Molecular Identification and Antifungal Susceptibility of Yeasts and Molds Isolated from Patients with Otomycosis.


Journal

Mycopathologia
ISSN: 1573-0832
Titre abrégé: Mycopathologia
Pays: Netherlands
ID NLM: 7505689

Informations de publication

Date de publication:
May 2021
Historique:
received: 27 09 2020
accepted: 17 02 2021
pubmed: 16 3 2021
medline: 28 9 2021
entrez: 15 3 2021
Statut: ppublish

Résumé

Fungal otitis externa, an infection of the external auditory canal caused by molds and yeasts, accounts for approximately 10-20% of ear canal infections accompanying high recurrence. The purpose of the current study was to assess the pattern of etiological agents of otomycosis and resistance profile as well as the rate of tympanic membrane perforation. A total of 1040 patients with symptoms of fungal otitis externa, in a period of two years, were investigated. The mycological tests revealed the presence of different fungi in 237 ears (22.8%). Fungal otitis was more related to filamentous fungi of the species Aspergillus flavus (54.43%), A. tubingensis (10.97%), and A. niger (8.86%), followed by yeasts, Candida orthopsilosis (7.59%), C. albicans (6.75%), and C. parapsilosis (5.06%). Tympanic membrane perforation rate was found to be 6.75% and was more common with otomycosis caused by A. flavus, A. tubingensis and C. albicans. In antifungal susceptibility tests, all tested drugs showed generally good activity against most isolates of molds and yeasts, while tolnaftate, clotrimazole, nystatin, and terbinafine had lowest effects. We found that among Aspergillus isolates, one A. niger isolate was resistant to voriconazole, and one A. flavus isolate was resistant to amphotericin B. Furthermore, among Candida species, three isolates of C. orthopsilosis showed high MIC values to fluconazole, two C. albicans isolates were considered fluconazole resistant and one isolate of C. parapsilosis was resistant to caspofungin and 3 isolates were resistant to fluconazole. Regarding the existence of the cases with perforated tympanic membrane and emerging species causing fungal otitis in the current report, the importance of the early physical examination, precise molecular identification, and the antifungal susceptibility evaluation is highlighted.

Identifiants

pubmed: 33718990
doi: 10.1007/s11046-021-00537-1
pii: 10.1007/s11046-021-00537-1
doi:

Substances chimiques

Antifungal Agents 0
Amphotericin B 7XU7A7DROE
Fluconazole 8VZV102JFY

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

245-257

Subventions

Organisme : Mazandaran University of Medical Sciences
ID : 1222

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Auteurs

Keyvan Kiakojuri (K)

Department of Ear, Nose, and Throat, Faculty of Medicine, Roohani Hospital, Babol University of Medical Sciences, Babol, Iran.

Saeid Mahdavi Omran (S)

Department of Medical Mycology, School of Medicine, Babol University of Medical Sciences, 47745-47176, Babol, Iran. s.mahdavi@mubabol.ac.ir.

Somayeh Roodgari (S)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.
Student Research Committee, Mazandaran University of Medical Sciences, Sari, Iran.

Mojtaba Taghizadeh Armaki (M)

Department of Medical Mycology, School of Medicine, Babol University of Medical Sciences, 47745-47176, Babol, Iran.

Mohammad Taghi Hedayati (MT)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.

Tahereh Shokohi (T)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.

Iman Haghani (I)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.

Javad Javidnia (J)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.
Student Research Committee, Mazandaran University of Medical Sciences, Sari, Iran.

Firoozeh Kermani (F)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.
Student Research Committee, Mazandaran University of Medical Sciences, Sari, Iran.

Hamid Badali (H)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran.

Mahdi Abastabar (M)

Invasive Fungi Research Center, Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran. mabastabar@gmail.com.
Department of Medical Mycology, School of Medicine, Mazandaran University of Medical Sciences, 48175-1665, Sari, Iran. mabastabar@gmail.com.

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