SUR1, newly expressed in astrocytes, mediates neuropathic pain in a mouse model of peripheral nerve injury.


Journal

Molecular pain
ISSN: 1744-8069
Titre abrégé: Mol Pain
Pays: United States
ID NLM: 101242662

Informations de publication

Date de publication:
Historique:
entrez: 31 3 2021
pubmed: 1 4 2021
medline: 18 11 2021
Statut: ppublish

Résumé

Neuropathic pain following peripheral nerve injury (PNI) is linked to neuroinflammation in the spinal cord marked by astrocyte activation and upregulation of interleukin 6 (IL Wild type (WT) mice and mice with global or Sciatic nerve cuffing in WT mice resulted in pain behaviors (mechanical allodynia, thermal hyperalgesia) and newly upregulated SUR1-TRPM4 in dorsal horn astrocytes. Global and SUR1-TRPM4 may represent a novel non-addicting target for neuropathic pain.

Sections du résumé

BACKGROUND
Neuropathic pain following peripheral nerve injury (PNI) is linked to neuroinflammation in the spinal cord marked by astrocyte activation and upregulation of interleukin 6 (IL
METHODS
Wild type (WT) mice and mice with global or
RESULTS
Sciatic nerve cuffing in WT mice resulted in pain behaviors (mechanical allodynia, thermal hyperalgesia) and newly upregulated SUR1-TRPM4 in dorsal horn astrocytes. Global and
CONCLUSION
SUR1-TRPM4 may represent a novel non-addicting target for neuropathic pain.

Identifiants

pubmed: 33788643
doi: 10.1177/17448069211006603
pmc: PMC8020112
doi:

Substances chimiques

Abcc8 protein, mouse 0
Sulfonylurea Receptors 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

17448069211006603

Subventions

Organisme : NINDS NIH HHS
ID : R01 NS104297
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS105633
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS099245
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS107262
Pays : United States
Organisme : RRD VA
ID : I01 RX003060
Pays : United States

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Auteurs

Orest Tsymbalyuk (O)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.

Volodymyr Gerzanich (V)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.

Aaida Mumtaz (A)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.

Sanketh Andhavarapu (S)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.

Svetlana Ivanova (S)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.

Tapas K Makar (TK)

Research Service, Veterans Affairs Maryland Health Care System, Baltimore, MD, USA.

Charles A Sansur (CA)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.

Asaf Keller (A)

Department of Anatomy & Neurobiology, University of Maryland School of Medicine, Baltimore, MD, USA.

Yumiko Nakamura (Y)

Pacific Northwest Diabetes Research Institute, Seattle, WA, USA.

Joseph Bryan (J)

Pacific Northwest Diabetes Research Institute, Seattle, WA, USA.

J Marc Simard (JM)

Department of Neurosurgery, University of Maryland School of Medicine, Baltimore, MD, USA.
Research Service, Veterans Affairs Maryland Health Care System, Baltimore, MD, USA.
Department of Pathology, University of Maryland School of Medicine, Baltimore, MD, USA.
Department of Physiology, University of Maryland School of Medicine, Baltimore, MD, USA.

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