Effect of preload reducing therapy on right ventricular size and function in patients with arrhythmogenic right ventricular cardiomyopathy.


Journal

Heart rhythm
ISSN: 1556-3871
Titre abrégé: Heart Rhythm
Pays: United States
ID NLM: 101200317

Informations de publication

Date de publication:
07 2021
Historique:
received: 30 11 2020
revised: 23 02 2021
accepted: 09 03 2021
pubmed: 17 3 2021
medline: 11 2 2022
entrez: 16 3 2021
Statut: ppublish

Résumé

Arrhythmogenic right ventricular cardiomyopathy (ARVC) is an important cause of sudden cardiac death in young people and athletes. To date, no treatment has proven to slow the progression of the disease. Preload reducing agents such as nitrates and diuretics have shown promising results in preventing training-induced development of ARVC in a murine model. The purpose of this study was to describe our experience with preload reducing therapy in patients with ARVC and symptomatic right ventricular (RV) dysfunction. We performed retrospective chart review of prospectively collected registry data and included 20 patients with definite ARVC who had serial echocardiographic measurements and an implantable cardioverter-defibrillator. Six of the 20 patients with RV end-diastolic area (RVEDA) above median (>25 cm Patients who received preload reducing agents (n = 6) were older and had larger RVs with lower FAC at baseline. However, treatment with preload reducing agents was associated with less RVEDA enlargement during mean 3.3 (range 1-6.7) years of treatment in multivariate analysis (% change in RVEDA associated with treatment -7.71; 95% confidence interval -13.29 to -2.13; P = .007). Preload reducing agents show promising results in slowing RV enlargement in patients with ARVC and show possible disease-modifying potential.

Sections du résumé

BACKGROUND
Arrhythmogenic right ventricular cardiomyopathy (ARVC) is an important cause of sudden cardiac death in young people and athletes. To date, no treatment has proven to slow the progression of the disease. Preload reducing agents such as nitrates and diuretics have shown promising results in preventing training-induced development of ARVC in a murine model.
OBJECTIVE
The purpose of this study was to describe our experience with preload reducing therapy in patients with ARVC and symptomatic right ventricular (RV) dysfunction.
METHODS
We performed retrospective chart review of prospectively collected registry data and included 20 patients with definite ARVC who had serial echocardiographic measurements and an implantable cardioverter-defibrillator. Six of the 20 patients with RV end-diastolic area (RVEDA) above median (>25 cm
RESULTS
Patients who received preload reducing agents (n = 6) were older and had larger RVs with lower FAC at baseline. However, treatment with preload reducing agents was associated with less RVEDA enlargement during mean 3.3 (range 1-6.7) years of treatment in multivariate analysis (% change in RVEDA associated with treatment -7.71; 95% confidence interval -13.29 to -2.13; P = .007).
CONCLUSION
Preload reducing agents show promising results in slowing RV enlargement in patients with ARVC and show possible disease-modifying potential.

Identifiants

pubmed: 33722762
pii: S1547-5271(21)00210-1
doi: 10.1016/j.hrthm.2021.03.018
pii:
doi:

Substances chimiques

Drug Combinations 0
Vasodilator Agents 0
buthiazide, spironolactone drug combination 0
Hydrochlorothiazide 0J48LPH2TH
Spironolactone 27O7W4T232
Isosorbide Dinitrate IA7306519N

Types de publication

Comparative Study Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1186-1191

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2021 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

Auteurs

Shadi Kalantarian (S)

Department of Cardiology, University of California San Francisco, San Francisco, California. Electronic address: skalanta@post.harvard.edu.

Eric Vittinghoff (E)

Department of Biostatistics and Epidemiology, University of California San Francisco, San Francisco, California.

Liviu Klein (L)

Department of Cardiology, University of California San Francisco, San Francisco, California.

Melvin M Scheinman (MM)

Department of Cardiology, University of California San Francisco, San Francisco, California.

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Classifications MeSH