Heart valve calcification and cardiac hemodynamics.


Journal

Echocardiography (Mount Kisco, N.Y.)
ISSN: 1540-8175
Titre abrégé: Echocardiography
Pays: United States
ID NLM: 8511187

Informations de publication

Date de publication:
04 2021
Historique:
revised: 05 01 2021
received: 25 11 2020
accepted: 25 01 2021
pubmed: 12 3 2021
medline: 10 7 2021
entrez: 11 3 2021
Statut: ppublish

Résumé

Heart valve calcification (VC) is associated with increased cardiovascular risk, but the hemodynamic and functional profile of patients affected by VC has not been fully explored. The study population was formed by consecutive unselected patients included in seven echocardiographic laboratories in a 2-week period. A comprehensive echocardiographic examination was performed. VC was defined by the presence of calcification on at least one valve. Population was formed of 1098 patients (mean age 65 ± 15 years; 47% female). VC was present in 31% of the overall population. Compared with subjects without VC, VC patients were older (60 ± 14 vs 75 ± 9; P < .0001), had more hypertension (40% vs 57%; P = .0005), diabetes (11% vs 18%; P = .002), coronary artery disease (22% vs 38%; P = .04), and chronic kidney disease (4% vs 8%; P = .007). Furthermore, VC patients had lower ejection fraction (55 ± 14 vs 53 ± 25; P < .0001), worse diastolic function (E/e' 8.5 ± 4.6 vs 13.0 ± 7.1; P < .0001) and higher pulmonary artery pressure (29 ± 9 vs 37 ± 12; P < .0001). The association between VC and EF was not independent of etiology (p for VC 0.13), whereas the association with E/e' and PASP was independent in a full multivariate model (P < .0001 and P = .0002, respectively). Heart valve calcification patients were characterized by a worse functional and hemodynamic profile compared to patients with normal valve. The association between VC and diastolic function and PASP were independent in comprehensive multivariate models.

Identifiants

pubmed: 33705585
doi: 10.1111/echo.14994
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

525-530

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

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Auteurs

Andrea Rossi (A)

Section of Cardiology, Department of Medicine, School of Medicine, Verona, Italy.

Andrea Barbieri (A)

Department of Cardiology, Policlinico Hospital, Modena and Reggio Emilia University, Modena, Italy.

Giovanni Benfari (G)

Section of Cardiology, Department of Medicine, School of Medicine, Verona, Italy.

Nicola Gaibazzi (N)

Cardiology Division, Parma Hospital, Parma, Italy.

Andrea Erlicher (A)

Cardiology Division, Bolzano Hospital, Bolzano, Italy.

Gianfrancesco Mureddu (G)

Cardiology Division, S. Giovanni Hospital, Roma, Italy.

Silvia Frattini (S)

Cardiology Division, Cremona Hospital, Cremona, Italy.

Giacomo Faden (G)

Cardiology Division, Pieve di Coriano, Brescia, Italy.

Marcella Manicardi (M)

Department of Cardiology, Policlinico Hospital, Modena and Reggio Emilia University, Modena, Italy.

Monica Beraldi (M)

Cardiology Division, Mantova Hospital, Mantova, Italy.

Francesco Agostini (F)

Cardiology Division, Mantova Hospital, Mantova, Italy.

Valentina Lazzarini (V)

Cardiology Division, Castiglione delle Stiviere Hospital, Mantova, Italy.

Antonella Moreo (A)

Cardiology Division, Niguarda Hospital, Milano, Italy.

Pier Luigi Temporelli (P)

Division of Cardiology, Istituti Clinici Scientifici Maugeri, IRCCS, Veruno, Italy.

Giovanna Magni (G)

Veneto Institute of Oncology IOV - IRCCS, Padua, Italy.

Gregg Pressman (G)

Einstein Medical Center, Philadelphia, PA, USA.

Pompilio Faggiano (P)

Cardiology Division, Spedali Civili and University of Brescia, Brescia, Italy.

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