Up-regulation of HDAC6 Results in Poor Prognosis and Chemoresistance in Patients With Advanced Ovarian High-grade Serous Carcinoma.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
Mar 2021
Historique:
received: 13 11 2020
revised: 28 01 2021
accepted: 01 02 2021
entrez: 31 3 2021
pubmed: 1 4 2021
medline: 10 4 2021
Statut: ppublish

Résumé

Ovarian high-grade serous carcinoma (HGSC) gradually acquires chemoresistance after recurrence. Our previous study on ovarian clear-cell carcinoma found histone deacetylase 6 (HDAC6) overexpression led to chemoresistance. This study aimed to evaluate HDAC6 as a predictor of chemoresistance and a therapeutic target for ovarian HGSC. The clinical significance of HDAC6 as a predictor of prognosis and chemoresistance in HGSC was immunohistochemically evaluated. In addition, expression of programmed cell death ligand-1 (PD-L1), and hypoxia-inducible factor-1α (HIF1α) were analyzed using clinical samples from 88 patients with ovarian HGSC, and their clinicopathological characteristics were reviewed. Twenty-three patients had high HDAC6 expression, 10 positive PD-L1 expression, and 33 high HIF-1α expression. HDAC6 up-regulation was correlated with not undergoing interval debulking surgery (p<0.001), incomplete surgical resection (p=0.002), and frequent occurrence of stable disease/progressive disease according to the Response Evaluation Criteria in Solid Tumors (p=0.005) criteria. On Kaplan-Meier analysis, high HDAC6 expression was significantly associated with reduced progression-free (p=0.001) and overall (p=0.008) survival. On multivariate analysis, high HDAC6 expression (hazard ratio=1.65, 95% confidence interval 1.03-2.66; p=0.039) and surgery status were independent prognostic factors of progression-free survival. PD-L1 and HIF1α expression positively correlated with that of HDAC6. HDAC6 may become a potential therapeutic target in patients with ovarian HGSC since its up-regulation is considered to be associated with a poor prognosis in patients with this cancer.

Sections du résumé

BACKGROUND BACKGROUND
Ovarian high-grade serous carcinoma (HGSC) gradually acquires chemoresistance after recurrence. Our previous study on ovarian clear-cell carcinoma found histone deacetylase 6 (HDAC6) overexpression led to chemoresistance. This study aimed to evaluate HDAC6 as a predictor of chemoresistance and a therapeutic target for ovarian HGSC.
PATIENTS AND METHODS METHODS
The clinical significance of HDAC6 as a predictor of prognosis and chemoresistance in HGSC was immunohistochemically evaluated. In addition, expression of programmed cell death ligand-1 (PD-L1), and hypoxia-inducible factor-1α (HIF1α) were analyzed using clinical samples from 88 patients with ovarian HGSC, and their clinicopathological characteristics were reviewed.
RESULTS RESULTS
Twenty-three patients had high HDAC6 expression, 10 positive PD-L1 expression, and 33 high HIF-1α expression. HDAC6 up-regulation was correlated with not undergoing interval debulking surgery (p<0.001), incomplete surgical resection (p=0.002), and frequent occurrence of stable disease/progressive disease according to the Response Evaluation Criteria in Solid Tumors (p=0.005) criteria. On Kaplan-Meier analysis, high HDAC6 expression was significantly associated with reduced progression-free (p=0.001) and overall (p=0.008) survival. On multivariate analysis, high HDAC6 expression (hazard ratio=1.65, 95% confidence interval 1.03-2.66; p=0.039) and surgery status were independent prognostic factors of progression-free survival. PD-L1 and HIF1α expression positively correlated with that of HDAC6.
CONCLUSION CONCLUSIONS
HDAC6 may become a potential therapeutic target in patients with ovarian HGSC since its up-regulation is considered to be associated with a poor prognosis in patients with this cancer.

Identifiants

pubmed: 33788761
pii: 41/3/1647
doi: 10.21873/anticanres.14927
doi:

Substances chimiques

B7-H1 Antigen 0
CD274 protein, human 0
HIF1A protein, human 0
Hypoxia-Inducible Factor 1, alpha Subunit 0
HDAC6 protein, human EC 3.5.1.98
Histone Deacetylase 6 EC 3.5.1.98

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1647-1654

Informations de copyright

Copyright © 2021 International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Mitsutake Yano (M)

Department of Pathology, Saitama Medical University International Medical Center, Saitama, Japan.
Department of Obstetrics and Gynaecology, Oita University Faculty of Medicine, Oita, Japan.

Mariko Miyazawa (M)

Department of Obstetrics and Gynaecology, Tokai University School of Medicine, Kanagawa, Japan.

Naoki Ogane (N)

Division of Pathology, Ashigarakami Hospital, Kanagawa, Japan.

Aiko Ogasawara (A)

Department of GynaecologicOncology, Saitama Medical University International Medical Center, Saitama, Japan.

Kosei Hasegawa (K)

Department of GynaecologicOncology, Saitama Medical University International Medical Center, Saitama, Japan.

Hisashi Narahara (H)

Department of Obstetrics and Gynaecology, Oita University Faculty of Medicine, Oita, Japan.

Masanori Yasuda (M)

Department of Pathology, Saitama Medical University International Medical Center, Saitama, Japan; m_yasuda@saitama-med.ac.jp.

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Classifications MeSH