Estimation of intracranial pressure by ultrasound of the optic nerve sheath in an animal model of intracranial hypertension.


Journal

Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia
ISSN: 1532-2653
Titre abrégé: J Clin Neurosci
Pays: Scotland
ID NLM: 9433352

Informations de publication

Date de publication:
Apr 2021
Historique:
received: 11 12 2020
revised: 11 01 2021
accepted: 15 01 2021
entrez: 29 3 2021
pubmed: 30 3 2021
medline: 1 6 2021
Statut: ppublish

Résumé

Ultrasound of the optic nerve sheath diameter (ONSD) has been used as a non-invasive and cost-effective bedside alternative to invasive intracranial pressure (ICP) monitoring. However, ONSD time-lapse behavior in intracranial hypertension (ICH) and its relief by means of either saline infusion or surgery are still unknown. The objective of this study was to correlate intracranial pressure (ICP) and ultrasonography of the optic nerve sheath (ONS) in an experimental animal model of ICH and determine the interval needed for ONSD to return to baseline levels. An experimental study was conducted on 30 pigs. ONSD was evaluated by ultrasound at different ICPs generated by intracranial balloon inflation, saline infusion, and balloon deflation, and measured using an intraventricular catheter. All variables obtained by ONS ultrasonography such as left, right, and average ONSD (AON) were statistically significant to estimate the ICP value. ONSD changed immediately after balloon inflation and returned to baseline after an average delay of 30 min after balloon deflation (p = 0.016). No statistical significance was observed in the ICP and ONSD values with hypertonic saline infusion. In this swine model, ICP and ONSD showed linear correlation and ICP could be estimated using the formula: -80.5 + 238.2 × AON. In the present study, ultrasound to measure ONSD showed a linear correlation with ICP, although a short delay in returning to baseline levels was observed in the case of sudden ICH relief.

Sections du résumé

BACKGROUND BACKGROUND
Ultrasound of the optic nerve sheath diameter (ONSD) has been used as a non-invasive and cost-effective bedside alternative to invasive intracranial pressure (ICP) monitoring. However, ONSD time-lapse behavior in intracranial hypertension (ICH) and its relief by means of either saline infusion or surgery are still unknown. The objective of this study was to correlate intracranial pressure (ICP) and ultrasonography of the optic nerve sheath (ONS) in an experimental animal model of ICH and determine the interval needed for ONSD to return to baseline levels.
METHODS METHODS
An experimental study was conducted on 30 pigs. ONSD was evaluated by ultrasound at different ICPs generated by intracranial balloon inflation, saline infusion, and balloon deflation, and measured using an intraventricular catheter.
RESULTS RESULTS
All variables obtained by ONS ultrasonography such as left, right, and average ONSD (AON) were statistically significant to estimate the ICP value. ONSD changed immediately after balloon inflation and returned to baseline after an average delay of 30 min after balloon deflation (p = 0.016). No statistical significance was observed in the ICP and ONSD values with hypertonic saline infusion. In this swine model, ICP and ONSD showed linear correlation and ICP could be estimated using the formula: -80.5 + 238.2 × AON.
CONCLUSION CONCLUSIONS
In the present study, ultrasound to measure ONSD showed a linear correlation with ICP, although a short delay in returning to baseline levels was observed in the case of sudden ICH relief.

Identifiants

pubmed: 33775322
pii: S0967-5868(21)00030-8
doi: 10.1016/j.jocn.2021.01.021
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

174-179

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Brasil Chian Ping Jeng (BCP)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil.

Almir Ferreira de Andrade (AF)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil.

Sérgio Brasil (S)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil. Electronic address: sbrasil@alumni.usp.br.

Edson Bor-Seng-Shu (E)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil.

Alessandro Rodrigo Belon (AR)

Laboratory of Experimental Surgery, University of São Paulo Medical School, São Paulo, Brazil.

Maira Robertis (M)

Laboratory of Experimental Surgery, University of São Paulo Medical School, São Paulo, Brazil.

Marcelo de-Lima-Oliveira (M)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil.

Andrés Mariano Rubiano (AM)

Universidad El Bosque, Calle 134 Cra 9, Bogotá, Colombia.

Daniel Agustín Godoy (DA)

Neurointensive Care Unit, Sanatorio Pasteur. Intensive Care Unit, Hospital Carlos G. Malbran, Catamarca, Argentina.

Manoel Jacobsen Teixeira (MJ)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil.

Wellingson Silva Paiva (WS)

Division of Neurosurgery, University of São Paulo Medical School, São Paulo, Brazil; Hospital Samaritano, Americas Serviços Médicos, São Paulo, Brazil.

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Classifications MeSH