Candida pathogens induce protective mitochondria-associated type I interferon signalling and a damage-driven response in vaginal epithelial cells.


Journal

Nature microbiology
ISSN: 2058-5276
Titre abrégé: Nat Microbiol
Pays: England
ID NLM: 101674869

Informations de publication

Date de publication:
05 2021
Historique:
received: 30 03 2020
accepted: 01 02 2021
pubmed: 24 3 2021
medline: 1 9 2021
entrez: 23 3 2021
Statut: ppublish

Résumé

Vaginal candidiasis is an extremely common disease predominantly caused by four phylogenetically diverse species: Candida albicans; Candida glabrata; Candida parapsilosis; and Candida tropicalis. Using a time course infection model of vaginal epithelial cells and dual RNA sequencing, we show that these species exhibit distinct pathogenicity patterns, which are defined by highly species-specific transcriptional profiles during infection of vaginal epithelial cells. In contrast, host cells exhibit a homogeneous response to all species at the early stages of infection, which is characterized by sublethal mitochondrial signalling inducing a protective type I interferon response. At the later stages, the transcriptional response of the host diverges in a species-dependent manner. This divergence is primarily driven by the extent of epithelial damage elicited by species-specific mechanisms, such as secretion of the toxin candidalysin by C. albicans. Our results uncover a dynamic, biphasic response of vaginal epithelial cells to Candida species, which is characterized by protective mitochondria-associated type I interferon signalling and a species-specific damage-driven response.

Identifiants

pubmed: 33753919
doi: 10.1038/s41564-021-00875-2
pii: 10.1038/s41564-021-00875-2
doi:

Substances chimiques

Fungal Proteins 0
Interferon Type I 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

643-657

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Auteurs

Marina Pekmezovic (M)

Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany.

Hrant Hovhannisyan (H)

Bioinformatics and Genomics Programme, Centre for Genomic Regulation, Barcelona Institute of Science and Technology, Barcelona, Spain.
Universitat Pompeu Fabra, Barcelona, Spain.
Life Sciences Department, Barcelona Supercomputing Center, Barcelona, Spain.
Mechanisms of Disease Department, Institute for Research in Biomedicine, Barcelona, Spain.

Mark S Gresnigt (MS)

Junior Research Group Adaptive Pathogenicity Strategies, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany.

Elise Iracane (E)

School of Biomedical and Biomolecular Science and UCD Conway Institute of Biomolecular and Biomedical Research, Conway Institute, University College Dublin, Dublin, Ireland.

João Oliveira-Pacheco (J)

School of Biomedical and Biomolecular Science and UCD Conway Institute of Biomolecular and Biomedical Research, Conway Institute, University College Dublin, Dublin, Ireland.

Sofía Siscar-Lewin (S)

Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany.

Eric Seemann (E)

Institute for Biochemistry I, Jena University Hospital-Friedrich Schiller University, Jena, Germany.

Britta Qualmann (B)

Institute for Biochemistry I, Jena University Hospital-Friedrich Schiller University, Jena, Germany.

Till Kalkreuter (T)

Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany.

Sylvia Müller (S)

Institute of Immunology, Universitätsklinikum Jena, Jena, Germany.

Thomas Kamradt (T)

Institute of Immunology, Universitätsklinikum Jena, Jena, Germany.

Selene Mogavero (S)

Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany.

Sascha Brunke (S)

Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany.

Geraldine Butler (G)

School of Biomedical and Biomolecular Science and UCD Conway Institute of Biomolecular and Biomedical Research, Conway Institute, University College Dublin, Dublin, Ireland.

Toni Gabaldón (T)

Bioinformatics and Genomics Programme, Centre for Genomic Regulation, Barcelona Institute of Science and Technology, Barcelona, Spain. toni.gabaldon.bcn@gmail.com.
Universitat Pompeu Fabra, Barcelona, Spain. toni.gabaldon.bcn@gmail.com.
Institució Catalana de Recerca i Estudis Avançats, Barcelona, Spain. toni.gabaldon.bcn@gmail.com.
Life Sciences Department, Barcelona Supercomputing Center, Barcelona, Spain. toni.gabaldon.bcn@gmail.com.
Mechanisms of Disease Department, Institute for Research in Biomedicine, Barcelona, Spain. toni.gabaldon.bcn@gmail.com.

Bernhard Hube (B)

Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology, Hans Knoell Institute, Jena, Germany. bernhard.hube@leibniz-hki.de.
Institute of Microbiology, Friedrich Schiller University, Jena, Germany. bernhard.hube@leibniz-hki.de.

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