 0
S-Phase Kinase-Associated Proteins 0
Cyclin-Dependent Kinase Inhibitor p27 147604-94-2
EGFR protein, human EC 2.7.10.1
ErbB Receptors EC 2.7.10.1
Proto-Oncogene Proteins c-akt EC 2.7.11.1
TOR Serine-Threonine Kinases EC 2.7.11.1
Cetuximab PQX0D8J21J

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5259

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Auteurs

Kohei Okuyama (K)

Department of Oral and Maxillofacial Surgery, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University, 1-5-45, Yushima, Bunkyo-ku, Tokyo, 113-8510, Japan. okuyamak.0429@gmail.com.
Department of Radiation Medical Sciences, Atomic Bomb Disease Institute, Nagasaki University, Nagasaki, Japan. okuyamak.0429@gmail.com.

Keiji Suzuki (K)

Department of Radiation Medical Sciences, Atomic Bomb Disease Institute, Nagasaki University, Nagasaki, Japan.

Tomofumi Naruse (T)

Department of Clinical Oral Oncology, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.

Hiroki Tsuchihashi (H)

Department of Clinical Oral Oncology, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Department of Maxillofacial Diagnostic and Surgical Science, Field of Oral and Maxillofacial Rehabilitation, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan.

Souichi Yanamoto (S)

Department of Clinical Oral Oncology, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.

Atsushi Kaida (A)

Department of Cancer Biology, The University of Kansas Medical Center, Kansas City, KS, USA.
Division of Oral Health Science, Department of Oral Radiation Oncology, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University, Tokyo, Japan.

Masahiko Miura (M)

Division of Oral Health Science, Department of Oral Radiation Oncology, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University, Tokyo, Japan.

Masahiro Umeda (M)

Department of Clinical Oral Oncology, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.

Shunichi Yamashita (S)

Department of Radiation Medical Sciences, Atomic Bomb Disease Institute, Nagasaki University, Nagasaki, Japan.
Center for Global Exchange, Fukushima Medical University, Fukushima, Japan.
Center for Advanced Radiation Emergency Medicine, National Institutes for Quantum and Radiological Science and Technology, Chiba, Japan.

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Classifications MeSH