Mechanisms for establishment of GABA signaling in adrenal medullary chromaffin cells.


Journal

Journal of neurochemistry
ISSN: 1471-4159
Titre abrégé: J Neurochem
Pays: England
ID NLM: 2985190R

Informations de publication

Date de publication:
07 2021
Historique:
revised: 05 03 2021
received: 23 07 2020
accepted: 07 03 2021
pubmed: 12 3 2021
medline: 16 11 2021
entrez: 11 3 2021
Statut: ppublish

Résumé

γ-Aminobutyric acid (GABA) is thought to play a paracrine role in adrenal medullary chromaffin (AMC) cells. Comparative physiological and immunocytochemical approaches were used to address the issue of how the paracrine function of GABA in AMC cells is established. GABA

Identifiants

pubmed: 33704788
doi: 10.1111/jnc.15345
doi:

Substances chimiques

Chloride Channels 0
Receptors, GABA-A 0
Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide 0
gamma-Aminobutyric Acid 56-12-2
Pregnanolone BXO86P3XXW
Glutamate Decarboxylase EC 4.1.1.15
glutamate decarboxylase 1 EC 4.1.1.15
Hydrocortisone WI4X0X7BPJ

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

153-168

Informations de copyright

© 2021 International Society for Neurochemistry.

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Auteurs

Keita Harada (K)

Department of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan.

Hidetada Matsuoka (H)

Department of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan.

Yumiko Toyohira (Y)

Department of Pharmacology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan.

Yuchio Yanagawa (Y)

Department of Genetic and Behavioral Neuroscience, Gunma University Graduate School of Medicine, Maebashi, Japan.

Masumi Inoue (M)

Department of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan.

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