A Comparison of Growth Factors and Cytokines in Fresh Frozen Plasma and Never Frozen Plasma.


Journal

The Journal of surgical research
ISSN: 1095-8673
Titre abrégé: J Surg Res
Pays: United States
ID NLM: 0376340

Informations de publication

Date de publication:
08 2021
Historique:
received: 16 08 2020
revised: 28 01 2021
accepted: 17 02 2021
pubmed: 28 3 2021
medline: 24 9 2021
entrez: 27 3 2021
Statut: ppublish

Résumé

Fresh frozen plasma (FFP) contains proinflammatory mediators released from cellular debris during frozen storage. In addition, recent studies have shown that transfusion of never-frozen plasma (NFP), instead of FFP, may be superior in trauma patients. We hypothesized that FFP would have higher levels of inflammatory mediators when compared to NFP. FFP (n = 8) and NFP (n = 8) samples were obtained from an urban, level 1 trauma center blood bank. The cytokines in these samples were compared using a Milliplex (Milliplex Sigma) human cytokine magnetic bead panel multiplex assay for 41 different biomarkers. Growth factors that were higher in NFP included platelet-derived growth factor-AA (PDGF-AA; 8.09 versus 108.00 pg/mL, P < 0.001) and PDGF-AB (0.00 versus 215.20, P= 0.004). Soluble CD40-ligand (sCD40L), a platelet activator and pro-coagulant, was higher in NFP (31.81 versus 80.45 pg/mL, P< 0.001). RANTES, a leukocyte chemotactic cytokine was higher in NFP (26.19 versus 1418.00 pg/mL, P< 0.001). Interleukin-4 (5.70 versus 0.00 pg/mL, P= 0.03) and IL-8 (2.20 versus 0.52 pg/ml, P= 0.03) levels were higher in were higher in FFP. Frozen storage of plasma may result in decrease of several growth factors and/or pro-coagulants found in NFP. In addition, the freezing and thawing process may induce release of pro-inflammatory chemokines. Further studies are needed to determine if these cytokines result in improved outcomes with NFP over FFP in transfusion of trauma patients.

Sections du résumé

BACKGROUND
Fresh frozen plasma (FFP) contains proinflammatory mediators released from cellular debris during frozen storage. In addition, recent studies have shown that transfusion of never-frozen plasma (NFP), instead of FFP, may be superior in trauma patients. We hypothesized that FFP would have higher levels of inflammatory mediators when compared to NFP.
MATERIALS AND METHODS
FFP (n = 8) and NFP (n = 8) samples were obtained from an urban, level 1 trauma center blood bank. The cytokines in these samples were compared using a Milliplex (Milliplex Sigma) human cytokine magnetic bead panel multiplex assay for 41 different biomarkers.
RESULTS
Growth factors that were higher in NFP included platelet-derived growth factor-AA (PDGF-AA; 8.09 versus 108.00 pg/mL, P < 0.001) and PDGF-AB (0.00 versus 215.20, P= 0.004). Soluble CD40-ligand (sCD40L), a platelet activator and pro-coagulant, was higher in NFP (31.81 versus 80.45 pg/mL, P< 0.001). RANTES, a leukocyte chemotactic cytokine was higher in NFP (26.19 versus 1418.00 pg/mL, P< 0.001). Interleukin-4 (5.70 versus 0.00 pg/mL, P= 0.03) and IL-8 (2.20 versus 0.52 pg/ml, P= 0.03) levels were higher in were higher in FFP.
CONCLUSIONS
Frozen storage of plasma may result in decrease of several growth factors and/or pro-coagulants found in NFP. In addition, the freezing and thawing process may induce release of pro-inflammatory chemokines. Further studies are needed to determine if these cytokines result in improved outcomes with NFP over FFP in transfusion of trauma patients.

Identifiants

pubmed: 33773321
pii: S0022-4804(21)00087-1
doi: 10.1016/j.jss.2021.02.002
pii:
doi:

Substances chimiques

Cytokines 0
Intercellular Signaling Peptides and Proteins 0

Types de publication

Comparative Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

51-57

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Auteurs

Sharven Taghavi (S)

Tulane University School of Medicine, Department of Surgery, New Orleans, Louisiana. Electronic address: staghavi@tulane.edu.

Olan Jackson-Weaver (O)

Tulane University School of Medicine, Department of Surgery, New Orleans, Louisiana.

Sarah Abdullah (S)

Tulane University School of Medicine, Department of Surgery, New Orleans, Louisiana.

Amy Goldberg (A)

Temple University School of Medicine, Department of Surgery, Philadelphia, Pennyslvania.

Shaun Lawicki (S)

Louisiana State University School of Medicine, Department of Pathology, New Orleans, Louisiana.

Mary Killackey (M)

Tulane University School of Medicine, Department of Surgery, New Orleans, Louisiana.

Juan Duchesne (J)

Tulane University School of Medicine, Department of Surgery, New Orleans, Louisiana.

Derek Pociask (D)

Tulane University School of Medicine, Center for Translational Research in Infection and Inflammation, New Orleans, Louisiana.

Chad Steele (C)

Tulane University School of Medicine, Department of Microbiology and Immunology, New Orleans, Louisiana.

Jay Kolls (J)

Tulane University School of Medicine, Center for Translational Research in Infection and Inflammation, New Orleans, Louisiana.

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Classifications MeSH