Increased peripheral inflammation in schizophrenia is associated with worse cognitive performance and related cortical thickness reductions.


Journal

European archives of psychiatry and clinical neuroscience
ISSN: 1433-8491
Titre abrégé: Eur Arch Psychiatry Clin Neurosci
Pays: Germany
ID NLM: 9103030

Informations de publication

Date de publication:
Jun 2021
Historique:
received: 11 10 2020
accepted: 03 02 2021
pubmed: 25 3 2021
medline: 15 12 2021
entrez: 24 3 2021
Statut: ppublish

Résumé

While the biological substrates of brain and behavioural changes in persons with schizophrenia remain unclear, increasing evidence implicates that inflammation is involved. In schizophrenia, including first-episode psychosis and anti-psychotic naïve patients, there are numerous reports of increased peripheral inflammation, cognitive deficits and neuropathologies such as cortical thinning. Research defining the relationship between inflammation and schizophrenia symptomatology and neuropathology is needed. Therefore, we analysed the level of C-reactive protein (CRP), a peripheral inflammation marker, and its relationship with cognitive functioning in a cohort of 644 controls and 499 schizophrenia patients. In a subset of individuals who underwent MRI scanning (99 controls and 194 schizophrenia cases), we tested if serum CRP was associated with cortical thickness. CRP was significantly increased in schizophrenia patients compared to controls, co-varying for age, sex, overweight/obesity and diabetes (p < 0.006E-10). In schizophrenia, increased CRP was mildly associated with worse performance in attention, controlling for age, sex and education (R =- 0.15, p = 0.001). Further, increased CRP was associated with reduced cortical thickness in three regions related to attention: the caudal middle frontal, the pars opercularis and the posterior cingulate cortices, which remained significant after controlling for multiple comparisons (all p < 0.05). Together, these findings indicate that increased peripheral inflammation is associated with deficits in cognitive function and brain structure in schizophrenia, especially reduced attention and reduced cortical thickness in associated brain regions. Using CRP as a biomarker of peripheral inflammation in persons with schizophrenia may help to identify vulnerable patients and those that may benefit from adjunctive anti-inflammatory treatments.

Identifiants

pubmed: 33760971
doi: 10.1007/s00406-021-01237-z
pii: 10.1007/s00406-021-01237-z
doi:

Substances chimiques

Biomarkers 0
C-Reactive Protein 9007-41-4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

595-607

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Auteurs

Hayley F North (HF)

School of Psychiatry, University of New South Wales, Sydney, NSW, Australia.
Neuroscience Research Australia, Randwick, Sydney, NSW, Australia.

Jason Bruggemann (J)

School of Psychiatry, University of New South Wales, Sydney, NSW, Australia.
Neuroscience Research Australia, Randwick, Sydney, NSW, Australia.

Vanessa Cropley (V)

Melbourne Neuropsychiatry Centre, Department of Psychiatry, The University of Melbourne and Melbourne Health, Parkville, VIC, Australia.

Vaidy Swaminathan (V)

Department of Psychiatry, School of Clinical Sciences, Monash University, Clayton, VIC, Australia.
Mental Health Program,, Monash Health, Clayton, VIC, Australia.

Suresh Sundram (S)

Department of Psychiatry, School of Clinical Sciences, Monash University, Clayton, VIC, Australia.
Mental Health Program,, Monash Health, Clayton, VIC, Australia.
Northern Psychiatry Research Centre, North Western Mental Health, Melbourne Health, Melbourne, VIC, Australia.
Molecular Psychopharmacology Laboratory, The Florey Institute of Neuroscience and Mental Health, Parkville, VIC, Australia.

Rhoshel Lenroot (R)

School of Psychiatry, University of New South Wales, Sydney, NSW, Australia.
Neuroscience Research Australia, Randwick, Sydney, NSW, Australia.
Department of Psychiatry and Behavioral Sciences, School of Medicine, University of New Mexico, Albuquerque, NM, USA.

Avril M Pereira (AM)

Melbourne Neuropsychiatry Centre, Department of Psychiatry, The University of Melbourne and Melbourne Health, Parkville, VIC, Australia.
Molecular Psychopharmacology Laboratory, The Florey Institute of Neuroscience and Mental Health, Parkville, VIC, Australia.

Andrew Zalesky (A)

Melbourne Neuropsychiatry Centre, Department of Psychiatry, The University of Melbourne and Melbourne Health, Parkville, VIC, Australia.

Chad Bousman (C)

Melbourne Neuropsychiatry Centre, Department of Psychiatry, The University of Melbourne and Melbourne Health, Parkville, VIC, Australia.
Department of Medical Genetics, Psychiatry, and Physiology and Pharmacology, University of Calgary, Calgary, AB, Canada.

Christos Pantelis (C)

Melbourne Neuropsychiatry Centre, Department of Psychiatry, The University of Melbourne and Melbourne Health, Parkville, VIC, Australia.

Thomas W Weickert (TW)

School of Psychiatry, University of New South Wales, Sydney, NSW, Australia.
Neuroscience Research Australia, Randwick, Sydney, NSW, Australia.
Department of Neuroscience and Physiology, Upstate Medical University, Syracuse, NY, 13210, USA.

Cynthia Shannon Weickert (C)

School of Psychiatry, University of New South Wales, Sydney, NSW, Australia. weickerc@upstate.edu.
Neuroscience Research Australia, Randwick, Sydney, NSW, Australia. weickerc@upstate.edu.
Department of Neuroscience and Physiology, Upstate Medical University, Syracuse, NY, 13210, USA. weickerc@upstate.edu.

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