Dietary Nitrate Intake Is Positively Associated with Muscle Function in Men and Women Independent of Physical Activity Levels.


Journal

The Journal of nutrition
ISSN: 1541-6100
Titre abrégé: J Nutr
Pays: United States
ID NLM: 0404243

Informations de publication

Date de publication:
11 05 2021
Historique:
received: 07 10 2020
revised: 03 11 2020
accepted: 01 12 2020
pubmed: 25 3 2021
medline: 2 9 2021
entrez: 24 3 2021
Statut: ppublish

Résumé

Nitrate supplements can improve vascular and muscle function. Whether higher habitual dietary nitrate is associated with better muscle function remains underexplored. The aim was to examine whether habitual dietary nitrate intake is associated with better muscle function in a prospective cohort of men and women, and whether the relation was dependent on levels of physical activity. The sample (n = 3759) was drawn from the Australian Diabetes, Obesity, and Lifestyle Study (AusDiab) (56% female; mean ± SD baseline age: 48.6 ± 11.1 y). Habitual dietary intake was assessed over 12 y by obtaining an average [of at least 2 time points, e.g., baseline (2000/2001) and 2004/2005 and/or 2011/2012] from a food-frequency questionnaire. Nitrate intake was calculated from a validated nitrate database and other published literature. Muscle function was quantified by knee extension strength (KES) and the 8-ft-timed-up-and-go (8ft-TUG) test performed in 2011/2012. Physical activity was assessed by questionnaire. Generalized linear models and logistic regression were used to analyze the data. Median (IQR) total nitrate intake was 65 (52-83) mg/d, with ∼81% derived from vegetables. Individuals in the highest tertile of nitrate intake (median intake: 91 mg/d) had 2.6 kg stronger KES (11%) and 0.24 s faster 8ft-TUG (4%) compared with individuals in the lowest tertile of nitrate intake (median intake: 47 mg/d; both P < 0.05). Similarly, individuals in the highest tertile of nitrate intake had lower odds for weak KES (adjusted OR: 0.69; 95% CI: 0.47, 0.73) and slow 8ft-TUG (adjusted OR: 0.63; 95% CI: 0.50, 0.78) compared with those in the lowest tertile. Physical activity did not influence the relationship between nitrate intake and muscle function (KES; P-interaction = 0.86; 8ft-TUG; P-interaction = 0.99). Higher habitual dietary nitrate intake, predominantly from vegetables, could be an effective way to promote lower-limb muscle strength and physical function in men and women.

Sections du résumé

BACKGROUND
Nitrate supplements can improve vascular and muscle function. Whether higher habitual dietary nitrate is associated with better muscle function remains underexplored.
OBJECTIVE
The aim was to examine whether habitual dietary nitrate intake is associated with better muscle function in a prospective cohort of men and women, and whether the relation was dependent on levels of physical activity.
METHODS
The sample (n = 3759) was drawn from the Australian Diabetes, Obesity, and Lifestyle Study (AusDiab) (56% female; mean ± SD baseline age: 48.6 ± 11.1 y). Habitual dietary intake was assessed over 12 y by obtaining an average [of at least 2 time points, e.g., baseline (2000/2001) and 2004/2005 and/or 2011/2012] from a food-frequency questionnaire. Nitrate intake was calculated from a validated nitrate database and other published literature. Muscle function was quantified by knee extension strength (KES) and the 8-ft-timed-up-and-go (8ft-TUG) test performed in 2011/2012. Physical activity was assessed by questionnaire. Generalized linear models and logistic regression were used to analyze the data.
RESULTS
Median (IQR) total nitrate intake was 65 (52-83) mg/d, with ∼81% derived from vegetables. Individuals in the highest tertile of nitrate intake (median intake: 91 mg/d) had 2.6 kg stronger KES (11%) and 0.24 s faster 8ft-TUG (4%) compared with individuals in the lowest tertile of nitrate intake (median intake: 47 mg/d; both P < 0.05). Similarly, individuals in the highest tertile of nitrate intake had lower odds for weak KES (adjusted OR: 0.69; 95% CI: 0.47, 0.73) and slow 8ft-TUG (adjusted OR: 0.63; 95% CI: 0.50, 0.78) compared with those in the lowest tertile. Physical activity did not influence the relationship between nitrate intake and muscle function (KES; P-interaction = 0.86; 8ft-TUG; P-interaction = 0.99).
CONCLUSIONS
Higher habitual dietary nitrate intake, predominantly from vegetables, could be an effective way to promote lower-limb muscle strength and physical function in men and women.

Identifiants

pubmed: 33760920
pii: S0022-3166(22)00156-0
doi: 10.1093/jn/nxaa415
doi:

Substances chimiques

Nitrates 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1222-1230

Commentaires et corrections

Type : CommentIn

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press on behalf of the American Society for Nutrition.

Auteurs

Marc Sim (M)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.
Medical School, Royal Perth Hospital Unit, The University Western Australia, Perth, Western Australia, Australia.

Lauren C Blekkenhorst (LC)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.
Medical School, Royal Perth Hospital Unit, The University Western Australia, Perth, Western Australia, Australia.

Nicola P Bondonno (NP)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.
School of Biomedical Sciences, The University Western Australia, Perth, Western Australia, Australia.

Simone Radavelli-Bagatini (S)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.

Peter Peeling (P)

School of Human Sciences (Exercise and Sports Science), The University Western Australia, Perth, Western Australia, Australia.
Western Australian Institute of Sport, Mt Claremont, Western Australia, Australia.

Catherine P Bondonno (CP)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.
Medical School, Royal Perth Hospital Unit, The University Western Australia, Perth, Western Australia, Australia.

Dianna J Magliano (DJ)

Baker Heart and Diabetes Institute, Melbourne, Victoria, Australia.
Monash University, School of Public Health and Preventive Medicine, Melbourne, Victoria, Australia.

Jonathan E Shaw (JE)

Baker Heart and Diabetes Institute, Melbourne, Victoria, Australia.
Monash University, School of Public Health and Preventive Medicine, Melbourne, Victoria, Australia.

Richard Woodman (R)

Flinders Centre for Epidemiology and Biostatistics, Flinders University, Adelaide, South Australia, Australia.

Kevin Murray (K)

School of Population and Global Health, The University Western Australia, Perth, Western Australia, Australia.

Joshua R Lewis (JR)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.
Medical School, Royal Perth Hospital Unit, The University Western Australia, Perth, Western Australia, Australia.
Centre for Kidney Research, Children's Hospital at Westmead, School of Public Health, Sydney Medical School, The University of Sydney, Sydney, New South Wales, Australia.

Robin M Daly (RM)

Institute for Physical Activity and Nutrition, School of Exercise and Nutrition Sciences, Deakin University, Geelong, Victoria, Australia.

Jonathan M Hodgson (JM)

Institute for Nutrition Research, School of Medical and Health Sciences, Edith Cowan University, Joondalup, Western Australia, Australia.
Medical School, Royal Perth Hospital Unit, The University Western Australia, Perth, Western Australia, Australia.

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