Deepen into sleep and wake patterns across Alzheimer's disease phenotypes.

Alzheimer's disease Brainstem neuromodulatory subcortical systems selective vulnerability sleep

Journal

Alzheimer's & dementia : the journal of the Alzheimer's Association
ISSN: 1552-5279
Titre abrégé: Alzheimers Dement
Pays: United States
ID NLM: 101231978

Informations de publication

Date de publication:
08 2021
Historique:
revised: 31 12 2020
received: 17 09 2020
accepted: 12 01 2021
pubmed: 13 3 2021
medline: 20 11 2021
entrez: 12 3 2021
Statut: ppublish

Résumé

Although, the clinical variants of Alzheimer's disease (AD) show distinct patterns of cognitive and behavioral decline, disease progression, and neuropathological features, it is unclear if this clinical heterogeneity extends to sleep-wake patterns. Sleep and wake disturbances are frequent in typical AD, often preceding memory loss and negatively impacting the quality of life of patients and caregivers alike. Still, sleep and wake disorders are often misdiagnosed and undertreated in typical AD. Better characterization of sleep-wake features in AD clinical variants is an unmet gap of high importance because these differing patterns may require tailored treatment strategies. Moreover, as wake-promoting neurons are located in subcortical nuclei and degenerate early in typical AD, contrasting the profiles of sleep-wake patterns in typical and atypical AD aids diagnosis and brings a unique opportunity to uncover the mechanisms underlying AD clinical variants at the subcortical level and mechanisms for selective neuronal vulnerability.

Identifiants

pubmed: 33710762
doi: 10.1002/alz.12304
pmc: PMC8364869
mid: NIHMS1698421
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1403-1406

Subventions

Organisme : NIA NIH HHS
ID : U01 AG057195
Pays : United States
Organisme : NIA NIH HHS
ID : K24 AG053435
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG060477
Pays : United States
Organisme : NIA NIH HHS
ID : RF1 AG032289
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG064314
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG032289
Pays : United States

Informations de copyright

© 2021 the Alzheimer's Association.

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Auteurs

Neus Falgàs (N)

Department of Neurology, Memory and Aging Center, Weill Institute for Neurosciences, University of California San Francisco, San Francisco, California, USA.
Global Brain Health Institute, University of California San Francisco, San Francisco, California, USA.
Alzheimer's disease and other cognitive disorders Unit, Hospital Clínic de Barcelona, Barcelona, Spain.

Christine M Walsh (CM)

Department of Neurology, Memory and Aging Center, Weill Institute for Neurosciences, University of California San Francisco, San Francisco, California, USA.

Thomas C Neylan (TC)

Department of Neurology, Memory and Aging Center, Weill Institute for Neurosciences, University of California San Francisco, San Francisco, California, USA.
Department of Psychiatry, University of California San Francisco, San Francisco, California, USA.

Lea T Grinberg (LT)

Department of Neurology, Memory and Aging Center, Weill Institute for Neurosciences, University of California San Francisco, San Francisco, California, USA.
Global Brain Health Institute, University of California San Francisco, San Francisco, California, USA.
Department of Pathology, University of Sao Paulo Medical School, Sao Paulo, Brazil.
Department of Pathology, University of California San Francisco, San Francisco, California, USA.

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