Characteristics and risk factors for poor outcome in patients with systemic vasculitis involving the gastrointestinal tract.


Journal

Seminars in arthritis and rheumatism
ISSN: 1532-866X
Titre abrégé: Semin Arthritis Rheum
Pays: United States
ID NLM: 1306053

Informations de publication

Date de publication:
04 2021
Historique:
received: 26 11 2020
revised: 04 02 2021
accepted: 01 03 2021
pubmed: 13 3 2021
medline: 23 9 2021
entrez: 12 3 2021
Statut: ppublish

Résumé

Gastrointestinal (GI) involvement was described to be a poor prognostic factor in systemic necrotizing vasculitis. Its prognostic significance may vary according to clinical presentation and vasculitis subtype. This study investigated risk-factors associated to poor outcome in GI-involvement of vasculitis. Patients with systemic vasculitis as defined by the 2012 Chapel Hill Consensus Conference and presenting with GI involvement were retrospectively included. Baseline characteristics, treatments and outcome were recorded. Primary endpoint was a composite of admission to intensive care unit (ICU), emergency surgical procedure, or death. Two hundred and thirteen patients were included. Vasculitis were distributed as follows: 41% IgA vasculitis, 27% ANCA-associated vasculitis, 17% polyarteritis nodosa (PAN), and 15% other vasculitis. Eighty-three (39%) patients fulfilled the composite primary endpoint within 6 months. Predictive factors associated with the primary endpoint included PAN subtype (OR 3.08, 95% CI 1.29-7.34), performance status (OR 1.40, 1.05-1.87), use of morphine (OR 2.51, 0.87-7.24), abdominal guarding (OR 3.08, 1.01-9.37), ileus (OR 2.29, 0.98-5.32), melena (OR 2.74, 1.17-6.42), increased leukocytes (per G/L, OR 1.05, 1.00-1.10), low hemoglobin (per g/dL, OR 0.80, 0.71-0.91) and increased CRP (log mg/L, OR 1.21, 0.94-1.56). A risk prediction model for the achievement of primary endpoint had a very good performance [C-statistics 0.853 (0.810 to 0.895], and for overall survival as well. Vasculitis presenting with GI involvement have a poor outcome in more than one third of cases. An easy-to-use risk prediction model had a very good performance to predict the admission to ICU, emergency surgical procedure, or death.

Sections du résumé

BACKGROUND
Gastrointestinal (GI) involvement was described to be a poor prognostic factor in systemic necrotizing vasculitis. Its prognostic significance may vary according to clinical presentation and vasculitis subtype.
AIMS
This study investigated risk-factors associated to poor outcome in GI-involvement of vasculitis.
METHODS
Patients with systemic vasculitis as defined by the 2012 Chapel Hill Consensus Conference and presenting with GI involvement were retrospectively included. Baseline characteristics, treatments and outcome were recorded. Primary endpoint was a composite of admission to intensive care unit (ICU), emergency surgical procedure, or death.
RESULTS
Two hundred and thirteen patients were included. Vasculitis were distributed as follows: 41% IgA vasculitis, 27% ANCA-associated vasculitis, 17% polyarteritis nodosa (PAN), and 15% other vasculitis. Eighty-three (39%) patients fulfilled the composite primary endpoint within 6 months. Predictive factors associated with the primary endpoint included PAN subtype (OR 3.08, 95% CI 1.29-7.34), performance status (OR 1.40, 1.05-1.87), use of morphine (OR 2.51, 0.87-7.24), abdominal guarding (OR 3.08, 1.01-9.37), ileus (OR 2.29, 0.98-5.32), melena (OR 2.74, 1.17-6.42), increased leukocytes (per G/L, OR 1.05, 1.00-1.10), low hemoglobin (per g/dL, OR 0.80, 0.71-0.91) and increased CRP (log mg/L, OR 1.21, 0.94-1.56). A risk prediction model for the achievement of primary endpoint had a very good performance [C-statistics 0.853 (0.810 to 0.895], and for overall survival as well.
CONCLUSIONS
Vasculitis presenting with GI involvement have a poor outcome in more than one third of cases. An easy-to-use risk prediction model had a very good performance to predict the admission to ICU, emergency surgical procedure, or death.

Identifiants

pubmed: 33711774
pii: S0049-0172(21)00032-9
doi: 10.1016/j.semarthrit.2021.03.002
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

436-441

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declarations of Competing Interest None.

Auteurs

Ségolène Gendreau (S)

Department of Internal Medicine, Hôpital Cochin, AP-HP, Paris, France.

Raphael Porcher (R)

Centre de Recherche Épidémiologie et Statistique Sorbonne Paris Cité, Hôtel-Dieu, Paris, France.

Benjamin Thoreau (B)

Department of Internal Medicine, Hôpital Cochin, AP-HP, Paris, France.

Romain Paule (R)

Department of Internal Medicine, Hôpital Foch, Suresnes, France.

François Maurier (F)

Department of Internal Medicine, Hôpital Sainte-Blandine De Metz, Metz, France.

Tiphaine Goulenok (T)

Department of Internal Medicine, Hôpital Bichat - Claude-Bernard, AP-HP, Paris, France.

Laure Frumholtz (L)

Department of Dermatology, Hôpital Saint-Louis, AP-HP, Paris, France.

Charlotte Bernigaud (C)

Department of Dermatology, Hôpital Henri Mondor, AP-HP, Créteil, France.

Saskia Ingen-Housz-Oro (S)

Department of Dermatology, Hôpital Henri Mondor, AP-HP, Créteil, France.

Arsène Mekinian (A)

Department of Internal Medicine, Hôpital Saint-Antoine, AP-HP, Paris, France.

Alexandra Audemard-Verger (A)

Department of Internal Medicine and Clinical Immunology, CHRU Tours, Tours, France.

Antoine Gaillet (A)

Intensive-care unit, Hôpital Bichat-Claude-Bernard, AP-HP, Paris, France.

Laurent Perard (L)

Department of Internal Medicine, Centre Hospitalier Saint Joseph Saint Luc, Lyon, France.

Maxime Samson (M)

Department of Clinical Immunology and Internal Medicine, CHU de Dijon, Dijon, France.

Romain Sonneville (R)

Intensive-care unit, Hôpital Bichat-Claude-Bernard, AP-HP, Paris, France.

Jean-Benoît Arlet (JB)

Department of Internal Medicine, Hôpital européen Georges Pompidou, AP-HP, Paris, France.

Adrien Mirouse (A)

Intensive care unit, Hôpital Saint-Louis, AP-HP, France.

Jean-Emmanuel Kahn (JE)

Department of Internal Medicine, Hôpital Ambroise Paré, AP-HP, Boulogne, France.

Julien Charpentier (J)

Intensive care unit, Hôpital Cochin, AP-HP, Paris, France.

Éric Hachulla (É)

Department of Internal Medicine and Clinical Immunology, Hôpital Claude Huriez, Lille, France.

Aurélie Hummel (A)

Department of Nephrology, Hôpital Necker, AP-HP, Paris, France.

Thomas Pires (T)

Department of Internal Medicine, CHU Bordeaux, Bordeaux, France.

Pierre-Louis Carron (PL)

Department of Nephrology, Hôpital Nord - CHU Grenoble, Grenoble, France.

Cécile-Audrey Durel (CA)

Department of Internal Medicine, Hôpital Edouard Herriot, CHU Lyon, Lyon, France.

Wendy Jourde (W)

Department of Internal Medicine, CHU Bordeaux, Bordeaux, France.

Xavier Puechal (X)

Department of Internal Medicine, Hôpital Cochin, AP-HP, Paris, France.

Jean-Christophe Lega (JC)

Department of Internal Medicine, Hôpital Edouard Herriot, CHU Lyon, Lyon, France.

Françoise Sarrot-Reynauld (F)

Department of Internal Medicine, Hôpital Nord - CHU Grenoble, Grenoble, France.

Nathalie Tieulie (N)

Department of Internal Medicine, Hôpital de Nice, Nice, France.

Elisabeth Diot (E)

Department of Internal Medicine and Clinical Immunology, CHRU Tours, Tours, France.

Loïc Guillevin (L)

Department of Internal Medicine, Hôpital Cochin, AP-HP, Paris, France.

Benjamin Terrier (B)

Department of Internal Medicine, Hôpital Cochin, AP-HP, Paris, France. Electronic address: benjamin.terrier@aphp.fr.

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