Palmitoylated acyl protein thioesterase APT2 deforms membranes to extract substrate acyl chains.


Journal

Nature chemical biology
ISSN: 1552-4469
Titre abrégé: Nat Chem Biol
Pays: United States
ID NLM: 101231976

Informations de publication

Date de publication:
04 2021
Historique:
received: 09 06 2020
accepted: 26 01 2021
revised: 27 12 2020
pubmed: 13 3 2021
medline: 29 4 2021
entrez: 12 3 2021
Statut: ppublish

Résumé

Many biochemical reactions require controlled recruitment of proteins to membranes. This is largely regulated by posttranslational modifications. A frequent one is S-acylation, which consists of the addition of acyl chains and can be reversed by poorly understood acyl protein thioesterases (APTs). Using a panel of computational and experimental approaches, we dissect the mode of action of the major cellular thioesterase APT2 (LYPLA2). We show that soluble APT2 is vulnerable to proteasomal degradation, from which membrane binding protects it. Interaction with membranes requires three consecutive steps: electrostatic attraction, insertion of a hydrophobic loop and S-acylation by the palmitoyltransferases ZDHHC3 or ZDHHC7. Once bound, APT2 is predicted to deform the lipid bilayer to extract the acyl chain bound to its substrate and capture it in a hydrophobic pocket to allow hydrolysis. This molecular understanding of APT2 paves the way to understand the dynamics of APT2-mediated deacylation of substrates throughout the endomembrane system.

Identifiants

pubmed: 33707782
doi: 10.1038/s41589-021-00753-2
pii: 10.1038/s41589-021-00753-2
pmc: PMC7610442
mid: EMS114923
doi:

Substances chimiques

Proteins 0
LYPLA2 protein, human EC 3.1.2.-
Thiolester Hydrolases EC 3.1.2.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

438-447

Subventions

Organisme : Swiss National Science Foundation
ID : 176393
Pays : Switzerland
Organisme : Swiss National Science Foundation
ID : 192608
Pays : Switzerland
Organisme : European Research Council
ID : 340260
Pays : International

Commentaires et corrections

Type : CommentIn

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Auteurs

Laurence Abrami (L)

Global Health Institute, School of Life Sciences, EPFL, Lausanne, Switzerland.

Martina Audagnotto (M)

Institute of Bioengineering, School of Life Sciences, EPFL, Lausanne, Switzerland.

Sylvia Ho (S)

Global Health Institute, School of Life Sciences, EPFL, Lausanne, Switzerland.

Maria Jose Marcaida (MJ)

Institute of Bioengineering, School of Life Sciences, EPFL, Lausanne, Switzerland.

Francisco S Mesquita (FS)

Global Health Institute, School of Life Sciences, EPFL, Lausanne, Switzerland.

Muhammad U Anwar (MU)

Global Health Institute, School of Life Sciences, EPFL, Lausanne, Switzerland.

Patrick A Sandoz (PA)

Global Health Institute, School of Life Sciences, EPFL, Lausanne, Switzerland.
Department of Applied Physics, Science for Life Laboratory, KTH Royal Institute of Technology, Solna, Sweden.

Giulia Fonti (G)

Institute of Bioengineering, School of Life Sciences, EPFL, Lausanne, Switzerland.

Florence Pojer (F)

Protein Production and Structure Core Facility, School of Life Sciences, EPFL, Lausanne, Switzerland.

Matteo Dal Peraro (M)

Institute of Bioengineering, School of Life Sciences, EPFL, Lausanne, Switzerland. matteo.dalperaro@epfl.ch.

F Gisou van der Goot (FG)

Global Health Institute, School of Life Sciences, EPFL, Lausanne, Switzerland. gisou.vandergoot@epfl.ch.

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