Maternal seafood intake during pregnancy, prenatal mercury exposure and child body mass index trajectories up to 8 years.


Journal

International journal of epidemiology
ISSN: 1464-3685
Titre abrégé: Int J Epidemiol
Pays: England
ID NLM: 7802871

Informations de publication

Date de publication:
30 08 2021
Historique:
received: 02 03 2020
accepted: 15 02 2021
pubmed: 14 3 2021
medline: 5 10 2021
entrez: 13 3 2021
Statut: ppublish

Résumé

Maternal seafood intake during pregnancy and prenatal mercury exposure may influence children's growth trajectories. This study, based on the Norwegian Mother, Father and Child Cohort Study (MoBa), includes 51 952 mother-child pairs recruited in pregnancy during 2002-08 and a subsample (n = 2277) with maternal mercury concentrations in whole blood. Individual growth trajectories were computed by modelling based on child's reported weight and length/height from 1 month to 8 years. We used linear mixed-effects regression analysis and also conducted discordant-sibling analysis. Maternal lean fish was the main contributor to total seafood intake in pregnancy and was positively but weakly associated with child body mass index (BMI) growth trajectory. Higher prenatal mercury exposure (top decile) was associated with a reduction in child's weight growth trajectory, with the estimates ranging from -130 g [95% Confidence Intervals (CI) = -247, -12 g] at 18 months to -608 g (95% CI = -1.102, -113 g) at 8 years. Maternal fatty fish consumption was positively associated with child weight and BMI growth trajectory, but only in the higher mercury-exposed children (P-interaction = 0.045). Other seafood consumption during pregnancy was negatively associated with child weight growth compared with no intake, and this association was stronger for higher mercury-exposed children (P-interaction = 0.004). No association was observed between discordant maternal seafood intake and child growth in the sibling analysis. Within a population with moderate seafood consumption and low mercury exposure, we found that maternal seafood consumption in pregnancy was associated with child growth trajectories, and the direction of the association varied by seafood type and level of prenatal mercury exposure. Prenatal mercury exposure was negatively associated with child growth. Our findings on maternal seafood intake are likely non-causal.

Sections du résumé

BACKGROUND
Maternal seafood intake during pregnancy and prenatal mercury exposure may influence children's growth trajectories.
METHODS
This study, based on the Norwegian Mother, Father and Child Cohort Study (MoBa), includes 51 952 mother-child pairs recruited in pregnancy during 2002-08 and a subsample (n = 2277) with maternal mercury concentrations in whole blood. Individual growth trajectories were computed by modelling based on child's reported weight and length/height from 1 month to 8 years. We used linear mixed-effects regression analysis and also conducted discordant-sibling analysis.
RESULTS
Maternal lean fish was the main contributor to total seafood intake in pregnancy and was positively but weakly associated with child body mass index (BMI) growth trajectory. Higher prenatal mercury exposure (top decile) was associated with a reduction in child's weight growth trajectory, with the estimates ranging from -130 g [95% Confidence Intervals (CI) = -247, -12 g] at 18 months to -608 g (95% CI = -1.102, -113 g) at 8 years. Maternal fatty fish consumption was positively associated with child weight and BMI growth trajectory, but only in the higher mercury-exposed children (P-interaction = 0.045). Other seafood consumption during pregnancy was negatively associated with child weight growth compared with no intake, and this association was stronger for higher mercury-exposed children (P-interaction = 0.004). No association was observed between discordant maternal seafood intake and child growth in the sibling analysis.
CONCLUSIONS
Within a population with moderate seafood consumption and low mercury exposure, we found that maternal seafood consumption in pregnancy was associated with child growth trajectories, and the direction of the association varied by seafood type and level of prenatal mercury exposure. Prenatal mercury exposure was negatively associated with child growth. Our findings on maternal seafood intake are likely non-causal.

Identifiants

pubmed: 33713119
pii: 6169473
doi: 10.1093/ije/dyab035
pmc: PMC8407875
doi:

Substances chimiques

Mercury FXS1BY2PGL

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1134-1146

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press on behalf of the International Epidemiological Association.

Auteurs

Eleni Papadopoulou (E)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Jérémie Botton (J)

Faculty of Pharmacy, Univ. Paris-Sud, Université Paris-Saclay, Châtenay-Malabry, France.

Ida Henriette Caspersen (IH)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Jan Alexander (J)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Merete Eggesbø (M)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Margaretha Haugen (M)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Nina Iszatt (N)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Bo Jacobsson (B)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.
Department of Obstetrics and Gynecology, Sahlgrenska University Hospital Gothenburg/Östra, Gothenburg, Sweden.

Helle Katrine Knutsen (HK)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Helle Margrete Meltzer (HM)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Verena Sengpiel (V)

Department of Obstetrics and Gynecology, Sahlgrenska University Hospital Gothenburg/Östra, Gothenburg, Sweden.

Nikos Stratakis (N)

Department of Preventive Medicine, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA.

Kristine Vejrup (K)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

Anne Lise Brantsæter (AL)

Norwegian Institute of Public Health, Skoyen, Oslo, Norway.

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