Satellite cells deficiency and defective regeneration in dynamin 2-related centronuclear myopathy.


Journal

FASEB journal : official publication of the Federation of American Societies for Experimental Biology
ISSN: 1530-6860
Titre abrégé: FASEB J
Pays: United States
ID NLM: 8804484

Informations de publication

Date de publication:
04 2021
Historique:
revised: 23 11 2020
received: 27 05 2020
accepted: 21 12 2020
entrez: 14 3 2021
pubmed: 15 3 2021
medline: 22 7 2021
Statut: ppublish

Résumé

Dynamin 2 (DNM2) is a ubiquitously expressed protein involved in many functions related to trafficking and remodeling of membranes and cytoskeleton dynamics. Mutations in the DNM2 gene cause the autosomal dominant centronuclear myopathy (AD-CNM), characterized mainly by muscle weakness and central nuclei. Several defects have been identified in the KI-Dnm2

Identifiants

pubmed: 33715228
doi: 10.1096/fj.202001313RRR
doi:

Substances chimiques

Myogenic Regulatory Factors 0
DNM2 protein, mouse EC 3.6.5.5
Dynamin II EC 3.6.5.5

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e21346

Informations de copyright

© 2021 Federation of American Societies for Experimental Biology.

Références

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Auteurs

Camila F Almeida (C)

Laboratory of Muscle Proteins and Comparative Histopathology, Human Genome and Stem Cell Research Center, Biosciences Institute, University of São Paulo, São Paulo, Brazil.
INSERM, Institute of Myology, Centre of Research in Myology, UMRS 974, Sorbonne Université, Paris, France.

Marc Bitoun (M)

INSERM, Institute of Myology, Centre of Research in Myology, UMRS 974, Sorbonne Université, Paris, France.

Mariz Vainzof (M)

Laboratory of Muscle Proteins and Comparative Histopathology, Human Genome and Stem Cell Research Center, Biosciences Institute, University of São Paulo, São Paulo, Brazil.

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