Plasma HMGB1 levels and physical performance in ICU survivors.


Journal

Acta anaesthesiologica Scandinavica
ISSN: 1399-6576
Titre abrégé: Acta Anaesthesiol Scand
Pays: England
ID NLM: 0370270

Informations de publication

Date de publication:
08 2021
Historique:
revised: 12 02 2021
received: 06 10 2020
accepted: 23 02 2021
pubmed: 17 3 2021
medline: 16 10 2021
entrez: 16 3 2021
Statut: ppublish

Résumé

Physical impairment after critical illness is recognized as a part of the post-intensive care syndrome (PICS). About one third of intensive care unit (ICU) survivors suffer from long-term physical disability, yet the underlying pathophysiological mechanisms remain poorly understood. The pro-inflammatory alarmin, high mobility group box 1 (HMGB1), promotes muscle dysfunction in experimental models, and HMGB1 stays elevated in some patients after ICU discharge. Accordingly, we investigated the relationship between HMGB1 plasma levels and physical performance in ICU survivors. Prospective cohort study of 100 ICU survivors from the general ICU at the Karolinska University Hospital, Sweden. Patients returned for follow up at 3 (58 patients) and 6 months (51 patients) after ICU discharge. Blood samples were collected, and a 6-minute walk test (6-MWT), a handgrip-strength test (HST), and a timed-stands test (TST) were performed. Compared to reference values of the different physical tests, 16% of patients underperformed at all tests at 3 months and 12% at 6 months. All test results, except hand-grip strength left, improved significantly over the follow-up period (P < .05). There was no significant association between plasma HMGB1 levels at 3 and 6 months and scores on the three tests (6-MWT, TST, and HST) (P = .50-0.69). In this follow-up study of ICU survivors, we found no significant association between plasma HMGB1 levels and physical performance. Additional follow-up studies of HMGB1 plasma levels and muscle function in ICU survivors are still warranted. HMGB-1, a marker of cell damage and activation, is known to increase in ICU patients. In study participants at 3- to 6-month post-ICU stay, HMGB-1 levels were still elevated, although no association to the primary outcome, physical performance, was found. Mechanisms for failure to recover physical performance post-ICU remain unclear, and investigations into cause of post-intensive care syndrome need to continue. ClinicalTrials.gov identifier NCT02914756.

Identifiants

pubmed: 33725363
doi: 10.1111/aas.13815
doi:

Substances chimiques

HMGB1 Protein 0

Banques de données

ClinicalTrials.gov
['NCT02914756']

Types de publication

Clinical Trial Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

921-927

Informations de copyright

© 2021 The Authors. Acta Anaesthesiologica Scandinavica published by John Wiley & Sons Ltd on behalf of Acta Anaesthesiologica Scandinavica Foundation.

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Auteurs

Emily Brück (E)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.
Laboratory of Immunobiology, Center for Bioelectronic Medicine, MedTechLabs, Department of Medicine, Solna, Karolinska University Hospital, Solna, Sweden.

Anna Svensson-Raskh (A)

Department of Neurobiology, Care Science and Society, Division of Physiotherapy, Karolinska Institutet, Huddinge, Sweden.
Department of Allied Health Professionals, Functional Area Occupational Therapy & Physiotherapy, Karolinska University Hospital, Stockholm, Sweden.

Jacob W Larsson (JW)

Laboratory of Immunobiology, Center for Bioelectronic Medicine, MedTechLabs, Department of Medicine, Solna, Karolinska University Hospital, Solna, Sweden.

April S Caravaca (AS)

Laboratory of Immunobiology, Center for Bioelectronic Medicine, MedTechLabs, Department of Medicine, Solna, Karolinska University Hospital, Solna, Sweden.

Alessandro L Gallina (AL)

Laboratory of Immunobiology, Center for Bioelectronic Medicine, MedTechLabs, Department of Medicine, Solna, Karolinska University Hospital, Solna, Sweden.

Michael Eberhardson (M)

Laboratory of Immunobiology, Center for Bioelectronic Medicine, MedTechLabs, Department of Medicine, Solna, Karolinska University Hospital, Solna, Sweden.

Peter V Sackey (PV)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.

Peder S Olofsson (PS)

Laboratory of Immunobiology, Center for Bioelectronic Medicine, MedTechLabs, Department of Medicine, Solna, Karolinska University Hospital, Solna, Sweden.
Institute of Bioelectronic Medicine, Feinstein Institutes for Medical Research, Manhasset, NY, USA.

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