Pancreatic cancer in patients with autoimmune pancreatitis: A scoping review.


Journal

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
ISSN: 1424-3911
Titre abrégé: Pancreatology
Pays: Switzerland
ID NLM: 100966936

Informations de publication

Date de publication:
Aug 2021
Historique:
received: 03 11 2020
revised: 25 02 2021
accepted: 11 03 2021
pubmed: 30 3 2021
medline: 6 1 2022
entrez: 29 3 2021
Statut: ppublish

Résumé

Chronic pancreatitis is a known risk factor of pancreatic cancer (PDAC). A similar association has been suggested but not demonstrated for autoimmune pancreatitis (AIP). The aim of our study was to identify and analyse all published cases of AIP and PDAC co-occurrence, focusing on the interval between the diagnoses and the cancer site within the pancreas. Relevant studies were identified through automatic searches of the MEDLINE, EMBASE, Scopus, and Web of Science databases, and supplemented by manual checks of reference lists in all retrieved articles. Missing/unpublished data were obtained from the authors of relevant publications in the form of pre-prepared questionnaires. A total of 45 cases of PDAC in AIP patients were identified, of which 12 were excluded from the analysis due to suspicions of duplicity or lack of sufficient data. Thirty-one patients (94%) had type 1 AIP. Synchronous occurrence of PDAC and AIP was reported in 11 patients (33%), metachronous in 22 patients (67%). In the metachronous group, the median period between diagnoses was 66.5 months (2-186) and a majority of cancers (86%) occurred more than two years after AIP diagnosis. In most patients (70%), the cancer originated in the part of the pancreas affected by AIP. In the literature, there are reports on numerous cases of PDAC in AIP patients. PDAC is more frequent in AIP type 1 patients, typically metachronous in character, and generally found in the part of the pancreas affected by AIP.

Sections du résumé

BACKGROUND BACKGROUND
Chronic pancreatitis is a known risk factor of pancreatic cancer (PDAC). A similar association has been suggested but not demonstrated for autoimmune pancreatitis (AIP).
OBJECTIVE OBJECTIVE
The aim of our study was to identify and analyse all published cases of AIP and PDAC co-occurrence, focusing on the interval between the diagnoses and the cancer site within the pancreas.
METHODS METHODS
Relevant studies were identified through automatic searches of the MEDLINE, EMBASE, Scopus, and Web of Science databases, and supplemented by manual checks of reference lists in all retrieved articles. Missing/unpublished data were obtained from the authors of relevant publications in the form of pre-prepared questionnaires.
RESULTS RESULTS
A total of 45 cases of PDAC in AIP patients were identified, of which 12 were excluded from the analysis due to suspicions of duplicity or lack of sufficient data. Thirty-one patients (94%) had type 1 AIP. Synchronous occurrence of PDAC and AIP was reported in 11 patients (33%), metachronous in 22 patients (67%). In the metachronous group, the median period between diagnoses was 66.5 months (2-186) and a majority of cancers (86%) occurred more than two years after AIP diagnosis. In most patients (70%), the cancer originated in the part of the pancreas affected by AIP.
CONCLUSIONS CONCLUSIONS
In the literature, there are reports on numerous cases of PDAC in AIP patients. PDAC is more frequent in AIP type 1 patients, typically metachronous in character, and generally found in the part of the pancreas affected by AIP.

Identifiants

pubmed: 33775564
pii: S1424-3903(21)00101-0
doi: 10.1016/j.pan.2021.03.007
pii:
doi:

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

928-937

Informations de copyright

Copyright © 2021 IAP and EPC. All rights reserved.

Auteurs

Peter Macinga (P)

Department of Gastroenterology and Hepatology, Institute for Clinical and Experimental Medicine, Prague, Czech Republic.

Lukas Bajer (L)

Department of Gastroenterology and Hepatology, Institute for Clinical and Experimental Medicine, Prague, Czech Republic.

Marco Del Chiaro (M)

Department of Surgery, University of Colorado Anschutz Medical Campus, Aurora, CO, 80045, USA.

Suresh T Chari (ST)

Department of Gastroenterology, Hepatology and Nutrition, University of Texas MD Anderson Cancer Center, Houston, TX, 77030, USA.

Petr Dite (P)

Clinic of Internal Medicine, University Hospital Ostrava, Ostrava, Czech Republic.

Luca Frulloni (L)

Gastroenterology Unit, Pancreas Center, University of Verona, Verona, Italy.

Tsukasa Ikeura (T)

Third Department of Internal Medicine, Kansai Medical University, Osaka, Japan.

Terumi Kamisawa (T)

Department of Internal Medicine, Tokyo Metropolitan Komagome Hospital, Tokyo, Japan.

Kensuke Kubota (K)

Department of Gastroenterology and Hepatology, Yokohama City University School of Medicine, Yokohama, Japan.

Itaru Naitoh (I)

Department of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan.

Kazuichi Okazaki (K)

Third Department of Internal Medicine, Kansai Medical University, Osaka, Japan.

Raffaele Pezzilli (R)

Department of Gastroenterology, San Carlo Hospital, Potenza, Italy.

Miroslav Vujasinovic (M)

Department of Medicine Huddinge, Karolinska Institute, Stockholm, Sweden; Department for Upper Digestive Diseases, Karolinska University Hospital, Stockholm, Sweden.

Julius Spicak (J)

Department of Gastroenterology and Hepatology, Institute for Clinical and Experimental Medicine, Prague, Czech Republic.

Tomas Hucl (T)

Department of Gastroenterology and Hepatology, Institute for Clinical and Experimental Medicine, Prague, Czech Republic. Electronic address: tomas.hucl@ikem.cz.

Matthias Lӧhr (M)

CLINTEC, Karolinska Institute, Stockholm, Sweden; Center for Digestive Diseases, Karolinska University Hospital, Stockholm, Sweden.

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Classifications MeSH