Emetine suppresses SARS-CoV-2 replication by inhibiting interaction of viral mRNA with eIF4E.


Journal

Antiviral research
ISSN: 1872-9096
Titre abrégé: Antiviral Res
Pays: Netherlands
ID NLM: 8109699

Informations de publication

Date de publication:
05 2021
Historique:
received: 28 11 2020
revised: 23 02 2021
accepted: 27 02 2021
pubmed: 13 3 2021
medline: 29 4 2021
entrez: 12 3 2021
Statut: ppublish

Résumé

Emetine is a FDA-approved drug for the treatment of amebiasis. Previously we demonstrated the antiviral efficacy of emetine against some RNA and DNA viruses. In this study, we evaluated the in vitro antiviral efficacy of emetine against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and found it to be a low nanomolar (nM) inhibitor. Interestingly, emetine exhibited protective efficacy against lethal challenge with infectious bronchitis virus (IBV; a chicken coronavirus) in the embryonated chicken egg infection model. Emetine treatment led to a decrease in viral RNA and protein synthesis without affecting other steps of viral life cycle such as attachment, entry and budding. In a chromatin immunoprecipitation (CHIP) assay, emetine was shown to disrupt the binding of SARS-CoV-2 mRNA with eIF4E (eukaryotic translation initiation factor 4E, a cellular cap-binding protein required for initiation of protein translation). Further, molecular docking and molecular dynamics simulation studies suggested that emetine may bind to the cap-binding pocket of eIF4E, in a similar conformation as m7-GTP binds. Additionally, SARS-CoV-2 was shown to exploit ERK/MNK1/eIF4E signalling pathway for its effective replication in the target cells. Collectively our results suggest that further detailed evaluation of emetine as a potential treatment for COVID-19 may be warranted.

Identifiants

pubmed: 33711336
pii: S0166-3542(21)00046-2
doi: 10.1016/j.antiviral.2021.105056
pmc: PMC7943376
pii:
doi:

Substances chimiques

Antiviral Agents 0
Eukaryotic Initiation Factor-4E 0
RNA, Messenger 0
RNA, Viral 0
Emetine X8D5EPO80M

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

105056

Subventions

Organisme : DBT-Wellcome Trust India Alliance
ID : IA/I/17/2/503313
Pays : India

Informations de copyright

Copyright © 2021 Elsevier B.V. All rights reserved.

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Auteurs

Ram Kumar (R)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Mohammad Afsar (M)

Department of Molecular Reproduction, Development and Genetics, Indian Institute of Science, Bengaluru, India.

Nitin Khandelwal (N)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Yogesh Chander (Y)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Thachamvally Riyesh (T)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Ramesh Kumar Dedar (RK)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Baldev R Gulati (BR)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Yash Pal (Y)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Sanjay Barua (S)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India.

Bhupendra N Tripathi (BN)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India. Electronic address: bntripathi1@yahoo.co.in.

Tanweer Hussain (T)

Department of Molecular Reproduction, Development and Genetics, Indian Institute of Science, Bengaluru, India. Electronic address: hussain@iisc.ac.in.

Naveen Kumar (N)

National Centre for Veterinary Type Cultures, ICAR-National Research Centre on Equines, Hisar, India. Electronic address: naveenkumar.icar@gmail.com.

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Classifications MeSH