Selective Autophagy Receptors in Antiviral Defense.


Journal

Trends in microbiology
ISSN: 1878-4380
Titre abrégé: Trends Microbiol
Pays: England
ID NLM: 9310916

Informations de publication

Date de publication:
09 2021
Historique:
received: 02 12 2020
revised: 08 02 2021
accepted: 10 02 2021
pubmed: 9 3 2021
medline: 15 12 2021
entrez: 8 3 2021
Statut: ppublish

Résumé

Autophagy ensures the degradation of cytosolic substrates by the lysosomal pathway. Cargoes destined to be eliminated are confined within double-membrane vesicles called autophagosomes, prior to fusion with endolysosomal vacuoles. Autophagy receptors selectively interact with cargoes and route them to elongating autophagic membranes, a process referred to as selective autophagy. Besides contributing to cell homeostasis, selective autophagy constitutes an important cell-autonomous defense mechanism against viruses. We review observations related to selective autophagy receptor engagement during host cell responses to virus infection. We examine the distinct roles of autophagy receptors in antiviral autophagy, consider the strategies viruses have evolved to escape or oppose such restrictions, and delineate the contributions of selective autophagy to the tailoring of antiviral innate responses. Finally, we mention some open and emerging questions in the field.

Identifiants

pubmed: 33678557
pii: S0966-842X(21)00042-1
doi: 10.1016/j.tim.2021.02.006
pii:
doi:

Substances chimiques

Receptors, Virus 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

798-810

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Interests No interests are declared.

Auteurs

Christophe Viret (C)

CIRI, Centre International de Recherche en Infectiologie, Team Autophagy Infection Immunity, Université de Lyon, Inserm U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, F-69007, Lyon, France.

Rémi Duclaux-Loras (R)

CIRI, Centre International de Recherche en Infectiologie, Team Autophagy Infection Immunity, Université de Lyon, Inserm U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, F-69007, Lyon, France; Department of Pediatric Hepatology, Gastroenterology and Nutrition, Femme-Mère-Enfant Hospital, Hospices Civils de Lyon, Bron, France.

Stéphane Nancey (S)

CIRI, Centre International de Recherche en Infectiologie, Team Autophagy Infection Immunity, Université de Lyon, Inserm U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, F-69007, Lyon, France; Department of Gastroenterology, Lyon Sud Hospital, Hospices Civils de Lyon, Lyon, France.

Aurore Rozières (A)

CIRI, Centre International de Recherche en Infectiologie, Team Autophagy Infection Immunity, Université de Lyon, Inserm U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, F-69007, Lyon, France.

Mathias Faure (M)

CIRI, Centre International de Recherche en Infectiologie, Team Autophagy Infection Immunity, Université de Lyon, Inserm U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, F-69007, Lyon, France; Equipe Labellisée par la Fondation pour la Recherche Médicale, FRM, France. Electronic address: mathias.faure@inserm.fr.

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Classifications MeSH