Oncolytic virotherapy induced CSDE1 neo-antigenesis restricts VSV replication but can be targeted by immunotherapy.
Animals
Cancer Vaccines
/ administration & dosage
Cell Line, Tumor
DNA-Binding Proteins
/ genetics
Female
Humans
Immunotherapy
/ methods
Interferon-beta
/ immunology
Mice, Inbred C57BL
Mutation
Oncolytic Virotherapy
/ methods
Oncolytic Viruses
/ immunology
RNA-Binding Proteins
/ genetics
T-Lymphocytes
/ immunology
Vesicular stomatitis Indiana virus
/ immunology
Virus Replication
/ immunology
Journal
Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555
Informations de publication
Date de publication:
26 03 2021
26 03 2021
Historique:
received:
22
07
2020
accepted:
25
02
2021
entrez:
27
3
2021
pubmed:
28
3
2021
medline:
13
4
2021
Statut:
epublish
Résumé
In our clinical trials of oncolytic vesicular stomatitis virus expressing interferon beta (VSV-IFNβ), several patients achieved initial responses followed by aggressive relapse. We show here that VSV-IFNβ-escape tumors predictably express a point-mutated CSDE1
Identifiants
pubmed: 33772027
doi: 10.1038/s41467-021-22115-1
pii: 10.1038/s41467-021-22115-1
pmc: PMC7997928
doi:
Substances chimiques
CSDE1 protein, human
0
Cancer Vaccines
0
DNA-Binding Proteins
0
RNA-Binding Proteins
0
Interferon-beta
77238-31-4
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1930Subventions
Organisme : NCI NIH HHS
ID : P50 CA210964
Pays : United States
Organisme : NIAID NIH HHS
ID : T32 AI132165
Pays : United States
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